scholarly journals Modelling song popularity as a contagious process

Author(s):  
Dora P. Rosati ◽  
Matthew H. Woolhouse ◽  
Benjamin M. Bolker ◽  
David J. D. Earn

Popular songs are often said to be ‘contagious’, ‘infectious’ or ‘viral’. We find that download count time series for many popular songs resemble infectious disease epidemic curves. This paper suggests infectious disease transmission models could help clarify mechanisms that contribute to the ‘spread’ of song preferences and how these mechanisms underlie song popularity. We analysed data from MixRadio, comprising song downloads through Nokia cell phones in Great Britain from 2007 to 2014. We compared the ability of the standard susceptible–infectious–recovered (SIR) epidemic model and a phenomenological (spline) model to fit download time series of popular songs. We fitted these same models to simulated epidemic time series generated by the SIR model. Song downloads are captured better by the SIR model, to the same extent that actual SIR simulations are fitted better by the SIR model than by splines. This suggests that the social processes underlying song popularity are similar to those that drive infectious disease transmission. We draw conclusions about song popularity within specific genres based on estimated SIR parameters. In particular, we argue that faster spread of preferences for Electronica songs may reflect stronger connectivity of the ‘susceptible community’, compared with the larger and broader community that listens to more common genres.

2020 ◽  
Author(s):  
Cory Simon

<div>Mathematical models of the dynamics of infectious disease transmission are used to forecast epidemics and assess mitigation strategies. We reveal that the classic Susceptible-Infectious-Recovered (SIR) epidemic model resembles a dynamic model of a batch reactor carrying out an autocatalytic reaction with catalyst deactivation. This analogy between disease transmission and chemical reactions enables the cross-pollination of ideas between epidemic and chemical kinetic modeling.</div>


2020 ◽  
Author(s):  
Cory Simon

<div>Mathematical models of the dynamics of infectious disease transmission are used to forecast epidemics and assess mitigation strategies. We reveal that the classic Susceptible-Infectious-Recovered (SIR) epidemic model resembles a dynamic model of a batch reactor carrying out an autocatalytic reaction with catalyst deactivation. This analogy between disease transmission and chemical reactions allows chemists and chemical engineers to peer into dynamic models of infectious disease transmission and gives insights into the kinetics of autocatalytic reactions.</div>


2020 ◽  
Author(s):  
Cory Simon

<div>Mathematical models of the dynamics of infectious disease transmission are used to forecast epidemics and assess mitigation strategies. We reveal that the classic Susceptible-Infectious-Recovered (SIR) epidemic model resembles a dynamic model of a batch reactor carrying out an autocatalytic reaction with catalyst deactivation. This analogy between disease transmission and chemical reactions enables the cross-pollination of ideas between epidemic and chemical kinetic modeling.</div>


2020 ◽  
Vol 2 ◽  
pp. e14 ◽  
Author(s):  
Cory M. Simon

Mathematical models of the dynamics of infectious disease transmission are used to forecast epidemics and assess mitigation strategies. In this article, we highlight the analogy between the dynamics of disease transmission and chemical reaction kinetics while providing an exposition on the classic Susceptible–Infectious–Removed (SIR) epidemic model. Particularly, the SIR model resembles a dynamic model of a batch reactor carrying out an autocatalytic reaction with catalyst deactivation. This analogy between disease transmission and chemical reaction enables the exchange of ideas between epidemic and chemical kinetic modeling communities.


2020 ◽  
Author(s):  
Angela Maria Cadavid Restrepo ◽  
Luis Furuya-Kanamori ◽  
Helen Mayfield ◽  
Eric J. Nilles ◽  
Colleen L. Lau

2012 ◽  
Vol 54 (1-2) ◽  
pp. 23-36 ◽  
Author(s):  
E. K. WATERS ◽  
H. S. SIDHU ◽  
G. N. MERCER

AbstractPatchy or divided populations can be important to infectious disease transmission. We first show that Lloyd’s mean crowding index, an index of patchiness from ecology, appears as a term in simple deterministic epidemic models of the SIR type. Using these models, we demonstrate that the rate of movement between patches is crucial for epidemic dynamics. In particular, there is a relationship between epidemic final size and epidemic duration in patchy habitats: controlling inter-patch movement will reduce epidemic duration, but also final size. This suggests that a strategy of quarantining infected areas during the initial phases of a virulent epidemic might reduce epidemic duration, but leave the population vulnerable to future epidemics by inhibiting the development of herd immunity.


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