Post-traumatic stress disorder symptoms in first-time myocardial infarction patients: roles of attachment and alexithymia

2015 ◽  
Vol 71 (11) ◽  
pp. 2575-2584 ◽  
Author(s):  
Wen Gao ◽  
Jing Zhao ◽  
Yang Li ◽  
Feng-Lin Cao
Keyword(s):  
Myocardial Infarction ◽  
Post Traumatic Stress Disorder ◽  
Traumatic Stress ◽  
Stress Disorder ◽  
Post Traumatic Stress ◽  
Post Traumatic ◽  
First Time

Post-Traumatic Stress Disorder in Birthmothers

1993 ◽  
Vol 17 (2) ◽  
pp. 30-32 ◽  
Author(s):  
Sue Wells
Keyword(s):  
International Society ◽  
Post Traumatic Stress Disorder ◽  
Traumatic Stress ◽  
Stress Disorder ◽  
World Conference ◽  
Post Traumatic Stress ◽  
Stress Studies ◽  
The World ◽  
Post Traumatic ◽  
First Time

Adoption was included for the first time at the world conference of the International Society for Traumatic Stress Studies last Easter in Amsterdam. Sue Wells presents extracts from her presentation to the conference, based upon her own research as a birthmother.

The Neuropsychiatry of Post-traumatic Stress Disorder

1988 ◽  
Vol 152 (2) ◽  
pp. 164-173 ◽  
Author(s):  
I. P. Burges Watson ◽  
L. Hoffman ◽  
G. V. Wilson
Keyword(s):  
Post Traumatic Stress Disorder ◽  
Traumatic Stress ◽  
Stress Disorder ◽  
Stressful Events ◽  
Psychosocial Issues ◽  
Post Traumatic Stress ◽  
Biological Perspective ◽  
Post Traumatic ◽  
Psychosocial Approaches ◽  
First Time

The publication of DSM-III introduced the diagnosis Post-Traumatic Stress Disorder (PTSD), thus providing, for the first time, a framework for studying the consequences of extremely stressful events. Previously, traumatic neuroses had attracted a wide variety of labels – as wide as the experiences that produced them. Competing explanations in psychological and biological terms have characterised the approach to these disorders, and social and legal issues have added to the confusion. In recent years, psychosocial issues have tended to dominate the literature in relation to PTSD. While acknowledging the importance of such phenomenological and psychosocial approaches, this paper seeks to redress the balance by focusing on a biological perspective.

scholarly journals Characteristics of Heart Rate Variability in War Veterans with Post-Traumatic Stress Disorder after Myocardial Infarction

2007 ◽  
Vol 172 (11) ◽  
pp. 1190-1193 ◽  
Author(s):  
Nenad Lakusic ◽  
Krunoslav Fuckar ◽  
Darija Mahovic ◽  
Dusko Cerovec ◽  
Marcel Majsec ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Heart Rate ◽  
Heart Rate Variability ◽  
Post Traumatic Stress Disorder ◽  
Traumatic Stress ◽  
Stress Disorder ◽  
Post Traumatic Stress ◽  
War Veterans ◽  
Post Traumatic

Physical fatigue and the morfofunctional state of myocardium in experimental chronic stress

2019 ◽  
Vol 485 (2) ◽  
pp. 247-250
Author(s):  
M. V. Kondashevskaya ◽  
V. E. Tseylikman ◽  
M. V. Komelkova ◽  
M. S. Lapshin ◽  
A. P. Sarapultsev ◽  
...  
Keyword(s):  
Chronic Stress ◽  
Post Traumatic Stress Disorder ◽  
Traumatic Stress ◽  
Stress Disorder ◽  
Male Rats ◽  
Structural Components ◽  
Post Traumatic Stress ◽  
Post Traumatic ◽  
First Time ◽  
The Relationship

The relationship between skeletal muscle fatigue and morpho-functional alterations in the myocardium was analyzed for the first time in Wistar male rats exposed to chronic stress. Post traumatic stress disorder (PTSD) was associated with signs of increased oxidative stress, which apparently induced the changes in cardiomyocyte structural components and the acceleration of skeletal and muscular fatigue.

scholarly journals Up-Regulation of αCaMKII Impairs Cued Fear Extinction and NMDAR-Dependent LTD in the Lateral Amygdala

2020 ◽  
Author(s):  
Shuming An ◽  
Jiayue Wang ◽  
Xuliang Zhang ◽  
Yanhong Duan ◽  
Junyan Lv ◽  
...  
Keyword(s):  
Normal Level ◽  
Post Traumatic Stress Disorder ◽  
Traumatic Stress ◽  
Stress Disorder ◽  
Fear Extinction ◽  
Post Traumatic Stress ◽  
Lateral Amygdala ◽  
Molecular Determinant ◽  
Post Traumatic ◽  
First Time

ABSTRACTImpaired fear extinction is one of the hallmark symptoms of post-traumatic stress disorder (PTSD). The roles of αCaMKII have been not extensively studied in fear extinction and LTD. Here, we found PTSD susceptible mice exhibited significant up-regulation of αCaMKII in the lateral amygdala (LA). Consistently, increasing αCaMKII in LA profoundly not only caused PTSD-like symptoms such as impaired fear extinction and anxiety-like behaviors, but also attenuated NMDAR-dependent LTD at thalamo-LA synapses, reduced GluA1-Ser845/Ser831 dephosphorylation and AMPAR internalization. Suppressing the elevated αCaMKII to normal level could completely reverse both PTSD-like symptoms and the impairments in LTD, GluA1-Ser845/Ser831 dephosphorylation and AMPAR internalization. Intriguingly, deficits in AMPAR internalization and GluA1-Ser845/Ser831 dephosphorylation were detected not only after impaired fear extinction, but also after attenuated LTD Our results demonstrate for the first time GluA1-Ser845/Ser831 dephosphorylation and AMPAR internalization are molecular links between LTD and fear extinction, and suggest αCaMKII may be a potential molecular determinant of PTSD.

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