Oxygen Kinetics: Maximally Accumulated Oxygen Deficit (MAOD) to Determine Energy System Contribution During 1,500-m Run & Excess Post-Exercise Consumption (EPOC)

2021 ◽  
pp. 83-107
Keyword(s):  
Energy System ◽  
Oxygen Deficit ◽  
Post Exercise ◽  
Oxygen Kinetics ◽  
Accumulated Oxygen Deficit

scholarly journals Energy System Contribution during 1500M Running in Untrained and Endurance-Trained Asian Male College Students

2021 ◽  
Vol 23 (2) ◽  
pp. 9-18
Author(s):  
Govindasamy Balasekaran ◽  
Loh Mun Keong ◽  
Viknesh Veeramuthu ◽  
Yong Tze Woon ◽  
Visvasuresh Victor Govindaswamy ◽  
...  
Keyword(s):  
Body Fat ◽  
Maximal Oxygen Consumption ◽  
Energy System ◽  
Oxygen Deficit ◽  
Accumulated Oxygen Deficit ◽  
Anaerobic Energy ◽  
The Mean ◽  
The Times ◽  
Study Participants ◽  
Aerobic And Anaerobic

OBJECTIVES To compare the aerobic and anaerobic energy system contribution during 1500m running between collegiate untrained (UT) and endurance trained (ET) subjects.METHODS Five Asian UT (age: 23.8 ± 0.4 yrs, body fat %: 15.9 ± 5.7 %, height: 174.0 ± 4.1 cm, weight: 65.5 ± 4.1 kg) and 5 Asian ET male participants (age: 24.4 ± 3.9 yrs, body fat %: 12.9 ± 6.9 %, height: 169.4 ± 5.1 cm, weight: 60.6 ± 8.1 kg) participated in this study. Participants attended 3 sessions to determine their body composition, submaximal and maximal oxygen consumption (VO<sub>2max</sub>) test, 1500m track running session (RS) and 1500m treadmill RS. The maximally accumulated oxygen deficit (MAOD) method was used to calculate energy system contribution.RESULTS The times for the 1500m track run for the UT and ET were 428.0 ± 48.7 and 331.6 ± 14.0 seconds (p=0.004) respectively. There were no significant differences in VO<sub>2</sub> between the 1500m track and treadmill RS indicating the participants ran to their personal best times for both trials. The mean VO<sub>2max</sub>(mL•kg<sup>-1</sup>•min<sup>-1</sup>) were significantly different between UT (45.1 ± 5.0) and ET participants (58.3 ± 2.2) (p=0.002). The mean relative contributions of the aerobic and anaerobic energy system during 1500m running were significantly different for the UT, 65.4 ± 7.0%, 34.6 ± 7.0 and ET, 75.7 ± 1.5%, 24.3 ± 1.5 % (p =0.011).CONCLUSIONS The point of equal contribution of the aerobic and the anaerobic systems occurred after thirty to forty seconds of intensive exhaustive running after which the aerobic contribution continues to increase while the anaerobic contribution decreases with increasing duration. By the end of 60th second of exhaustive running, the ET mean aerobic contribution is 71.5% compared to the UT’s 58.6%. This finding suggests a greater reliance on the aerobic energy system by the ET.

Maximal accumulated oxygen deficit in running and cycling

2011 ◽  
Vol 36 (6) ◽  
pp. 831-838 ◽  
Author(s):  
David W. Hill ◽  
Jakob L. Vingren
Keyword(s):  
Anaerobic Capacity ◽  
Oxygen Demand ◽  
Cycle Ergometer ◽  
Work Rate ◽  
Oxygen Deficit ◽  
Post Exercise ◽  
Accumulated Oxygen Deficit ◽  
The Mean ◽  
Curvilinear Relationships ◽  
The Relationship

The purpose of this study was to compare values of maximal accumulated oxygen deficit (MAOD; a measure of anaerobic capacity) in running and cycling. Twenty-seven women and 25 men performed exhaustive treadmill and cycle ergometer tests of ∼3 min, ∼5 min, and ∼7 min duration. Oxygen demands were estimated assuming a linear relationship between demand and intensity and also using upwardly curvilinear relationships. When oxygen demand was estimated using speed (with exponent 1.05), values for MAOD for the three running tests were virtually identical; the mean of the three values was 78 ± 7 mL·kg–1. Use of an oxygen demand that was estimated using work rate (with exponent 1.00) generated the most similar values for MAOD from the three cycling tests (mean of 59 ± 6 mL·kg–1). Consistent with the higher (p < 0.05) MAOD in running, peak post-exercise blood lactate concentrations were also higher (p < 0.05) in running (13.9 ± 2.2 mmol·L–1) than in cycling (12.6 ± 2.4 mmol·L–1). The results suggest that the relationship between oxygen demand and running speed is upwardly curvilinear for the speeds used to measure MAOD; the relationship between demand and cycle ergometer work rate is linear; MAOD is greater in running than in cycling.

Anaerobic contribution during maximal anaerobic running test: correlation with maximal accumulated oxygen deficit

2011 ◽  
Vol 21 (6) ◽  
pp. e222-e230 ◽  
Author(s):  
A. Zagatto ◽  
P. Redkva ◽  
J. Loures ◽  
C. Kalva Filho ◽  
V. Franco ◽  
...  
Keyword(s):  
Oxygen Deficit ◽  
Accumulated Oxygen Deficit ◽  
Test Correlation

scholarly journals Acute administration of high doses of taurine does not substantially improve high-intensity running performance and the effect on maximal accumulated oxygen deficit is unclear

2016 ◽  
Vol 41 (5) ◽  
pp. 498-503 ◽  
Author(s):  
Fabio Milioni ◽  
Elvis de Souza Malta ◽  
Leandro George Spinola do Amaral Rocha ◽  
Camila Angélica Asahi Mesquita ◽  
Ellen Cristini de Freitas ◽  
...  
Keyword(s):  
Oxygen Uptake ◽  
Exercise Intensity ◽  
Maximal Oxygen Uptake ◽  
High Intensity ◽  
Treadmill Running ◽  
Oxygen Deficit ◽  
Time To Exhaustion ◽  
Acute Administration ◽  
Running Performance ◽  
Accumulated Oxygen Deficit

The aim of the present study was to investigate the effects of acute administration of taurine overload on time to exhaustion (TTE) of high-intensity running performance and alternative maximal accumulated oxygen deficit (MAODALT). The study design was a randomized, placebo-controlled, crossover design. Seventeen healthy male volunteers (age: 25 ± 6 years; maximal oxygen uptake: 50.5 ± 7.6 mL·kg−1·min−1) performed an incremental treadmill-running test until voluntary exhaustion to determine maximal oxygen uptake and exercise intensity at maximal oxygen uptake. Subsequently, participants completed randomly 2 bouts of supramaximal treadmill-running at 110% exercise intensity at maximal oxygen uptake until exhaustion (placebo (6 g dextrose) or taurine (6 g) supplementation), separated by 1 week. MAODALT was determined using a single supramaximal effort by summating the contribution of the phosphagen and glycolytic pathways. When comparing the results of the supramaximal trials (i.e., placebo and taurine conditions) no differences were observed for high-intensity running TTE (237.70 ± 66.00 and 277.30 ± 40.64 s; p = 0.44) and MAODALT (55.77 ± 8.22 and 55.06 ± 7.89 mL·kg−1; p = 0.61), which seem to indicate trivial and unclear differences using the magnitude-based inferences approach, respectively. In conclusion, acute 6 g taurine supplementation before exercise did not substantially improve high-intensity running performance and showed an unclear effect on MAODALT.

Maximal accumulated oxygen deficit is influenced by chronological age and is related to intensity of VO2PEAK

2018 ◽  
Vol 15 (1) ◽  
pp. 109-114
Author(s):  
Vitor Luiz de Andrade ◽  
Carlos Augusto Kalva-Filho ◽  
Eduardo Zapaterra Campos ◽  
Marcelo Papoti
Keyword(s):  
Oxygen Deficit ◽  
Chronological Age ◽  
Accumulated Oxygen Deficit

The Maximal Accumulated Oxygen Deficit Method

2010 ◽  
Vol 40 (4) ◽  
pp. 285-302 ◽  
Author(s):  
Dionne A. Noordhof ◽  
Jos J. de Koning ◽  
Carl Foster
Keyword(s):  
Oxygen Deficit ◽  
Accumulated Oxygen Deficit

scholarly journals Antioxidants Facilitate High–intensity Exercise IL–15 Expression in Skeletal Muscle

2018 ◽  
Vol 40 (01) ◽  
pp. 16-22 ◽  
Author(s):  
Alberto Pérez-López ◽  
Marcos Martin-Rincon ◽  
Alfredo Santana ◽  
Ismael Perez-Suarez ◽  
Cecilia Dorado ◽  
...  
Keyword(s):  
Skeletal Muscle ◽  
Protein Expression ◽  
High Intensity ◽  
Human Skeletal Muscle ◽  
Vastus Lateralis ◽  
Acute Hypoxia ◽  
High Intensity Exercise ◽  
Oxygen Deficit ◽  
Post Exercise ◽  
Sprint Exercise

AbstractInterleukin (IL)-15 stimulates mitochondrial biogenesis, fat oxidation, glucose uptake and myogenesis in skeletal muscle. However, the mechanisms by which exercise triggers IL-15 expression remain to be elucidated in humans. This study aimed at determining whether high-intensity exercise and exercise-induced RONS stimulate IL-15/IL-15Rα expression and its signaling pathway (STAT3) in human skeletal muscle. Nine volunteers performed a 30-s Wingate test in normoxia and hypoxia (PIO2=75 mmHg), 2 h after placebo or antioxidant administration (α-lipoic acid, vitamin C and E) in a randomized double-blind design. Blood samples and muscle biopsies (vastus lateralis) were obtained before, immediately after, and 30 and 120 min post-exercise. Sprint exercise upregulated skeletal muscle IL-15 protein expression (ANOVA, P=0.05), an effect accentuated by antioxidant administration in hypoxia (ANOVA, P=0.022). In antioxidant conditions, the increased IL-15 expression at 120 min post-exercise (33%; P=0.017) was associated with the oxygen deficit caused by the sprint (r=–0.54; P=0.020); while, IL-15 and Tyr705-STAT3 AUCs were also related (r=0.50; P=0.036). Antioxidant administration promotes IL-15 protein expression in human skeletal muscle after sprint exercise, particularly in severe acute hypoxia. Therefore, during intense muscle contraction, a reduced PO2 and glycolytic rate, and possibly, an attenuated RONS generation may facilitate IL-15 production, accompanied by STAT3 activation, in a process that does not require AMPK phosphorylation.

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