scholarly journals Exercise-Induced Sustained Ventricular Tachycardia without Structural Heart Disease: A Case Report

2020 ◽  
Vol 21 ◽  
Author(s):  
Michelle Audrey Darmadi ◽  
Axel Duval ◽  
Hanaa Khadraoui ◽  
Alberto N. Romero ◽  
Blanca Simon ◽  
...  
Cardiology ◽  
1996 ◽  
Vol 87 (1) ◽  
pp. 33-41 ◽  
Author(s):  
Shih-Huang Lee ◽  
Shih-Ann Chen ◽  
Ching-Tai Tai ◽  
Chern-En Chiang ◽  
Tsu-Juey Wu ◽  
...  

2017 ◽  
Vol 2 (43) ◽  
pp. 18-22
Author(s):  
Agnieszka Wojdyła-Hordyńska ◽  
Grzegorz Hordyński ◽  
Patrycja Pruszkowska-Skrzep ◽  
Oskar Kowalski

Nonsustained ventricular tachycardia (NSVT) is usually a mild entity without serious clinical consequences. Nevertheless, exercise-induced NSVT, and short cycle length of tachycardia, significant arrhythmia burden may predict increased cardiac mortality. NSVT is defined as 3 or more consecutive ventricular beats with a rate over 100 beats/min or more, lasting less than 30 s, that can be diagnosed on the basis of electrocardiography, Holter, telemetry, cardiac monitors or exercise test, after careful wide QRS tachycardia differential diagnostics, artefacts, supraventricular tachycardia with aberration, Hiss-Purkinje block or additional atrio-ventricular pathway descending conduction exclusion. It is necessary to assess homogeneity of the tachycardia (monomorphic or polymorphic), exclude ischemic, structural and genetic heart disease. The treatment is based on observation, farmacotherapy (mainly with beta-blockers, calcium channels blockers and antiarrhythmics) and percutaneous ablation. Implantable cardioverter-defibrillator is not recommended. The prognosis is usually good.


2011 ◽  
Vol 5 ◽  
pp. CMC.S8504 ◽  
Author(s):  
Shahriar Dadkhah ◽  
Samaneh Dowlatshahi ◽  
Korosh Sharain ◽  
Roza Sharain

Diagnostic stress echo testing is commonly performed in patients with known or suspected cardiovascular disease. There has been considerable debate in management of exercise induced non-sustained ventricular tachycardia (NSVT). In this case report, we present our experience with a case of exercise induced NSVT, and subsequent angiographically significant left anterior descending (LAD) coronary artery lesion.


2021 ◽  
Vol 8 ◽  
Author(s):  
Xiaoyong Xu ◽  
Ming Ye ◽  
Yaxun Sun ◽  
Qiang Liu ◽  
Fusheng Ma ◽  
...  

Background: The substrate for ventricular tachycardia (VT) in patients with structural heart disease is usually complex and often requires extensive ablation. As a result, the incidence of major procedure-related complications has been reported to be higher when compared to patients without structural heart disease. In this study, we present a rare complication after extensive substrate modification of scar-related VT.Case: A 65-year-old man with ischemic cardiomyopathy was referred to the electrophysiology laboratory for radiofrequency ablation of VT following repetitive implantable cardioverter defibrillator shocks within a short period. As with hemodynamic intolerance of induced VT, an approach involving extensive endocardial substrate modification to reduce the arrhythmogenicity of the scars was adopted. After the procedure, the heart function of the patient deteriorated significantly. The postprocedural ECG showed a bizarre, extremely wide surface QRS complex (360 ms), termed as homologous ventricular separation. The pronounced dyssynchrony of the ventricle was corrected by an upgrade to cardiac resynchronization therapy with defibrillation (CRT-D). As a result, the symptoms of the patient improved significantly. The width of the intrinsic QRS complex was not recovered during an 18-month follow-up.Conclusion: Homologous ventricular separation is a rare arrhythmia, manifested as two separated QRS waves. This case report demonstrates, for the first time, that homologous ventricular separation may occur after extensive substrate modification of scar-related VT. CRT-D can correct the dyssynchronous ventricle caused by homologous ventricular separation.


ESC CardioMed ◽  
2018 ◽  
pp. 2259-2265
Author(s):  
Alfred E. Buxton

Non-sustained ventricular tachycardia (NSVT) is classified in a variety of ways, depending on the clinical situation. The two primary distinctions are whether the arrhythmia occurs in the presence or absence of structural heart disease, and whether or not the arrhythmia causes symptoms. The prevalence of NSVT is highest in patients with structural heart disease. NSVT in patients with heart disease rarely causes symptoms, but may be associated with increased total mortality and sudden cardiac death risk. However, sudden cardiac death risk is usually not elevated out of proportion to the increased total mortality risk, suggesting that NSVT is merely a marker of sicker patients, rather than having a mechanistic relation to arrhythmias causing cardiac arrest. Furthermore, no trial has demonstrated that suppression of NSVT reduces mortality. In contrast, patients with symptoms due to NSVT usually do not have underlying structural heart disease. In these patients, treatment may be necessary to relieve symptoms and improve quality of life. Appropriate treatment of NSVT in this setting also does not improve mortality, because NSVT in the absence of structural heart disease is not associated with increased mortality or sudden death risk (excepting patients with ion channelopathies, such as long QT syndrome). The exception to this rule is the recognition that ventricular dysfunction may be caused by frequent or incessant episodes of NSVT. In this case, treatment of the arrhythmia may not only improve symptoms, but presumably also improve survival.


Author(s):  
Matthias Antz

An electrophysiological (EP) study is performed for diagnostic reasons, for risk assessment, and for therapy of arrhythmias. It can be useful in athletes with palpitations or documented arrhythmias of unclear origin, in non-sustained ventricular tachycardia, structural heart disease, or electrical abnormalities such as the Brugada syndrome, and for treatment of ectopic beats, supraventricular or ventricular tachycardia, and atrial fibrillation.


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