Expression pattern of HIF1alpha and vasohibins during follicle maturation and corpus luteum function in the bovine ovary

2016 ◽  
Vol 52 (1) ◽  
pp. 130-139 ◽  
Author(s):  
B Berisha ◽  
D Schams ◽  
D Rodler ◽  
F Sinowatz ◽  
MW Pfaffl
1934 ◽  
Vol 30 (6) ◽  
pp. 636-636
Author(s):  
P. Manenkov

The author points out that uterine bleeding can be caused either by weakness of uterine muscles caused by tumor, inflammation, etc., or by disturbances in ovarian processes (follicle maturation, ovulation, corpus luteum phase, etc.), -The author points out that this may be caused by weakness of the uterine muscles due to a tumor, inflammation, etc.; by an increased menstruation; by a disturbance of the ovarian processes (follicle maturation, ovulation and corpus luteum phase), leading to a shortened cycle time with a shortened corpus luteum phase; or finally by carcinomas, polyps and cystic glandular hyperplasia, which cause abnormal bleeding.


2017 ◽  
Vol 41 (5) ◽  
pp. S37
Author(s):  
Moafaq Samir ◽  
Dareen Mattar ◽  
Pillip Knight
Keyword(s):  

2007 ◽  
Vol 76 (5) ◽  
pp. 749-758 ◽  
Author(s):  
Tatiana Kisliouk ◽  
Aharon Friedman ◽  
Eyal Klipper ◽  
Qun-Yong Zhou ◽  
Dieter Schams ◽  
...  

Reproduction ◽  
2000 ◽  
pp. 243-249 ◽  
Author(s):  
I Reibiger ◽  
K Spanel-Borowski

The bovine ovary contains a considerable number of leucocytes which can be located with an antibody against the CD18 molecule. In the present study, subtyping and cell counting were carried out on histological sections stained with Sirius red for eosinophils and with toluidine blue for mast cells. The CD18(+) cells were identified immunohistologically. Eosinophils and mast cells contributed considerably to the CD18(+) pool. The number of eosinophils in the corpus luteum increased rapidly in early development to approximately 90% of the CD18(+) cells, and decreased to 30% during secretion and to 10% during regression. Mast cells were not detectable in the follicles, the corpus luteum and the periphery of the cortex, but were observed in the interstitial cortical stroma and the medulla. The number of mast cells in these regions, which corresponded to 60-76% of the CD18(+) cells, did not change significantly throughout the oestrous cycle. It is concluded that eosinophils are selectively recruited at the periovulatory period and that mast cells are unevenly distributed.


Reproduction ◽  
2003 ◽  
pp. 791-800 ◽  
Author(s):  
NH Machell ◽  
R Farookhi

Cadherins, a family of Ca(2+)-dependent cell adhesion molecules, play an important role in ovarian tissue remodelling processes. The aim of this study was to examine the expression pattern of E- and N-cadherin in rat preovulatory follicles, luteinizing follicles and corpora lutea. Immature female rats were treated with equine chorionic gonadotrophin (eCG) to promote preovulatory follicle development. At 48 h after eCG treatment, the rats were injected with an ovulatory dose of hCG. Ovaries were analysed by western blot analysis and immunofluorescence for E- and N-cadherin expression at 48 h after eCG injection, and at 24 and 72 h after hCG injection. Ovaries of cyclic adult rats were examined to assess whether the changes in the expression pattern of cadherin were in agreement with those of the gonadotrophin-treated rats. Finally, expression of E-cadherin in luteinizing granulosa cells in vitro was assessed by RT-PCR and western blot analysis. Immunofluorescence results indicate that E-cadherin is expressed in the theca-interstial cells surrounding preovulatory follicles. N-cadherin expression is prominent in the membrana granulosa of these follicles. The initiation of luteinization with hCG leads to a decreased expression of N-cadherin in the membrana granulosa, whereas expression of E-cadherin starts within the luteinizing follicle. Both cadherins are prominently expressed in the fully formed corpus luteum at 72 h after hCG treatment. Immunofluorescence results revealed that the patterns of E- and N-cadherin expression in the gonadotrophin-treated rats were similar to those of the cyclic adult rats. Western blot analysis reflected similar changes for N-cadherin in the ovaries of both the cyclic adults and gonadotrophin-treated rats; however, they were different in E-cadherin expression. The expression of E-cadherin mRNA and protein was induced in vitro in luteinized granulosa cells. These results support the hypothesis that modulation of cadherin expression is an integral component of remodelling processes, including corpus luteum formation, in the ovary. The results also indicate that expression of E- and N-cadherin in granulosa-lutein cells appear to be under hormonal control.


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