scholarly journals Functional genetic characterization of salivary gland development in Aedes aegypti

EvoDevo ◽  
2013 ◽  
Vol 4 (1) ◽  
pp. 9 ◽  
Author(s):  
Chilinh Nguyen ◽  
Emily Andrews ◽  
Christy Le ◽  
Longhua Sun ◽  
Zeinab Annan ◽  
...  
2013 ◽  
Vol 2013 ◽  
pp. 1-7 ◽  
Author(s):  
Anthony C. Nichols ◽  
Michelle Chan-Seng-Yue ◽  
John Yoo ◽  
Sumit K. Agrawal ◽  
Maud H. W. Starmans ◽  
...  

We describe the presentation, management, and clinical outcome of a massive acinic cell carcinoma of the parotid gland. The primary tumor and blood underwent exome sequencing which revealed deletions in CDKN2A as well as PPP1R13B, which induces p53. A damaging nonsynonymous mutation was noted in EP300, a histone acetylase which plays a role in cellular proliferation. This study provides the first insights into the genetic underpinnings of this cancer. Future large-scale efforts will be necessary to define the mutational landscape of salivary gland malignancies to identify therapeutic targets and biomarkers of treatment failure.


Author(s):  
Proawpilart Intayot ◽  
Atchara Phumee ◽  
Kanyarat Kraivichian ◽  
Sriwatapron Sor-suwan ◽  
Rungfar Boonserm ◽  
...  

2013 ◽  
Vol 7 (10) ◽  
pp. e2486 ◽  
Author(s):  
Jeff Liesch ◽  
Lindsay L. Bellani ◽  
Leslie B. Vosshall

2021 ◽  
Vol 0 (0) ◽  
pp. 0
Author(s):  
GH Galhena ◽  
H.A.K.M. Dharmarathne ◽  
O.V.D.S.J. Weerasena ◽  
K.L.N.S. Perera

2001 ◽  
Vol 120 (5) ◽  
pp. A166-A166
Author(s):  
S FUJII ◽  
T KUSAKA ◽  
T KAIHARA ◽  
Y UEDA ◽  
T CHIBA ◽  
...  

2009 ◽  
Vol 221 (03) ◽  
Author(s):  
R Vagkopoulou ◽  
C Eckert ◽  
U Ungethüm ◽  
G Körner ◽  
M Stanulla ◽  
...  

Tick-borne encephalitis virus (TBEV) was isolated for the first time in Sweden in 1958 (from ticks and from 1 tick-borne encephalitis [TBE] patient).1 In 2003, Haglund and colleagues reported the isolation and antigenic and genetic characterization of 14 TBEV strains from Swedish patients (samples collected 1991–1994).2 The first serum sample, from which TBEV was isolated, was obtained 2–10 days after onset of disease and found to be negative for anti-TBEV immunoglobulin M (IgM) by enzyme-linked immunosorbent assay (ELISA), whereas TBEV-specific IgM (and TBEV-specific immunoglobulin G/cerebrospinal fluid [IgG/CSF] activity) was demonstrated in later serum samples taken during the second phase of the disease.


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