The normal complement problem and the structure of the unitary subgroup

2020 ◽  
Vol 48 (8) ◽  
pp. 3628-3636
Author(s):  
Surinder Kaur ◽  
Manju Khan
Author(s):  
S.R. Allegra

The respective roles of the ribo somes, endoplasmic reticulum, Golgi apparatus and perhaps nucleus in the synthesis and maturation of melanosomes is still the subject of some controversy. While the early melanosomes (premelanosomes) have been frequently demonstrated to originate as Golgi vesicles, it is undeniable that these structures can be formed in cells in which Golgi system is not found. This report was prompted by the findings in an essentially amelanotic human cellular blue nevus (melanocytoma) of two distinct lines of melanocytes one of which was devoid of any trace of Golgi apparatus while the other had normal complement of this organelle.


1971 ◽  
Vol 66 (2) ◽  
pp. 266-272
Author(s):  
Anant P. Labhsetwar

ABSTRACT The ovarian response of adult rats made persistent-oestrus by the postnatal treatment with testosterone propionate was examined by determining the number of ova shed in response to small doses of LH and/or FSH, and placental gonadotrophins (PMSG or HCG). A dose of LH (10 μg/rat) estimated to be equivalent to twice the minimal amount secreted for ovulation in normal rats failed to release a normal complement of ova. The same dose of FSH induced ovulation in 40% of rats with an ova count of 5.7 ± 1.8/rat. Both placental gonadotrophins induced ovulation, although the ova yield was significantly below the normal oestrous control. It is inferred from these findings that the ovarian sensitivity of androgen-sterilized rats is far below that of the normal animals despite the presence of numerous follicles in the ovaries of the persistent-oestrous rats.


1977 ◽  
Vol 19 (2) ◽  
pp. 211-216 ◽  
Author(s):  
George Fedak

Meiosis was studied in four 8-chromosome haploid plants produced from Wiebe's barley (2n = 14 + 2 centric fragments) (Hordeum vulgare L.) in which the extra chromosome had lost its telomeres and was one-third the length of a normal chromosome. Although secondary associations occurred, pairing between the normal and fragmented chromosome was rare despite the presence of homologous segments between them, indicating that the deletion of the telomeres restricted chromosome synapsis. A high bivalent frequency was attributed to a translocation between a long and short chromosome of the normal complement that persisted through the haploidization process.


Development ◽  
1996 ◽  
Vol 122 (2) ◽  
pp. 491-500 ◽  
Author(s):  
W.M. ElShamy ◽  
S. Linnarsson ◽  
K.F. Lee ◽  
R. Jaenisch ◽  
P. Ernfors

Postnatal homozygous neurotrophin-3 mutant mice display a loss of about half the sympathetic superior cervical ganglion (SCG) neurons (Ernfors, P., Lee, K.-F., Kucera, J. and Jaenisch, R. (1994a) Cell 77, 503–512; Farinas, I., Jones, K. R., Backus, C., Wang, X. Y. and Reichardt, L. F. (1994) Nature 369, 658–661). We found that this loss is caused by excessive apoptosis of sympathetic neuroblasts leading to a failure to generate a normal number of neurons during neurogenesis. NT-3 was also found to be required postnatally. In Nt-3−/− mice, sympathetic fibers failed to invade pineal gland and external ear postnatally; whereas other targets of the external and internal carotid nerves, including the submandibular gland and the iris, displayed a normal complement of sympathetic innervation. Sympathetic fibers of mice carrying one functional copy of the Nt-3 gene (Nt-3+/− mice) invaded the pineal gland, but failed to branch and form a ground plexus. Cultured neonatal sympathetic neurons responded to NT-3 by neurite outgrowth and mRNA upregulation of the NT-3 receptor, trkC. Exogenously administered NT-3 promoted sympathetic growth and rescued the sympathetic target deficit of the mutant mice. We conclude that NT-3 is required for the survival of sympathetic neuroblasts during neurogenesis and for sympathetic innervation and branching in specific targets after birth.


1988 ◽  
Vol 89 (3) ◽  
pp. 405-413
Author(s):  
J. Overton

Chick corneal epithelium takes on its mature conformation between 11 and 16 days of incubation. Earlier work has shown that desmosome frequency increases during this period, reaching its highest rate at 15 1/2 days. In the present report aggregation rates of cells from embryos of 11 days and those of 15 1/2 days are compared. Younger cells, which form fewer desmosomes, aggregate at a more moderate rate than older cells. In addition, younger cells bind less concanavalin A (ConA) than older cells. To determine if increase in ConA binding could be related to these cellular responses, aggregating cells were exposed to endoglycosidase H (EndoH) and to deoxymannojirimycin. This treatment should permit comparison of the response of cells that have a normal complement of N-linked oligosaccharides with those that have reduced high-mannose or complex type sugars. The effectiveness of EndoH under the conditions used was confirmed by failure of treated glycoprotein after separation by SDS-PAGE and electroblotting to bind ConA. Aggregation rates of both older and younger cells were unaffected, as measured by disapperance of single cells, though older cells formed somewhat smaller aggregates at the highest dosage used. Desmosome formation was markedly reduced in the presence of the enzyme, even in the absence of other changes in the fine structure. At the highest dose of the enzyme the fine structure of older but not younger cells showed indications of blockage of transport. Deoxymannojirimycin appears to cause a build-up of high-mannose groups, since treated cells showed increased incorporation of [3H]mannose.(ABSTRACT TRUNCATED AT 250 WORDS)


2020 ◽  
Vol 22 (1) ◽  
Author(s):  
Alessandra Tesser ◽  
Luciana Martins de Carvalho ◽  
Paula Sandrin-Garcia ◽  
Alessia Pin ◽  
Serena Pastore ◽  
...  

2015 ◽  
Vol 14 (08) ◽  
pp. 1550129 ◽  
Author(s):  
Neha Makhijani ◽  
R. K. Sharma ◽  
J. B. Srivastava

Let 𝔽qD2N be the group algebra of D2N, the dihedral group of order 2N, over 𝔽q = GF (q). In this paper, we compute the order of the unitary subgroup of the group of units of 𝔽2kD2N with respect to the canonical involution ∗.


1982 ◽  
Vol 216 (1205) ◽  
pp. 415-426 ◽  

Freshly isolated ‘European’ algae phagocytosed by digestive cells of ‘European’ green hydra were distinguished from the pre-existing popu­lation of algae by prestaining with the fluorescent agent Calcofluor White. Only a small number of phagocytosed ‘European’ algae or algae cultured from Paramecium bursaria avoided lysosomal degradation and were transported to the cell base in symbiotic digestive cells, although in aposymbionts up to 50% of phagocytosed algae were transported. Degradation of almost all phagocytosed algae also occurred in digestive cells of hydra containing only half the normal complement of algae, and in those of hydra symbiotic with algae cultured from Paramecium . The presence of algae at the bases of digestive cells appears to negate the mechanism by which potentially symbiotic algae normally avoid lysosomal attack. This protects the host cell and its symbionts from invasion by ‘foreign’ algae and suggests that once established the green hydra symbiosis is conservative in nature.


1991 ◽  
Vol 69 (11) ◽  
pp. 1769-1773 ◽  
Author(s):  
H. A. Tissenbaum ◽  
D. J. Parry

The tibialis anterior (TA) muscle in one leg of normal (C57BL) and dystrophic (dy2j) mice was partially denervated by resection of a part of the lateral popliteal nerve. Two months later the muscle was injected with horseradish peroxidase to permit visualization of the motorneurons that survived. Partial denervation in both C57 and dy2j mice resulted in reduction of the number of motorneurons that supplied the muscle to approximately one-half the normal complement. The surviving motorneurons were found to be significantly larger (about 25%) than their contralateral counterparts. This condition persisted up to 18 months and is not considered to be a transient response to the trauma associated with the partial denervation. When the size of the target tissue was also reduced by extirpation of one-half of TA together with partial denervation, motorneuron size was not found to increase. It is suggested that the increase in size is a response to the metabolic demands placed upon the motorneuron by an increase in the size of the motor unit.Key words: mouse, tibialis anterior muscle, partial denervation, motorneuron size.


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