The relationship between air-pollution by gas flaring and lung function in Niger-Delta

Pneumologie ◽  
2018 ◽  
Vol 72 (S 01) ◽  
pp. S82-S83
Author(s):  
A Worlu ◽  
K Frank
Keyword(s):  
Air Pollution ◽  
Lung Function ◽  
Niger Delta ◽  
Gas Flaring ◽  
The Relationship

scholarly journals Effect of outdoor particulate air pollution on FEV1 in healthy adults: a systematic review and meta-analysis

2019 ◽  
Vol 76 (8) ◽  
pp. 583-591 ◽  
Author(s):  
Stefan Edginton ◽  
Dylan E O’Sullivan ◽  
Will King ◽  
M Diane Lougheed
Keyword(s):  
Air Pollution ◽  
Lung Function ◽  
Meta Analysis ◽  
Healthy Adults ◽  
Absolute Difference ◽  
Short Term ◽  
Particulate Air Pollution ◽  
Relationship Of ◽  
The Relationship

The effect of acute and long-term exposures to outdoor particulate air pollution on lung function in healthy adults is not well established. The objective of this study was to conduct a systematic literature review and meta-analysis of studies that assessed the relationship of outdoor particulate air pollution and lung function in healthy adults. Studies that contained data on outdoor air particulate matter levels (PM10 or PM2.5) and forced expiratory volume in 1 s (FEV1) in healthy adults were eligible for inclusion. Effect estimates, in relation to long-term and acute exposures, were quantified separately using random effects models. A total of 27 effect estimates from 23 studies were included in this review. Acute exposures were typically assessed with PM2.5, while long-term exposures were predominantly represented by PM10. A 10 µg/m3 increase in short-term PM2.5 exposure (days) was associated with a −7.02 mL (95% CI −11.75 to –2.29) change in FEV1. A 10 µg/m3 difference in long-term PM10 exposure was associated with a −8.72 mL (95% CI −15.39 to –2.07) annual change in FEV1 and an absolute difference in FEV1 of −71.36 mL (95% CI −134.47 to –8.24). This study provides evidence that acute and long-term exposure to outdoor particulate air pollution are associated with decreased FEV1 in healthy adults. Residual confounding from other risk factors, such as smoking, may explain some of the effect for long-term exposures. More studies are required to determine the relationship of long-term exposure to PM2.5 and short-term exposure to PM10, which may have different biologic mechanisms.

scholarly journals Gas Flaring in Niger Delta Region of Nigeria: Cost, Ecological and Human Health Implications

2017 ◽  
Vol 6 (2) ◽  
pp. 390 ◽  
Author(s):  
Christopher A. Osuoha ◽  
Michael A. Fakutiju
Keyword(s):  
Public Health ◽  
Human Health ◽  
Niger Delta ◽  
Social Cost ◽  
Delta Region ◽  
Oil Companies ◽  
Gas Flaring ◽  
Niger Delta Region ◽  
The Impact ◽  
The Relationship

The study examines the relationship between total gas produced, utilized and flared, also the social cost, economic cost and the public health consequences resulting from gas flaring in the Niger Delta region of Nigeria. The environmental and economic impacts caused by gas flaring activities in this part of the world between 1999 and 2015 were analyzed to establish the relationship gas flaring has with lost economic opportunities, ecological damage and human health challenges. The study examined and discussed the gas flaring cost, the volume of gas produced, public health cost, the cost of pollution abatement technology, social cost-benefit, the regulatory policies, and the reason why oil companies still flare gases. There is no single empirical approach, estimation technique or emission index to quantify the exact impact of gas flaring. The impact of gas flaring on the Niger Delta region is not an assumption but a reality that is supported by verifiable evidence. We find that gas flaring has a devastating impact on human health and the natural ecology. Available cost-effective technological solutions can be deployed by the oil companies to abate the flaring, expand revenue and improve the environmental quality in the Niger Delta area. 

The Effect Of Body Mass Index On The Relationship Between Reduced Air Pollution And Lung Function Decline

2011 ◽  
Author(s):  
Tamara Schikowski ◽  
Emmanuel Schaffner ◽  
Harish Phuleria ◽  
Andrea Vierkoetter ◽  
Christian Schindler ◽  
...  
Keyword(s):  
Body Mass Index ◽  
Air Pollution ◽  
Lung Function ◽  
Body Mass ◽  
Lung Function Decline ◽  
The Relationship

scholarly journals Maternal immune response and air pollution exposure during pregnancy: insights from the Early Markers for Autism (EMA) study

2020 ◽  
Vol 12 (1) ◽  
Author(s):  
Heather E. Volk ◽  
Bo Park ◽  
Calliope Hollingue ◽  
Karen L. Jones ◽  
Paul Ashwood ◽  
...  
Keyword(s):  
Air Pollution ◽  
Intellectual Disability ◽  
Prenatal Screening ◽  
Maternal Serum ◽  
Serum Samples ◽  
Maternal Immune Activation ◽  
Early Markers ◽  
Air Pollution Exposure ◽  
Pollution Exposure ◽  
The Relationship

Abstract Background Perinatal exposure to air pollution and immune system dysregulation are two factors consistently associated with autism spectrum disorders (ASD) and other neurodevelopmental outcomes. However, little is known about how air pollution may influence maternal immune function during pregnancy. Objectives To assess the relationship between mid-gestational circulating levels of maternal cytokines/chemokines and previous month air pollution exposure across neurodevelopmental groups, and to assess whether cytokines/chemokines mediate the relationship between air pollution exposures and risk of ASD and/or intellectual disability (ID) in the Early Markers for Autism (EMA) study. Methods EMA is a population-based, nested case–control study which linked archived maternal serum samples collected during weeks 15–19 of gestation for routine prenatal screening, birth records, and Department of Developmental Services (DDS) records. Children receiving DDS services for ASD without intellectual disability (ASD without ID; n = 199), ASD with ID (ASD with ID; n = 180), ID without ASD (ID; n = 164), and children from the general population (GP; n = 414) with no DDS services were included in this analysis. Serum samples were quantified for 22 cytokines/chemokines using Luminex multiplex analysis technology. Air pollution exposure for the month prior to maternal serum collection was assigned based on the Environmental Protection Agency’s Air Quality System data using the maternal residential address reported during the prenatal screening visit. Results Previous month air pollution exposure and mid-gestational maternal cytokine and chemokine levels were significantly correlated, though weak in magnitude (ranging from − 0.16 to 0.13). Ten pairs of mid-pregnancy immune markers and previous month air pollutants were significantly associated within one of the child neurodevelopmental groups, adjusted for covariates (p < 0.001). Mid-pregnancy air pollution was not associated with any neurodevelopmental outcome. IL-6 remained associated with ASD with ID even after adjusting for air pollution exposure. Conclusion This study suggests that maternal immune activation is associated with risk for neurodevelopmental disorders. Furthermore, that prenatal air pollution exposure is associated with small, but perhaps biologically relevant, effects on maternal immune system function during pregnancy. Additional studies are needed to better evaluate how prenatal exposure to air pollution affects the trajectory of maternal immune activation during pregnancy, if windows of heightened susceptibility can be identified, and how these factors influence neurodevelopment of the offspring.

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