Influence of Sarabikulovsk hydrogen sulfide baths on the dynamics of glutathione in cardiovascular patients

1937 ◽  
Vol 33 (4) ◽  
pp. 429-439
Author(s):  
E. B. Segen

Patients with a disorder of the activity of the cardiovascular system have various metabolic disorders: carbohydrate and protein metabolism suffers, basic metabolism and acid-base balance are disturbed, in the blood there is an excessive amount of under-oxidized metabolic products.

1935 ◽  
Vol 31 (8-9) ◽  
pp. 957-967
Author(s):  
Z. I. Malkin ◽  
T. A. Makarova ◽  
V. S. Zarbeeva

In the problem of the pathogenesis of cardiac decompensation, we are currently interested not only in issues of hemodynamics. It is known that a cardiovascular patient has various metabolic disorders, carbohydrate and protein metabolism suffers, basal metabolism and acid-base balance are disturbed, and undeoxidized metabolic products appear in the blood in excess. In the light of these data, it is of interest to solve the problem of the state of oxidative processes in the tissues of a cardiovascular patient.


1936 ◽  
Vol 32 (9) ◽  
pp. 1065-1071
Author(s):  
M. G. Mamish

It is known that a typhoid patient has various metabolic disorders, protein, fat, carbohydrate metabolism suffers (Dubrovin, cited by Rosenberg), basic metabolism and acid-base balance are disturbed (Vasyutochkina, Glinka, Otto, Beck, etc.).


1987 ◽  
Vol 253 (3) ◽  
pp. G330-G335
Author(s):  
D. S. Goldfarb ◽  
P. M. Ingrassia ◽  
A. N. Charney

We previously reported that systemic pH and HCO3 concentration affect ileal water and electrolyte absorption. To determine whether these effects could influence an ongoing secretory process, we measured transport in ileal loops exposed to either saline or 50-75 micrograms cholera toxin in mechanically ventilated Sprague-Dawley rats anesthetized with pentobarbital sodium. The effects of acute respiratory and metabolic acidosis and alkalosis were then examined. Decreases in systemic pH during respiratory acidosis caused equivalent increases in net water (54 +/- 8 microliters . cm-1 . h-1) and Na absorption (7 +/- 1 mu eq . cm- . h-1) and smaller increases in Cl absorption in cholera toxin compared with saline loops. These increases reversed the net secretion of these ions observed during alkalemia in the cholera toxin loops to net absorption. Metabolic acidosis and alkalosis and respiratory compensation of systemic pH of these metabolic disorders also altered cholera toxin-induced secretion in a direction consistent with the pH change. The increase in net HCO3 secretion caused by cholera toxin was unaffected by the respiratory disorders and did not vary with the HCO3 concentration in the metabolic disorders. These findings suggest that the systemic acid-base disorders that characterize intestinal secretory states may themselves alter intestinal absorptive function and fluid losses.


This chapter covers common metabolic disorders, principally disorders of glucose control, acid–base balance, and electrolyte balance. The nursing assessment and management of thyroid and adrenal gland emergencies are also covered.


Author(s):  
Dr Kevin Shotliffe ◽  
Dr Annabel Fountain ◽  
Dr Mike Jones ◽  
Dr Jennifer Gray ◽  
Dr Richard Leach ◽  
...  

Chapter 10 covers endocrinology and metabolic disorders, including diabetes and diabetic coma, abnormalities of sodium and potassium, calcium, magnesium, and phosphate, metabolism, acid-base balance, thyroid emergencies, pituitary emergencies, adrenal emergencies, and toxin-induced hyperthermic syndromes.


1927 ◽  
Vol 23 (12) ◽  
pp. 1216-1222
Author(s):  
V. F. Yakimov

The body has a complex regulatory mechanism to maintain its acid-base balance. The blood reaction, shifted somewhat to the alkaline side, fluctuates within very narrow limits. Even with increased production of acidic metabolic products, the deviation from the normal fluctuations of this individually constant value either does not occur at all or is very insignificant. A stronger deviation of blood Ph to one side or the other is incompatible with life.


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