Novel hybrid drilling protocol: evaluation for the implant healing - thermal changes, crestal bone loss, and bone-to-implant contact

2014 ◽  
Vol 26 (7) ◽  
pp. 753-760 ◽  
Author(s):  
José Luis Calvo-Guirado ◽  
Jorge Delgado-Peña ◽  
Jose E. Maté-Sánchez ◽  
Javier Mareque Bueno ◽  
Rafael Arcesio Delgado-Ruiz ◽  
...  
Keyword(s):  
Bone Loss ◽  
Crestal Bone Loss ◽  
Thermal Changes ◽  
Bone To Implant Contact ◽  
Protocol Evaluation

Survival of Short and Ultra-Short Locking-Taper Implants Supporting Single Crowns in the Posterior Mandible: A 3-Year Retrospective Study

2020 ◽  
Vol 46 (4) ◽  
pp. 396-406 ◽  
Author(s):  
Giorgio Lombardo ◽  
Annarita Signoriello ◽  
Miguel Simancas-Pallares ◽  
Mauro Marincola ◽  
Pier Francesco Nocini
Keyword(s):  
Retrospective Study ◽  
Bone Loss ◽  
Contact Point ◽  
Implant Survival ◽  
Point Position ◽  
Crestal Bone Loss ◽  
Short Implants ◽  
Single Crown ◽  
Bone To Implant Contact ◽  
Single Crowns

The purpose of this retrospective study was to determine survival and peri-implant marginal bone loss of short and ultra-short implants placed in the posterior mandible. A total of 98 patients received 201 locking-taper implants between January 2014 and January 2015. Implants were placed with a 2-stage approach and restored with single crowns. Clinical and radiographic examinations were performed at 3-year recall appointments. At that time, the proportion of implant survival by length, and variations of crestal bone levels (mean crestal bone loss and mean apical shift of the “first bone-to-implant contact point” position) were assessed. Significance level was set at 0.05. The total number of implants examined 36 months after loading included: 71 implants, 8.0 mm in length; 82 implants, 6.0 mm in length; and 48 implants, 5.0 mm in length. Five implants failed. The overall proportion of survival was 97.51%, with 98.59% for the 8.0-mm implants, 97.56% for the 6.0-mm implants, and 95.83% for the 5.0-mm implants. No statistically significant differences were found among the groups regarding implant survival (P = .73), mean crestal bone loss (P = .31), or mean apical shift of the “first bone-to-implant contact point” position (P = .36). Single-crown short and ultra-short implants may offer predictable outcomes in the atrophic posterior mandibular regions, though further investigations with longer follow-up evaluations are necessary to validate our results.

scholarly journals Influence of Implant Neck Design on Peri-Implant Tissue Dimensions: A Comparative Study in Dogs

Materials ◽  
2018 ◽  
Vol 11 (10) ◽  
pp. 2007 ◽  
Author(s):  
José Calvo-Guirado ◽  
Raúl Jiménez-Soto ◽  
Carlos Pérez Albacete-Martínez ◽  
Manuel Fernández-Domínguez ◽  
Sérgio Gehrke ◽  
...  
Keyword(s):  
Bone Loss ◽  
Soft Tissues ◽  
Surface Group ◽  
Crestal Bone Loss ◽  
Bone Preservation ◽  
Group A ◽  
Bone To Implant Contact ◽  
Group B ◽  
Cervical Area

This in vivo study assessed (hard and soft) peri-implant tissue remodeling around implants with micro-ring and open-thread neck designs placed in a dog model. Twenty histological sections corresponding to four different implant designs that were placed in America Foxhound dogs were obtained from previous studies. All the implants had been placed under identical conditions and were divided into four groups: Group A, micro-rings on implant neck plus 0.5 mm refined surface; Group B, micro-rings on implant neck; Group C, open-thread neck; and, Group D, double-spiral neck. Eight weeks after surgery, the integrated implants were removed and processed for histological examination. Crestal bone loss and bone-to-implant contact was greater for micro-ring necks than open-thread necks. Soft tissues showed significant differences on both buccal and lingual aspects, so that the distance from peri-implant mucosa to the apical portion of the barrier epithelium was smaller in the micro-ring groups. So, in spite of generating greater bone-to-implant contact, implants with micro rings produced more bone loss than open-thread implants. Moreover, the outcomes that were obtained IPX implants smooth neck design produced less bone loss in the cervical area, following by Facility implants when compared with the other open thread and microthreaded implant designs. Implant thread design can influence on bone remodeling in the cervical area, related to bundle bone preservation.

scholarly journals Role of Melatonin in Bone Remodeling Around Titanium Dental Implants: Meta-Analysis

Coatings ◽  
2021 ◽  
Vol 11 (3) ◽  
pp. 271
Author(s):  
Nansi López-Valverde ◽  
Beatriz Pardal-Peláez ◽  
Antonio López-Valverde ◽  
Juan Manuel Ramírez
Keyword(s):  
Nervous System ◽  
Bone Loss ◽  
Bone Remodeling ◽  
Random Effects ◽  
Meta Analysis ◽  
Titanium Implants ◽  
Crestal Bone Loss ◽  
Bone To Implant Contact ◽  
Titanium Dental Implants

The theory, known as the “brain-bone axis” theory, involves the central nervous system in bone remodeling. The alteration of the nervous system could lead to abnormal bone remodeling. Melatonin produced by the pineal gland is a hormone that is characterized by its antioxidant properties. The aim of this meta-analysis was to examine the role of melatonin in the growth of new bone around titanium dental implants in vivo. A manual search of the PubMed and Web of Science databases was conducted to identify scientific studies published until November 2020. We included randomized clinical trials (RCTs) and animal studies where melatonin was used with titanium implants. Fourteen studies met the inclusion criteria. Quality was assessed using the Jadad scale and SYRCLE's risk of bias tool. Our meta-analysis revealed that the use of melatonin during implant placement improves bone-to-implant contact percentages in animals (difference of means, random effects: 9.59 [95% CI: 5.53−13.65]), reducing crestal bone loss in humans (difference in means, random effects: -0.55 [95% CI: 1.10–0.00]). In animals, titanium implants using melatonin increase bone-to-implant contact surface 2−6 weeks after their placement and reduce crestal bone loss in humans following six months. The results of this meta-analysis should be taken with caution, due to the small samples and the large heterogeneity among studies.

scholarly journals Maryland Bridge to prevent crestal bone loss during peri-implant healing phases- 6-month RCT

2018 ◽  
Vol 29 ◽  
pp. 112-112
Author(s):  
Fausto Zamparini ◽  
Arash Azizi ◽  
Stefano Chersoni ◽  
Andrea Spinelli ◽  
Maria Giovanna Gandolfi ◽  
...  
Keyword(s):  
Bone Loss ◽  
Crestal Bone Loss

scholarly journals Peri-Implant Crestal Bone Loss: A Putative Mechanism

2012 ◽  
Vol 2012 ◽  
pp. 1-14 ◽  
Author(s):  
Yuko Ujiie ◽  
Reynaldo Todescan ◽  
John E. Davies
Keyword(s):  
Bone Loss ◽  
Stromal Cells ◽  
Cell Types ◽  
Osteoclast Formation ◽  
Rt Pcr ◽  
Crestal Bone Loss ◽  
Bacterial Endotoxins ◽  
Immunological Mechanisms ◽  
Study Support ◽  
Putative Mechanism

Purpose. The immunological mechanisms of peri-implant crestal bone loss have, hitherto, not been elucidated. We hypothesized that bacterial products from the microgap cause upregulation of cytokines in otherwise healthy peri-implant cells, which results in osteoclast formation and, ultimately, in bone resorption.Materials and Methods. We used RT-PCR and ELISA to assay mediators of osteoclastogenesis in rat and human macrophages (r-and hMO); bone marrow derived stromal cells (r-and hBMCs); and human gingival fibroblasts (hGF)—with or without stimulation by LPS. TRAP positive multinucleate cells were assessed for their resorptive ability.Results. We show that IL-1α, IL-1β, and IL-6 were expressed by all examined cell types, and TNF-αwas upregulated in hGF. Secretion of IL-1αand IL-1βproteins was stimulated in hMO by LPS, and IL-6 protein secretion was highly stimulated in hBMCs and hGF. Both LPS and RANKL stimulated macrophages to form osteoclast-like TRAP positive cells, which resorbed calcium phosphate substrates.Conclusion. Taken together, the results of our study support the hypothesis that bacterial endotoxins upregulate enhanced mediators of osteoclastogenesis in resident cells found in the healthy peri-implant compartment and that the local synergistic action of cytokines secreted by such cells results in the genesis of resorptively active osteoclasts.

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