scholarly journals Facemask Use During High Intensity Interval Exercise in Temperate and Hot Environments

2021 ◽  
Vol Publish Ahead of Print ◽  
Author(s):  
Andrew Wells ◽  
Zachary Fennel ◽  
Jeremy Ducharme ◽  
Abdulaziz Masoud ◽  
Jonathan Houck ◽  
...  
2021 ◽  
pp. 1-23
Author(s):  
Kosar Valaei ◽  
Javad Mehrabani ◽  
Alexei Wong

Abstract L-citrulline (L-Cit) is a nonessential amino acid that stimulates nitric oxide (NO) production and improves exercise performance by reducing muscle damage indices; however, the direct benefits of L-Cit on antioxidant markers are unclear. The aim of this study was to examine antioxidant responses to high-intensity interval exercise following acute L-Cit supplementation. Nine young men (21 ± 1 years) participated in a double-blind crossover study in which they received 12 g of L-Cit and placebo (PL) an hour prior to high-intensity interval exercise on two occasions, separated by a seven-day washout period. Blood samples were obtained before (PRE), immediately after (IP), 10 (10P), and 30 min after exercise (30P) from the cubital vein using standard procedures. Serum concentrations of superoxide dismutase (SOD), glutathione peroxidase (GPx), catalase (CAT), and NO metabolites (NOx) were measured. The exercise protocol significantly elevated SOD (p = 0.01) and GPx (p = 0.048) from PRE to 10P in the L-Cit group with greater changes than the PL group. CAT concentrations increased IP (p = 0.014) and remained elevated at 10P (p = 0.03) and 30P (p = 0.015) in both the L-Cit and PL conditions. NOx concentrations increased IP (p = 0.05) in the L-Cit group with greater changes than PL group in PRE to IP, PRE to 10P, and PRE to 30P (p < 0.05). Our data indicate that L-Cit supplementation (single 12 g dose pre-exercise) induces improvements in antioxidant markers following a session of high-intensity interval exercise in young men.


2017 ◽  
Vol 31 (8) ◽  
pp. 2263-2269 ◽  
Author(s):  
Teresa C.B. Dantas ◽  
Luiz F. Farias Junior ◽  
Danniel T. Frazão ◽  
Paulo H.M. Silva ◽  
Altieres E. Sousa Junior ◽  
...  

Author(s):  
Bruno Ribeiro Ramalho Oliveira ◽  
Tony Meireles Santos ◽  
Renato de Carvalho Guerreiro ◽  
Igor D’Amorim ◽  
Eduardo Lattari ◽  
...  

2018 ◽  
Vol 243 (14) ◽  
pp. 1153-1160 ◽  
Author(s):  
Alexandra L Rodriguez ◽  
Michael Whitehurst ◽  
Brandon G Fico ◽  
Katelyn M Dodge ◽  
Peter J Ferrandi ◽  
...  

Obesity may attenuate the expression of brain-derived neurotrophic factor (BDNF), thereby increasing the risk of cognitive dysfunction. High-intensity interval exercise (HIIE) has been shown to be as or more effective than continuous moderate-intensity exercise (CME) in promoting the expression of BDNF in normal-weight individuals. Therefore, the primary purpose of this study was to examine whether or not acute HIIE could be utilized as a practical model to explore the BDNF response in obese versus normal-weight subjects when compared to acute CME. The potential relationship of exercise-induced BDNF with blood lactate and cortisol was also examined. Twelve male subjects (six obese and six normal-weight) participated in a counterbalanced and caloric equated experiment: HIIE (30 min, 4 intervals of 4 min at 80%–90% of VO2max with 3 min of active recovery at 50–60% VO2max) and CME (38 min at 50%–60% VO2max). Blood samples were collected prior to, immediately following exercise, and 1 h into recovery for measurements of serum BDNF, blood lactate, and plasma cortisol. Our results showed that the BDNF response to acute HIIE was greater than CME in obese subjects when compared to normal-weight subjects. Similarly, although acute HIIE induced greater blood lactate and plasma cortisol levels than CME, obese subjects produced less blood lactate, but no difference in cortisol than normal-weight subjects. These findings suggest that acute HIIE may be a more effective protocol to upregulate BDNF expression in an obese population, independent of increased lactate and cortisol levels. Impact statement High-intensity interval exercise (HIIE) has been shown to be a time-efficient exercise strategy that provides similar or superior physiological benefits as traditional continuous moderate-intensity exercise (CME). Our previous study demonstrated an equivalent elevation on the BDNF response in both obese and normal-weight individuals following 30 min of acute CME. To discover a time-efficient exercise strategy to improve brain health in an obese population, the present study found that obese individuals elicit a greater level of BDNF following acute HIIE versus CME than normal-weight individuals. These findings indicate that acute HIIE may be an effective strategy to upregulate BDNF expression in obese individuals.


2015 ◽  
Vol 47 ◽  
pp. 892
Author(s):  
Abraham S. Chiu ◽  
Peter Horvath ◽  
Goutham Ganesan ◽  
Robert V. Warren ◽  
Frank Zaldivar ◽  
...  

2021 ◽  
Vol 40 (10) ◽  
pp. 797-799
Author(s):  
Raphael José Perrier-Melo ◽  
Antônio Henrique Germano-Soares ◽  
Aline Freitas Brito ◽  
Iago Vilela Dantas ◽  
Manoel da Cunha Costa

2021 ◽  
Author(s):  
yanping yang ◽  
Junqiang Qiu ◽  
Mengyue Wang ◽  
Lin Feng ◽  
Dan Luo ◽  
...  

Abstract Background: The effects of pyruvate on metabolic acidosis and oxidative metabolism had been studied. The ability to attenuate acidosis and improve oxidative system contribution are critical to the performance of team sport athletes during perform multiple high-intensity exercise over a limited period of time. This study aimed to investigate the impact of pyruvate supplementation on energy metabolism and metabolic acidosis during high-intensity interval exercise (HIIE), as well as to evaluate its role on repeated sprint exercise (RSE) performance.Methods: 14 well-trained male college soccer athletes (age: 20 ± 2 years, body fat: 13.11 ± 3.50 %) were studied in a randomized, double-blind, cross-over study. The participants ingested either 0.1g/kg/d of pyruvate or a placebo for 1-week. Metabolic acidosis was induced by HIIE after the supplement period, and RSE ability in the acidosis state was assessed. Venous blood pH, bicarbonate (HCO3-) and base excess (BE) were measured at baseline, pre-HIIE, post-HIIE, pre-RSE and post-RSE. Finger-stick blood lactate were collected at baseline, immediately after each bout of HIIE and 3, 5, 7, 10 min after HIIE. The energy systems contribution during HIIE were estimated. Results: Blood pH, HCO3- and BE were significantly lower than baseline after HIIE (p < 0.01) in both pyruvate group (PYR) and placebo group (PLA). Compared to PLA, the blood pH, HCO3- and BE were significantly improved in PYR at pre-HIIE (p < 0.01), post-HIIE (p < 0.01) and pre-RSE (p < 0.01). Furthermore, blood BE remained higher in PYR than PLA till end of RSE (p < 0.05). The contribution of oxidative system in the fourth bout of HIIE was higher in PYR than PLA (p < 0.05). In PLA, the ratio of total anaerobic energy contribution during HIIE was higher than that of aerobic (oxidative) (p < 0.01), but not in PYR (p > 0.05). Relative peak power (RPP) of first, fifth sprint, relative average power (RAP) of fifth sprint, the average of RPP and RAP during RSE were significantly improved in PYR compared with PLA (p < 0.05). While no significant changes in the PD% of each bout (p > 0.05) or average PD% (p > 0.05) were observed between the two groups. Conclusion: Pyruvate supplementation for 1-week enhances oxidative system energy contribution and buffers metabolic acidosis during HIIE, and improves RSE performance in acidosis.


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