Strength Training Increases Endurance Time to Exhaustion During High-Intensity Exercise Despite No Change in Critical Power

2014 ◽  
Vol 28 (3) ◽  
pp. 601-609 ◽  
Author(s):  
Brandon J. Sawyer ◽  
David G. Stokes ◽  
Christopher J. Womack ◽  
R. Hugh Morton ◽  
Arthur Weltman ◽  
...  
2007 ◽  
Vol 293 (1) ◽  
pp. R392-R401 ◽  
Author(s):  
Andrew M. Jones ◽  
Daryl P. Wilkerson ◽  
Nicolas J. Berger ◽  
Jonathan Fulford

We hypothesized that a period of endurance training would result in a speeding of muscle phosphocreatine concentration ([PCr]) kinetics over the fundamental phase of the response and a reduction in the amplitude of the [PCr] slow component during high-intensity exercise. Six male subjects (age 26 ± 5 yr) completed 5 wk of single-legged knee-extension exercise training with the alternate leg serving as a control. Before and after the intervention period, the subjects completed incremental and high-intensity step exercise tests of 6-min duration with both legs separately inside the bore of a whole-body magnetic resonance spectrometer. The time-to-exhaustion during incremental exercise was not changed in the control leg [preintervention group (PRE): 19.4 ± 2.3 min vs. postintervention group (POST): 19.4 ± 1.9 min] but was significantly increased in the trained leg (PRE: 19.6 ± 1.6 min vs. POST: 22.0 ± 2.2 min; P < 0.05). During step exercise, there were no significant changes in the control leg, but end-exercise pH and [PCr] were higher after vs. before training. The time constant for the [PCr] kinetics over the fundamental exponential region of the response was not significantly altered in either the control leg (PRE: 40 ± 13 s vs. POST: 43 ± 10 s) or the trained leg (PRE: 38 ± 8 s vs. POST: 40 ± 12 s). However, the amplitude of the [PCr] slow component was significantly reduced in the trained leg (PRE: 15 ± 7 vs. POST: 7 ± 7% change in [PCr]; P < 0.05) with there being no change in the control leg (PRE: 13 ± 8 vs. POST: 12 ± 10% change in [PCr]). The attenuation of the [PCr] slow component might be mechanistically linked with enhanced exercise tolerance following endurance training.


2013 ◽  
Vol 305 (9) ◽  
pp. R1085-R1092 ◽  
Author(s):  
Weerapong Chidnok ◽  
Fred J. DiMenna ◽  
Jonathan Fulford ◽  
Stephen J. Bailey ◽  
Philip F. Skiba ◽  
...  

We investigated the responses of intramuscular phosphate-linked metabolites and pH (as assessed by 31P-MRS) during intermittent high-intensity exercise protocols performed with different recovery-interval durations. Following estimation of the parameters of the power-duration relationship, i.e., the critical power ( CP) and curvature constant ( W′), for severe-intensity constant-power exercise, nine male subjects completed three intermittent exercise protocols to exhaustion where periods of high-intensity constant-power exercise (60 s) were separated by different durations of passive recovery (18 s, 30 s and 48 s). The tolerable duration of exercise was 304 ± 68 s, 516 ± 142 s, and 847 ± 240 s for the 18-s, 30-s, and 48-s recovery protocols, respectively ( P < 0.05). The work done > CP ( W> CP) was significantly greater for all intermittent protocols compared with the subjects' W′, and this difference became progressively greater as recovery-interval duration was increased. The restoration of intramuscular phosphocreatine concentration during recovery was greatest, intermediate, and least for 48 s, 30 s, and 18 s of recovery, respectively ( P < 0.05). The W> CP in excess of W′ increased with greater durations of recovery, and this was correlated with the mean magnitude of muscle phosphocreatine reconstitution between work intervals ( r = 0.61; P < 0.01). The results of this study show that during intermittent high-intensity exercise, recovery intervals allow intramuscular homeostasis to be restored, with the degree of restoration being related to the duration of the recovery interval. Consequently, and consistent with the intermittent CP model, the ability to perform W> CP during intermittent high-intensity exercise and, therefore, exercise tolerance, increases when recovery-interval duration is extended.


Author(s):  
Keith Tolfrey

Chapter 40 focuses specifically on the scientific evidence that has come from studies involving children or adolescents who have completed maximal (high-) intensity exercise training and resistance exercise training. The aim is not to explore health gains but those that might be important when considering participation in sport.


2006 ◽  
Vol 23 (5) ◽  
pp. 1009-1024 ◽  
Author(s):  
N. Bessot ◽  
A. Nicolas ◽  
S. Moussay ◽  
A. Gauthier ◽  
B. Sesboüé ◽  
...  

2011 ◽  
Vol 21 (4) ◽  
pp. 311-317 ◽  
Author(s):  
David M. Morris ◽  
Rebecca S. Shafer ◽  
Kimberly R. Fairbrother ◽  
Mark W. Woodall

The authors sought to determine the effects of oral lactate consumption on blood bicarbonate (HCO3−) levels, pH levels, and performance during high-intensity exercise on a cycle ergometer. Subjects (N = 11) were trained male and female cyclists. Time to exhaustion (TTE) and total work were measured during high-intensity exercise bouts 80 min after the consumption of 120 mg/kg body mass of lactate (L), an equal volume of placebo (PL), or no treatment (NT). Blood HCO3− increased significantly after ingestion of lactate (p < .05) but was not affected in PL or NT (p > .05). No changes in pH were observed as a result of treatment. TTE and total work during the performance test increased significantly by 17% in L compared with PL and NT (p = .02). No significant differences in TTE and total work were seen between the PL and NT protocols (p = .85). The authors conclude that consuming 120 mg/kg body mass of lactate increases HCO3− levels and increases exercise performance during high-intensity cycling ergometry to exhaustion.


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