scholarly journals The Influence of Social Support on the Lived Experiences of Spinal Cord Injured Sportsmen

2003 ◽  
Vol 17 (2) ◽  
pp. 135-156 ◽  
Author(s):  
Tim Rees ◽  
Brett Smith ◽  
Andrew C. Sparkes

This study draws upon life history data to investigate the influence of social support on the lives of 6 men who had acquired a spinal cord injury and become disabled through playing sport. Interviews were analyzed utilizing categorical-content analysis (Lieblich, Tuval-Mashiach, & Zilber, 1998). The participants experienced emotional, esteem, informational, and tangible support (Rees & Hardy, 2000) from various sources. Alongside the positive influence of social support, examples are shown of inappropriate or negatively-experienced support and where participants considered sport to be lacking. The spinal cord injured person is encouraged to be proactive in resourcing social support, but providers might also be taught to recognize the impact, either positively or negatively, that their giving support can have.

2013 ◽  
Vol 94 (10) ◽  
pp. e26
Author(s):  
Bryce Sutton ◽  
Lisa Ottomanelli ◽  
Eni N. Njoh ◽  
Scott Barnett ◽  
Lance Goetz

2009 ◽  
Vol 35 (3) ◽  
pp. 256-260
Author(s):  
Andrea Ponte Rocha ◽  
Sergio Ricardo Menezes Mateus ◽  
Thomas Anthony Horan ◽  
Paulo Sérgio Siebra Beraldo

The aim of the study was to evaluate the performance of sniff nasal inspiratory pressure (SNIP) and MIP in individuals with spinal cord injury. We evaluated 26 patients with spinal cord injury. Mean FVC in those with tetraplegia was 52 ± 19% of predicted, compared with 78 ± 23% of predicted in those with paraplegia (p < 0.05). In contrast, the percentage of predicted SNIP was lower in those with tetraplegia than in those with paraplegia (p < 0.05). In all participants, SNIP correlated significantly with the level of the injury (r = 0.489; 95% CI: 0.125-0.737). The impact that the greater discriminatory power of SNIP has on the diagnosis of impaired pulmonary function in spinal cord-injured patients should be investigated further.


2016 ◽  
Vol 21 (1) ◽  
pp. 35-55 ◽  
Author(s):  
Timothy Barrett

This article offers a consideration of the gendered emotion of “shame” within the context of the lived experiences of spinal cord injured men, using “found life histories” as a source material. Drawing upon the Bourdieusian concept of “hysteresis,” I theorize the emergence of a gendered disjuncture between incorporated expectations, values and assumptions, and the substantive enactments of masculinity socially available in the aftermath of spinal cord injury. Shame was often experienced within contexts characterized by “dangerous” social proximity to pathologized “others” (namely, disabled people and children) against which hegemonic masculinity is defined. I conclude the article by highlighting the particular limitations that Bourdieusian social theory can be used to identify in relation to individualized therapeutic interventions designed to encourage spinal cord injured men to adopt new understandings of masculinity.


2011 ◽  
Vol 110 (5) ◽  
pp. 1204-1210 ◽  
Author(s):  
Yun Chau Long ◽  
Emil Kostovski ◽  
Hanneke Boon ◽  
Nils Hjeltnes ◽  
Anna Krook ◽  
...  

Skeletal muscle plays an important role in the regulation of energy homeostasis; therefore, the ability of skeletal muscle to adapt and alter metabolic gene expression in response to changes in physiological demands is critical for energy balance. Individuals with cervical spinal cord lesions are characterized by tetraplegia, impaired thermoregulation, and altered skeletal muscle morphology. We characterized skeletal muscle metabolic gene expression patterns, as well as protein content, in these individuals to assess the impact of spinal cord injury on critical determinants of skeletal muscle metabolism. Our results demonstrate that mRNA levels and protein expression of skeletal muscle genes essential for glucose storage are reduced, whereas expression of glycolytic genes is reciprocally increased in individuals with spinal cord injury. Furthermore, expression of genes essential for lipid oxidation is coordinately reduced in spinal cord injured subjects, consistent with a marked reduction of mitochondrial proteins. Thus spinal cord injury resulted in a profound and tightly coordinated change in skeletal muscle metabolic gene expression program that is associated with the aberrant metabolic features of the tissue.


Inflammation ◽  
2021 ◽  
Author(s):  
Shangrila Parvin ◽  
Clintoria R. Williams ◽  
Simone A. Jarrett ◽  
Sandra M. Garraway

Abstract— Accumulating evidence supports that spinal cord injury (SCI) produces robust inflammatory plasticity. We previously showed that the pro-inflammatory cytokine tumor necrosis factor (TNF)α is increased in the spinal cord after SCI. SCI also induces a systemic inflammatory response that can impact peripheral organ functions. The kidney plays an important role in maintaining cardiovascular health. However, SCI-induced inflammatory response in the kidney and the subsequent effect on renal function have not been well characterized. This study investigated the impact of high and low thoracic (T) SCI on C-fos, TNFα, interleukin (IL)-1β, and IL-6 expression in the kidney at acute and sub-chronic timepoints. Adult C57BL/6 mice received a moderate contusion SCI or sham procedures at T4 or T10. Uninjured mice served as naïve controls. mRNA levels of the proinflammatory cytokines IL-1β, IL-6, TNFα, and C-fos, and TNFα and C-fos protein expression were assessed in the kidney and spinal cord 1 day and 14 days post-injury. The mRNA levels of all targets were robustly increased in the kidney and spinal cord, 1 day after both injuries. Whereas IL-6 and TNFα remained elevated in the spinal cord at 14 days after SCI, C-fos, IL-6, and TNFα levels were sustained in the kidney only after T10 SCI. TNFα protein was significantly upregulated in the kidney 1 day after both T4 and T10 SCI. Overall, these results clearly demonstrate that SCI induces robust systemic inflammation that extends to the kidney. Hence, the presence of renal inflammation can substantially impact renal pathophysiology and function after SCI.


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