Heart Rate Detection Technology for Video Image and Its Application in Postoperative Evaluation of Free Skin Flap Surgery

Author(s):  
Chao-Chi Wu ◽  
Chun-Chieh Hsiao ◽  
Chao-Hua Lin ◽  
Chien-Hsing Wang ◽  
Ren-Guey Lee
PLoS ONE ◽  
2013 ◽  
Vol 8 (7) ◽  
pp. e65833 ◽  
Author(s):  
David P. de Sena ◽  
Daniela D. Fabricio ◽  
Maria Helena I. Lopes ◽  
Vinicius D. da Silva

2019 ◽  
Vol 14 (1) ◽  
Author(s):  
Xu Gao ◽  
Hailei Yin ◽  
Jixia Sun

Abstract Objectives By observing the infection and soft tissue defect on the wound surface of the foot and ankle, this paper attempts to explore the effect of preoperative irrigation and vacuum sealing drainage with antibiotic-containing drainage fluid (abPI-VSD) on the bacterial quantity and the local inflammatory response at the flap, and further to provide a basis for applying this technique before a reconstructive skin flap surgery of foot and ankle wounds. Methods Seventy-five patients were randomly divided into two groups, and all surgeries were done by one physician. The flap reconstructions were done to 31 cases with the abPI-VSD being used (group A); the flap reconstructions were done to the rest 44 cases after wound cleaning using antibiotic irrigation solution without the use of the abPI-VSD (group B). Quantitative bacteriology was made to group A before and after the use of abPI-VSD; quantitative bacteriology was made to group B before and after wound cleaning. Then, the reconstructive skin flap surgery was done. After the surgeries, the time of local inflammatory response at the flap in both groups were recorded. The measured bacterial quantity was evaluated in logarithm and by t test. Results The bacterial quantity was 3.2 ± 1.9 × 107 cfu/g in group A before the use of abPI-VSD and 2.3 ± 2.0 × 107 in group B (P > 0.05) before debridement. The bacterial quantity was 1.2 ± 2.0 × 104 cfu/g in group A after abPI-VSD and was 2.9 ± 4.0 × 106 in group B after wound cleaning (P < 0.05). The time of postoperative inflammatory response in the flap was 8 ± 2.5 days in group A and 13 ± 3.4 days in group B (P < 0.05). Conclusions abPI-VSD can distinctly reduce the bacterial quantity on the surface of the wound, provide a good condition of tissue bed for the flap reconstruction, and effectively control the local inflammatory response at the flap and hence improve the survival quality of the flap.


Author(s):  
Mitchell A. Stotland ◽  
Carolyn L. Kerrigan
Keyword(s):  

2005 ◽  
Vol 31 (9) ◽  
pp. 1107-1111 ◽  
Author(s):  
MATS BJELLERUP
Keyword(s):  

2009 ◽  
Vol 25 (06) ◽  
pp. 361-367 ◽  
Author(s):  
Shi-Min Chang ◽  
Chun-Lin Hou ◽  
Da-Chuan Xu

2018 ◽  
Vol 10 (4) ◽  
pp. 113-118
Author(s):  
Zeinab Javid ◽  
Abdolreza Norouzy ◽  
Zahra Bangaleh ◽  
Mahdi Shadnoush

Abstract Introduction: Skin flap application in the clinical practice is restricted due to the ischemic damage and flap necrosis. Rosmarinus oil has been shown to improve a skin flap survival. In the present work we studied the role of mammalian target of rapamycin (mTOR) signaling on rosmarinus-induced flap protection. Methods: A flap surgery was performed on Sprauge-Dawley rats (8 cm in by 3 cm). A week before and a week after the surgery the flaps were treated with topical rosmarinus oil (twice per day). Rapamycin (m-TOR inhibitor) was administered 30 minutes before the flap surgery in rosmarinus-treated or not treated groups. A week after the surgery the malondialdehyde (MDA) contents, myeloperoxidase (MPO) and superoxide dismutase (SOD) activities, expression of Bax, Bcl-2, mTOR and p-mTOR were measured in the flap tissue. Results: Topical application of the rosmarinus increased the flap survival (p<0.05), anti-oxidative enzyme activity (SOD, p<0.05) and anti-apoptotic protein Bcl-2 expression. Rosmarinus treatment decreased the flap MDA content, MPO activity, and pro-apoptotic protein Bax expression (p<0.05). Rosmarinus topical application did not change mTOR expression and phosphorylation in the flap tissue. Expression of p-mTOR in rosmarinus treated group was suppressed by rapamycin pre-treatment, which also abolished rosmarinus effects on the flap survival (p<0.05). Conclusion: These data suggested p-mTOR dependent mechanism in rosmarinus-induced flap survival.


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