Case report: beware the silver nitrate stick – a risk factor for bisphosphonate-related osteonecrosis of the jaw (BRONJ)

Dental Update ◽  
2015 ◽  
Vol 42 (8) ◽  
pp. 735-743 ◽  
Author(s):  
Michelle C de Souza ◽  
Gabriela Stepavoi
Author(s):  
Francisco Sousa ◽  
Joana Vicente ◽  
Sara Azevedo ◽  
Ana Pinto ◽  
Cecília Sousa

2021 ◽  
pp. 101642
Author(s):  
Anahita Ansari Djafari ◽  
Katayoun Hasanzadeh ◽  
Homa Masrour ◽  
Mahsa Ahadi ◽  
Majid Dargahi ◽  
...  

BDJ ◽  
2021 ◽  
Vol 230 (1) ◽  
pp. 23-26
Author(s):  
Thomas Dennis ◽  
Matthew Gahan

2021 ◽  
pp. 152660282110250
Author(s):  
Barbara Infante ◽  
Adelaide Di Lorenzo ◽  
Dario Troise ◽  
Angela M. R. Ferrante ◽  
Maurizio Ruggieri ◽  
...  

Pseudoaneurysm is due to a disruption in arterial wall continuity. It forms a sac that communicates with the vessel lumen and is surrounded by the compressed, surrounding tissues and not by the wall of the artery from which the lesion arises. Many causes can predispose to the formation of a pseudoaneurysm such as trauma, surgical procedures, anticoagulation. In our patient another important risk factor for the formation of a pseudoaneurysm is ADPKD (autosomal dominant polycystic kidney disease) that can cause vascular complication. The mechanisms leading to the genesis of the pseudoaneurysms in our patient are unknown, but the clinicians should bear in mind when evaluating this type of patients that ADPKD may have a various range of systemic cardiovascular manifestation.


2001 ◽  
Vol 87 (2) ◽  
pp. 111-112
Author(s):  
Jon Matthews ◽  
Giles W Beck ◽  
Douglas M G Bowley ◽  
Andrew N Kingsnorth

AbstractThe case of a 31 year old male presenting as an emergency with a recurrent colonic volvulus is described. A chest X-ray on admission to hospital showed the presence of hepato-diaphragmatic interposition of the colon, Chilaiditi’s Sign, which is known to be a risk factor for colonic volvulus. This is only the fourth reported case of colonic volvulus in association with Chilaiditi’s Syndrome and the first with recurrent colonic volvulus. The optimal treatment for recurrent volvulus in patients with risk factors such as Chilaiditi’s Syndrome or megacolon is also discussed.


2021 ◽  
Author(s):  
Hyeong-Jin Baek ◽  
Hyejin Lee ◽  
Jae-Ryun Lee ◽  
Jung-Hyun Park ◽  
Keun-Suh Kim ◽  
...  

Abstract Background: Osteonecrosis of the jaw (ONJ) is one of the complication of bisphosphonate (BP). Despite being a major risk factor for ONJ, tooth extraction cannot explain all cases of ONJ. However, disease that induce inflammation in the jawbone, such as pulp and periapical disease is underestimated, and studies suggesting relationship are arising. This necessitates the determination of the relationship between ONJ and inflammatory disease, which would contribute to the understanding and treatment of ONJ.Methods: We analyzed the relationship between ONJ and pulp and periapical disease, and caries in women aged over 50 years who were administered BP for over 1 year from 2010 to 2015, based on a nationwide cohort study. ONJ, pulp and periapical disease, and caries were defined according to the Korean Standard Classification of Diseases and Causes of Death-7 and claims data. ONJ was operationally defined into definite ONJ and possible ONJ because of the ambiguity of population-based ONJ diagnosis.Results: Pulp and periapical disease significantly increased ONJ development [hazard ratio 2.21 (95% CI 1.40-3.48) and 2.22 (95% CI 1.65-2.98) in definite ONJ and possible ONJ, respectively]. Additionally, the risk of ONJ development increased when caries addition to pulp and periapical disease [hazard ratio 2.28 (95% 1.50-3.47) and 2.05 (95% 1.55-2.70) in definite ONJ and possible ONJ, respectively]. Nonetheless, those in the caries were not significant. Root canal treatment in most cases did not increase ONJ development significantly, but the pulp and periapical disease or caries significantly increased possible ONJ development [hazard ratio 2.17 (95% CI 1.04-4.52)].Conclusions: It is necessary to focus on pulp and periapical disease resulting in inflammation as a major risk factor for ONJ. Future studies should determine the role of low-grade inflammation for ONJ on other ONJ-inducing drugs as anti-resorptive agents.


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