Developmental trajectories of adolescent depressive symptoms

2008 ◽  
Author(s):  
D. M. Costello ◽  
J. Swendsen ◽  
J. S. Rose ◽  
L. C. Dierker
2011 ◽  
Vol 63 (4) ◽  
pp. 214-223 ◽  
Author(s):  
John W. Toumbourou ◽  
Ian Williams ◽  
Primrose Letcher ◽  
Ann Sanson ◽  
Diana Smart

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Xiaonan Lin ◽  
Yanmiao Cao ◽  
Linqin Ji ◽  
Wenxin Zhang

AbstractMany efforts have been devoted to investigating the effect of the interaction between the serotonin transporter gene (5-HTTLPR) and environment (G × E) on depression, but they yield mixed results. The inconsistency has suggested that G × E effects may be more complex than originally conceptualized, and further study is warranted. This study explored the association among 5-HTTLPR, peer victimization and depressive symptoms and the underlying mediating role of inhibitory control in this association. A total of 871 Chinese Han adolescents (Mage = 15.32 years, 50.3% girls) participated and provided saliva samples from which the 5-HTTLPR was genotyped. This study found that 5-HTTLPR interacted with peer victimization in predicting depressive symptoms. Adolescents carrying L allele reported more depressive symptoms than SS carriers when exposed to higher level of peer victimization. Furthermore, adolescents’ inhibitory control deficits mediated the association between 5-HTTLPR × peer victimization and depressive symptoms. These findings suggested that one pathway in which G × E may confer vulnerability to depressive symptoms is through disruptions to adolescents’ inhibitory control system.


2021 ◽  
Author(s):  
Savannah Boele ◽  
Stefanie Nelemans ◽  
Jaap J. A. Denissen ◽  
Peter Prinzie ◽  
Anne Bülow ◽  
...  

This multi-sample study tested bidirectional within-family associations between parental sup-port and adolescents’ depressive symptoms on varying measurement intervals: Daily (N = 244, Mage = 13.8, 38% male), two-weekly (N=256, Mage=14.5, 29% male), three-monthly (N=245, Mage=13.9, 38% males), annual (N=1,664, Mage=11.1, 51% male), and biennial (N=502, Mage=13.8, 48% male). Pre-registered random-intercept cross-lagged panel models (RI-CLPM) showed negative between- and within-family correlations. Although no within-family lagged effects were found from parental support to depressive symptoms at any time interval, de-pressive symptoms predicted decreased parental support two weeks and three months later. Effects were moderated by adolescents’ sex and neuroticism. Findings mainly supported ado-lescent-driven effects, and illustrate that within-family lagged effects may not generalize across timescales.


2012 ◽  
Vol 36 (4) ◽  
pp. 247-257 ◽  
Author(s):  
David Y. C. Huang ◽  
H. Isabella Lanza ◽  
Debra A. Murphy ◽  
Yih-Ing Hser

This study used data from 5,382 adolescents from the 1997 United States (US) National Longitudinal Survey of Youth (NLSY97) to investigate developmental pathways of alcohol use, marijuana use, sexual risk behaviors, and delinquency across ages 14 to 20; examine interrelationships among these risk behaviors across adolescence; and evaluate association between risk behavior trajectories and depressive symptoms in adolescence. Group-based dual trajectory modeling, examining trajectories of two outcomes over time, revealed strong interrelationships among developmental trajectories of the four risk behaviors, and indicated potential pathways to co-occurring risk behaviors. Adolescents with higher levels of alcohol use or marijuana use were more likely to engage in higher levels of early sexual risk-taking and delinquency. Moreover, adolescents involved in higher levels of delinquency were at higher risk for engaging in early sexual risk-taking. Also, belonging to the highest risk trajectory of any of the four risk behaviors was positively associated with depressive symptoms in adolescence.


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