Telemedicine in Heart Disease: Role of Remote Patient Management in Guideline-Based Heart Failure Care

2006 ◽  
Vol 14 (Suppl 1) ◽  
pp. 15???18 ◽  
Author(s):  
Christoph Straub ◽  
Anne-Kathrin Haas ◽  
Jenny Mex
2021 ◽  
Vol 17 ◽  
Author(s):  
Mark T. Nolan ◽  
Neville Tan ◽  
Christopher J. Neil

Purpose of Review: To summarise and discuss the implications of recent technological advances in heart failure care. Recent Findings: Heart failure remains a significant source of morbidity and mortality in the US population despite multiple classes of approved pharmacological treatments. Novel cardiac devices and technologies may offer an opportunity to improve outcomes. Baroreflex Activation Therapy and Cardiac Contractility Remodelling may improve myocardial contractility by altering neurohormonal stimulation of the heart. Implantable Pulmonary Artery Monitors and Biatrial Shunts may prevent heart failure admissions by altering the trajectory of progressive congestion. Phrenic Nerve Stimulation offers potentially effective treatment for comorbid conditions. Smartphone applications offer an intriguing strategy for improving medication adherence. Summary: Novel heart failure technologies offer promise for reducing this public health burden. Randomized controlled studies are indicated for assessing the future role of these novel therapies.


2020 ◽  
Vol 7 (5) ◽  
pp. 2516-2526
Author(s):  
Tina Stegmann ◽  
Kerstin Koehler ◽  
Rolf Wachter ◽  
Volker Moeller ◽  
Samira Zeynalova ◽  
...  

2017 ◽  
Vol 89 (4) ◽  
pp. 91-94 ◽  
Author(s):  
A F Verbovoy ◽  
I A Tsanava ◽  
E V Mitroshina ◽  
L A Sharonova

Osteoprotegerin (OPG) is a glycoprotein that is a representative of the tumor necrosis factor-α receptor superfamily. Information about the possible role of OPG in the development of cardiovascular diseases has begun to appear in the literature in recent years. This review discusses the role of increasing the level of OPG in the development and progression of atherosclerosis and as a consequence of coronary heart disease and chronic heart failure.


2007 ◽  
Vol 292 (1) ◽  
pp. C45-C51 ◽  
Author(s):  
Åsa B. Gustafsson ◽  
Roberta A. Gottlieb

Loss of myocardial cells via apoptosis has been observed in many cardiovascular diseases and has been shown to contribute to the initiation and progression of heart failure. The Bcl-2 family members are important regulators of the mitochondrial pathway of apoptosis. These proteins decide whether the mitochondria should initiate the cell death program and release proapoptotic factors such as cytochrome c. The Bcl-2 proteins consist of anti- and proapoptotic members and play a key role in regulating apoptosis in the myocardium. The antiapoptotic proteins have been demonstrated to protect against various cardiac pathologies, whereas the antiapoptotic proteins have been reported to contribute to heart disease. This review summarizes the current understanding of the role of Bcl-2 proteins in the heart.


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