scholarly journals Endoscopic Intraventricular Atrial Adhesiolysis for the Treatment of Entrapped Temporal Horn after İntraventricular Tumor Surgery

2020 ◽  
Vol 11 (02) ◽  
pp. 337-340
Author(s):  
Bashar Abuzayed ◽  
Khaled Alawneh ◽  
Majdi Al Qawasmeh ◽  
Liqaa Raffee
Keyword(s):  
Third Ventricle ◽  
Brain Magnetic Resonance Imaging ◽  
Central Neurocytoma ◽  
Early Postoperative Period ◽  
Temporal Horn ◽  
Left Posterior ◽  
Level Of Consciousness ◽  
Posterior Parietal ◽  
Significant Regression ◽  
Ventricle Size

AbstractA 21-year-old male patient was operated for third ventricle tumor (central neurocytoma) and showed improvement in the early postoperative period. After 2 weeks of surgery, the patient neurologic status deteriorated with acute decreased level of consciousness. Neuroimaging of the brain revealed entrapped left temporo-occipital horns with adhesion bands at the level of the atrium. Patient was operated with neuronavigation-guided endoscopic approach to the left atrium through the left posterior parietal region. Band adhesiolysis was performed with no complications. The patient showed fast improvement, and follow-up brain magnetic resonance imaging after 2 years showed the release of the ventricular entrapment with significant regression of the left ventricle size.

scholarly journals Tacrolimus-induced neurotoxicity from bipolar disorder to status epilepticus under the therapeutic serum level: a case report

BMC Neurology ◽  
2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Bora Jin ◽  
Ga Yeon Kim ◽  
Sang-Myung Cheon
Keyword(s):  
Adverse Events ◽  
Therapeutic Range ◽  
Solid Organ Transplantation ◽  
Brain Magnetic Resonance Imaging ◽  
Solid Organ ◽  
Therapeutic Level ◽  
Hyperintense Lesion ◽  
Case Presentation ◽  
Delta Waves ◽  
Level Of Consciousness

Abstract Background Tacrolimus is a macrolide immunosuppressant widely used to prevent rejection after solid organ transplantation. In general, adverse events of tacrolimus occur more often as the concentration of tacrolimus in the blood increases. We report the case of a 39-year-old man who developed a variety of adverse events despite in the therapeutic level of tacrolimus in the blood. Case presentation A 39-year-old man underwent liver transplantation for liver cirrhosis due to alcoholic liver disease. The postoperative immunosuppressant consisted of tacrolimus (5 mg) and mycophenolate (500 mg) twice daily. Five months after taking tacrolimus, he presented with talkativeness, which gradually worsened. Brain magnetic resonance imaging performed 10 months after tacrolimus administration revealed a hyperintense lesion affecting the middle of the pontine tegmentum on T2WI. The blood concentration of tacrolimus was 7.2 ng/mL (therapeutic range 5–20 ng/mL). After 21 months, he exhibited postural tremor in both the hands. Twenty-four months after taking tacrolimus, he showed drowsy mentality, intention tremor, and dysdiadochokinesia. Electroencephalography presented generalized high-voltage rhythmic delta waves; therefore, tacrolimus was discontinued in suspicion of tacrolimus-induced neurotoxicity, and anticonvulsive treatment was started. The level of consciousness gradually improved, and the patient was able to walk independently with mild ataxia. Conclusion This case shows that tacrolimus-induced neurotoxicity can occur even at normal concentrations. Therefore, if a patient taking tacrolimus exhibits psychiatric or neurologic symptoms, neurotoxicity should be considered even when the blood tacrolimus is within the therapeutic range.

scholarly journals TMS-Adaptation Reveals Abstract Letter Selectivity in the Left Posterior Parietal Cortex

2009 ◽  
Vol 19 (10) ◽  
pp. 2321-2325 ◽  
Author(s):  
Z. Cattaneo ◽  
F. Rota ◽  
V. Walsh ◽  
T. Vecchi ◽  
J. Silvanto
Keyword(s):  
Parietal Cortex ◽  
Posterior Parietal Cortex ◽  
Left Posterior ◽  
Posterior Parietal

Third ventricle: size and appearance in normal fetuses through gestation.

Radiology ◽  
1997 ◽  
Vol 203 (3) ◽  
pp. 641-644 ◽  
Author(s):  
B S Hertzberg ◽  
M A Kliewer ◽  
K S Freed ◽  
P J McNally ◽  
D M DeLong ◽  
...  
Keyword(s):  
Third Ventricle ◽  
Ventricle Size

scholarly journals The anodal tDCS over the left posterior parietal cortex enhances attention toward a focus word in a sentence

2014 ◽  
Vol 8 ◽  
Author(s):  
Takehiro Minamoto ◽  
Miyuki Azuma ◽  
Ken Yaoi ◽  
Aoi Ashizuka ◽  
Tastuya Mima ◽  
...  
Keyword(s):  
Parietal Cortex ◽  
Posterior Parietal Cortex ◽  
Anodal Tdcs ◽  
Left Posterior ◽  
Posterior Parietal

scholarly journals Cerebrovascular Accident Secondary to Paradoxical Embolism Following Arteriovenous Graft Thrombectomy

2012 ◽  
Vol 2012 ◽  
pp. 1-3 ◽  
Author(s):  
Jolina Pamela Santos ◽  
Zaher Hamadeh ◽  
Naheed Ansari
Keyword(s):  
Vascular Access ◽  
Parietal Lobe ◽  
Paradoxical Embolism ◽  
Arteriovenous Graft ◽  
Common Procedure ◽  
Left Posterior ◽  
Posterior Parietal ◽  
Stage Renal Disease ◽  
End Stage ◽  
Posterior Parietal Lobe

Thrombectomy is a common procedure performed to declot thrombosed dialysis arteriovenous fistula (AVF) or arteriovenous graft (AVG). Complications associated with access thrombectomy like pulmonary embolism have been reported, but paradoxical embolism is extremely rare. We report a case of a 74-year-old black man with past medical history significant for end-stage renal disease (ESRD), atrial fibrillation on anticoagulation with warfarin, who presented to our hospital with lethargy, aphasia, and right-sided hemiparesis following thrombectomy of a clotted AVG. Computed tomography (CT) scan of brain showed a hypodensity within the left posterior parietal lobe. INR was 2.0 on admission. Echocardiogram revealed a normal sized left atrium with no intracardiac thrombus, and bubble study showed the presence of right-to-left shunting. These findings suggest that the stroke occurred as a result of an embolus originating from the AVG. Paradoxical cerebral embolism is uncommon but can occur after thrombectomy of clotted vascular access in ESRD patients. Clinicians and patients should be aware of this serious and potentially fatal complication of vascular access procedure.

Increased left posterior parietal–temporal cortex activation after d-fenfluramine in women with panic disorder

2000 ◽  
Vol 98 (3) ◽  
pp. 133-143 ◽  
Author(s):  
Jeffrey H Meyer ◽  
Richard Swinson ◽  
Sidney H Kennedy ◽  
Sylvain Houle ◽  
Gregory M Brown
Keyword(s):  
Panic Disorder ◽  
Temporal Cortex ◽  
Left Posterior ◽  
Posterior Parietal

Brain anatomy in non-affected parents of autistic probands: a MRI study

2005 ◽  
Vol 35 (10) ◽  
pp. 1411-1420 ◽  
Author(s):  
SASKIA J. M. C. PALMEN ◽  
HILLEKE E. HULSHOFF POL ◽  
CHANTAL KEMNER ◽  
HUGO G. SCHNACK ◽  
MARGRIET M. SITSKOORN ◽  
...  
Keyword(s):  
Brain Volume ◽  
Neurodevelopmental Disorder ◽  
Third Ventricle ◽  
Brain Magnetic Resonance Imaging ◽  
Occipital Lobe ◽  
Brain Anatomy ◽  
Significant Interaction Effect ◽  
Brain Volumes ◽  
Present Sample ◽  
The Brain

Background. Autism is a neurodevelopmental disorder with an estimated genetic origin of 90%. Previous studies have reported an increase in brain volume of approximately 5% in autistic subjects, especially in children. If this increase in brain volume is genetically determined, biological parents of autistic probands might be expected to show brain enlargement, or at least intracranial enlargement, as well. Identifying structural brain abnormalities under genetic control is of particular importance as these could represent endophenotypes of autism.Method. Using quantitative anatomic brain magnetic resonance imaging, volumes of intracranial, total brain, frontal, parietal, temporal and occipital lobe, cerebral and cortical gray and white matter, cerebellum, lateral ventricle, and third ventricle were measured in biological, non-affected parents of autistic probands (19 couples) and in healthy, closely matched control subjects (20 couples).Results. No significant differences were found between the parents of the autistic probands and healthy control couples in any of the brain volumes. Adding gender as a factor in a second analysis did not reveal a significant interaction effect of gender by group.Conclusions. The present sample of biological, non-affected parents of autistic probands did not show brain enlargements. As the intracranium is not enlarged, it is unlikely that the brain volumes of the parents of autistic probands have originally been enlarged and have been normalized. Thus, increased brain volume in autism might be caused by the interaction of paternal and maternal genes, possibly with an additional effect of environmental factors, or increased brain volumes might reflect phenotypes of autism.

scholarly journals Working Memory Retrieval: Contributions of the Left Prefrontal Cortex, the Left Posterior Parietal Cortex, and the Hippocampus

2009 ◽  
Vol 21 (3) ◽  
pp. 581-593 ◽  
Author(s):  
Ilke Öztekin ◽  
Brian McElree ◽  
Bernhard P. Staresina ◽  
Lila Davachi
Keyword(s):  
Working Memory ◽  
Parietal Cortex ◽  
Posterior Parietal Cortex ◽  
Inferior Frontal Gyrus ◽  
Long Term Memory ◽  
Term Memory ◽  
Focal Attention ◽  
Left Posterior ◽  
Posterior Parietal

Functional magnetic resonance imaging was used to identify regions involved in working memory (WM) retrieval. Neural activation was examined in two WM tasks: an item recognition task, which can be mediated by a direct-access retrieval process, and a judgment of recency task that requires a serial search. Dissociations were found in the activation patterns in the hippocampus and in the left inferior frontal gyrus (LIFG) when the probe contained the most recently studied serial position (where a test probe can be matched to the contents of focal attention) compared to when it contained all other positions (where retrieval is required). The data implicate the hippocampus and the LIFG in retrieval from WM, complementing their established role in long-term memory. Results further suggest that the left posterior parietal cortex (LPPC) supports serial retrieval processes that are often required to recover temporal order information. Together, these data suggest that the LPPC, the LIFG, and the hippocampus collectively support WM retrieval. Critically, the reported findings support accounts that posit a distinction between representations maintained in and outside of focal attention, but are at odds with traditional dual-store models that assume distinct mechanisms for short- and long-term memory representations.

scholarly journals Pituitary Apoplexy Presenting as Ophthalmoplegia and Altered Level of Consciousness without Headache

2018 ◽  
Vol 2018 ◽  
pp. 1-6
Author(s):  
Nooshin Salehi ◽  
Anthony Firek ◽  
Iqbal Munir
Keyword(s):  
Ct Scan ◽  
Cranial Nerve ◽  
Cranial Nerve Palsy ◽  
Nerve Palsy ◽  
Pituitary Apoplexy ◽  
Brain Magnetic Resonance Imaging ◽  
Acute Onset ◽  
Altered Level ◽  
Level Of Consciousness ◽  
The Right

Background. Pituitary apoplexy (PA) is a clinical syndrome caused by acute ischemic infarction or hemorrhage of the pituitary gland. The typical clinical presentation of PA includes acute onset of severe headache, visual disturbance, cranial nerve palsy, and altered level of consciousness. Case Report. A 78-year-old man presented to the emergency department with one-day history of ptosis and diplopia and an acute-onset episode of altered level of consciousness which was resolving. He denied having headache, nausea, or vomiting. Physical examination revealed third-cranial nerve palsy and fourth-cranial nerve palsy both on the right side. Noncontrast computed tomography (CT) scan of the head was unremarkable. Brain magnetic resonance imaging (MRI) showed a pituitary mass with hemorrhage (apoplexy) and extension to the right cavernous sinus. The patient developed another episode of altered level of consciousness in the hospital. Transsphenoidal resection of the tumor was done which resulted in complete recovery of the ophthalmoplegia and mental status. Conclusion. Pituitary apoplexy can present with ophthalmoplegia and altered level of consciousness without having headache, nausea, or vomiting. A CT scan of the head could be negative for hemorrhage. A high index of suspicion is needed for early diagnosis and timely management of pituitary apoplexy.

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