A case of Menkes syndrome with pyloric stenosis: An effect of copper deficiency on NOS1?

1999 ◽  
Vol 22 (2) ◽  
pp. 197-198 ◽  
Author(s):  
S. Cunningham ◽  
P. M. Crofton ◽  
T. G. Marshall ◽  
D. R. Fitzpatrick
2012 ◽  
Vol 265 (2) ◽  
pp. 209-220 ◽  
Author(s):  
Zivanit Ergaz ◽  
Dana Shoshani-Dror ◽  
Claire Guillemin ◽  
Meytal Neeman-azulay ◽  
Liza Fudim ◽  
...  

1966 ◽  
Vol 123 (1) ◽  
pp. 250-254 ◽  
Author(s):  
D. W. Bird ◽  
J. E. Savage ◽  
B. L. Odell

1969 ◽  
Vol 98 (1) ◽  
pp. 57-63 ◽  
Author(s):  
R. B. Rucker ◽  
H. E. Parker ◽  
J. C. Rogler

1967 ◽  
Vol 18 (1) ◽  
pp. 169 ◽  
Author(s):  
GI Alexander ◽  
JM Harvey ◽  
JH Lee ◽  
WC Stubbs

Four experiments described determined the effect of copper and cobalt therapy on the growth and productivity of cattle on the marine plains of central coastal Queensland. Copper was administered by subcutaneous injections of copper glycinate, and cobalt by dosing per os with heavy cobalt pellets. The growth of weaned cattle was significantly improved by copper, particularly from June to October when limited palatable feed on the high ground forced the animals to forage on the para grass swamps. During the same period, 2-year-old heifers also showed a growth response to copper. Their conception rate increased after 19 months of copper therapy but not after 10.5 months. The growth rate of their calves bas significantly increased by copper supplementation. Liver copper concentrations were always low in untreated cattle. Copper therapy maintained these reserves at higher levels, which varied according to the season and the rate of growth of the animals. Calves born to treated cows had higher initial liver copper reserves than those from untreated cows, but in the absence of copper therapy these reserves declined to low and comparable levels in all calves at weaning. Pasture analyses suggest that the copper deficiency revealed was due to interference with copper metabolism rather than to a low copper status in the diet; this interference did not appear to be due to molybdenum. Weaned cattle appeared to respond to cobalt during 1960 but not subsequently, while the cows and calves showed no response. The vitamin B12 status in liver and serum appeared adequate in both treated and untreated cattle.


1984 ◽  
Vol 35 (6) ◽  
pp. 735 ◽  
Author(s):  
MJ Wood ◽  
AD Robson

Wheat was grown in a soil at five levels of copper (ranging from levels deficient, to those luxurious, for plant growth), in the presence or absence of introduced take-all inoculum (oat kernels colonized by Gaeumannomyces graminis var. tritica). The incidence and severity of take-all were related to the copper supply and hence the copper status of the wheat. Plants grown without applied copper were more severely infected by take-all than were those grown with an adequate or luxurious supply of copper. The number of lesions per gram fresh weight of roots was reduced from 6.5 to 2.4 by increasing the copper supply from that severely deficient, to that adequate for plant growth. In seminal roots, increasing the copper supply from levels severely deficient to those adequate or luxurious for plant growth, decreased the length of proximal lesions (those closest to the seed). By contrast, in nodal roots, a similar increase in copper supply had no effect on the length of proximal lesions, but increased the length of uninfected root between the crown and proximal lesions. In both seminal and nodal roots, copper supply did not affect the intensity of lesions.


1976 ◽  
Vol 3 (2) ◽  
pp. 229 ◽  
Author(s):  
RD Graham

Leaf water potential, diffusive resistance, relative water content, weekly water use, yields and head bending were measured on wheat plants subjected to four copper levels (0, 0.4, 0.8 or 4.0 mg Cu per pot) and two water levels (6 or 12% soil water content). Severe copper deficiency (Cu 0) resulted in no grain yield, wilting, increased leaf diffusive resistance and, at the same time, increased leaf water potential relative to plants receiving 4.0 mg Cu (Cu 4.0). Water supply effects were observed but there was no interaction between copper and water treatments. Mild copper deficiency (Cu 0.4, Cu 0.8) resulted in small yield decreases, relative to Cu 4.0, and increased head bending towards maturity. It is concluded that wilting, characteristic of copper-deficient plants, is due to structural weakness (decreased lignification) and not to the water status of the plants; also, increased leaf diffusive resistance is due to a specific effect of copper deficiency on guard cells and not to decreased leaf water potential.


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