scholarly journals AB037. Long-term exposure to the mobile phone radiation decreased the sperm quality of Sprague Dawley rats

2018 ◽  
Vol 7 (S5) ◽  
pp. AB037-AB037
Author(s):  
Gang Yu ◽  
Zhiming Bai
2016 ◽  
Vol 5 (5) ◽  
pp. 167-173
Author(s):  
Ali S. H. Alchalabi ◽  
Erkihun Aklilu ◽  
Abd Rahman Aziz ◽  
Mohd Azam Khan

The mobile communication technology, although integral to our everyday life, has been accounted to suffer negative impacts on the living body via two effects, thermal and non-thermal. The aims of this study were to assess the thermal effects by using Infra-red camera techniques and thermographic analysis and to find out how much electromagnetic fields from mobile phones contribute to increase the skin temperature due to thermal effects from chronic exposure to Global System for Mobile communication (GSM) mobile phone radiation. Eighty female Sprague Dawley rats were employed throughout the experiment, and the animals were dealt into four groups, control, 15, 30 and 60 days respectively (n=20) for 1h/day whole body expo-sure at SAR levels of 0.048 W/Kg. GSM-like signals at a frequency of 1800 MHz were provided by a signal generator. Thermographic analysis was done by using FLIR Tool software to estimate the changes in skin temperature in different regions of the physical structure. Statistical analysis shows signifi-cant changes in skin temperature between unexposed and exposed groups for 15 and 30 days of exposure (P< 0.001). While the skin temperature of 60 days exposure group remained consistent with unexposed group values. Our data suggest that mobile phone radiation at frequency 1800 MHz has a ther-mal effect represented by skin temperature rises in the whole body. The infra-red image analysis results are anticipated to help change mobile phone users' behavior to minimize the negative effects of mobile phone radiation.


2006 ◽  
Vol 82 (4) ◽  
pp. 285-291 ◽  
Author(s):  
H. J. Lee ◽  
S. H. Kim ◽  
S. Y. Choi ◽  
Y. M. Gimm ◽  
J. K. Pack ◽  
...  

2006 ◽  
Vol 74 (7) ◽  
pp. 4387-4389 ◽  
Author(s):  
Rachel Marion ◽  
Asiya Baishanbo ◽  
Gilles Gargala ◽  
Arnaud François ◽  
Philippe Ducrotté ◽  
...  

ABSTRACT In 5-day-old immunocompetent Sprague-Dawley rats infected with either 102 or 105 Cryptosporidium parvum oocysts, transient infection resulted 120 days later in increased cardiovascular depressor response to jejunal distension and jejunal myeloperoxidase activity (P < 0.05). Nitazoxanide treatment normalized jejunal sensitivity (P < 0.001) but not myeloperoxidase levels (P > 0.05). Data warrant further evaluation of the role of early cryptosporidiosis in the development of chronic inflammatory gut conditions.


2017 ◽  
Vol 79 (3) ◽  
Author(s):  
Siti Balkis Budin ◽  
Fatin Farhana Jubaidi ◽  
Siti Nur Farahana Mohd Noor Azam ◽  
Nur Liyana Mohamed Yusof ◽  
Izatus Shima Taib ◽  
...  

Previous studies found that Kelulut Honey produced by Trigona spp. bees is able to prevent oxidative damage in various pathological conditions.  Thus, the present study aimed to determine whether Kelulut Honey could prevent the sperm and testicular damage in streptozotocin-induced diabetic rats. Male Adult male Sprague-Dawley rats were divided into four groups: Non-Diabetic (NDM), Non-Diabetic with Kelulut Honey supplementation (NDMKH), Diabetic without supplementation (DM) and Diabetic with Kelulut Honey supplementation (DMKH).  Kelulut honey was given at the dose of 2.0 g/kg weight daily via gavage for 28 consecutive days. Results showed that sperm quality produced by diabetic rats supplemented with Kelulut honey significantly improved compared to the diabetic control groups (p<0.05). SOD activity and GSH level increased significantly (p<0.05) whereas PC and MDA levels significantly decreased in sperm and testis of DMKH rats when compared to DM rats (p<0.05). Histological observation showed obvious increase in spermatozoa in the lumen of epididymis and increased spermatogenic cells density in the testis of DMKH group.  In conclusion, Kelulut Honey has a potential in preventing the damage of sperm and testis in diabetic rats.


Author(s):  
Lai Lei Lou

Although mobile phones have proved to be lifesaving in certain circumstances, wide concerns have been raised about brain tumors associated with their use. This article systematically reviews previous and current research in regards to mobile phone use and brain tumors. Recently, research (more than 10 years mobile phone use or cumulative mobile phone use more than 1640 hours) has been found that the amount of exposure to mobile phone radiation plays a key role in determining the significant associations between mobile phone use and gliomas, and acoustic neuroma. In general, those who use mobile phones for more than ten years, or cumulative call time for more than 1640 hours, have higher risks to develop brain tumors, especially glioma and acoustic neuroma, than those who use mobile phones for less than one year.


2001 ◽  
Vol 90 (5) ◽  
pp. 2001-2006 ◽  
Author(s):  
D. D. Fuller ◽  
A. G. Zabka ◽  
T. L. Baker ◽  
G. S. Mitchell

Episodic hypoxia evokes a sustained augmentation of respiratory motor output known as long-term facilitation (LTF). Phrenic LTF is prevented by pretreatment with the 5-hydroxytryptamine (5-HT) receptor antagonist ketanserin. We tested the hypothesis that 5-HT receptor activation is necessary for the induction but not maintenance of phrenic LTF. Peak integrated phrenic nerve activity (∫Phr) was monitored for 1 h after three 5-min episodes of isocapnic hypoxia (arterial Po 2 = 40 ± 2 Torr; 5-min hyperoxic intervals) in four groups of anesthetized, vagotomized, paralyzed, and ventilated Sprague-Dawley rats [ 1) control ( n = 11), 2) ketanserin pretreatment (2 mg/kg iv; n = 7), and ketanserin treatment 0 and 45 min after episodic hypoxia ( n = 7 each)]. Ketanserin transiently decreased ∫Phr, but it returned to baseline levels within 10 min. One hour after episodic hypoxia, ∫Phr was significantly elevated from baseline in control and in the 0- and 45-min posthypoxia ketanserin groups. Conversely, ketanserin pretreatment abolished phrenic LTF. We conclude that 5-HT receptor activation is necessary to initiate (during hypoxia) but not maintain (following hypoxia) phrenic LTF.


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