scholarly journals Study on the scarcity of red tide in the eutrophicated waters of ARIAKE bay - I. Dissolution of phosphate ion from bottom mud by hydrogen sulfide.

1980 ◽  
Vol 46 (8) ◽  
pp. 977-989 ◽  
Author(s):  
Shinya ISHIO ◽  
Keizo KONDO
Keyword(s):  
Hydrogen Sulfide ◽  
Red Tide ◽  
Ariake Bay ◽  
Phosphate Ion ◽  
Bottom Mud

scholarly journals Improved MODIS-Aqua Chlorophyll-a Retrievals in the Turbid Semi-Enclosed Ariake Bay, Japan

2018 ◽  
Vol 10 (9) ◽  
pp. 1335 ◽  
Author(s):  
Meng Meng Yang ◽  
Joji Ishizaka ◽  
Joaquim I. Goes ◽  
Helga do R. Gomes ◽  
Elígio de Raús Maúre ◽  
...  
Keyword(s):  
Chlorophyll A ◽  
Red Tide ◽  
Atmospheric Correction ◽  
Ariake Bay ◽  
Band Ratio ◽  
Simple Method ◽  
Chl A ◽  
Switching Algorithm ◽  
Economic Significance

The accurate retrieval of chlorophyll-a concentration (Chl-a) from ocean color satellite data is extremely challenging in turbid, optically complex coastal waters. Ariake Bay in Japan is a turbid semi-enclosed bay of great socio-economic significance, but it suffers from serious water quality problems, particularly due to red tide events. Chl-a derived from the MODerate resolution Imaging Spectroradiometer (MODIS) sensor on satellite Aqua in Ariake Bay was investigated, and it was determined that the causes of the errors were from inaccurate atmospheric correction and inappropriate in-water algorithms. To improve the accuracy of MODIS remote sensing reflectance (Rrs) in the blue and green bands, a simple method was adopted using in situ Rrs data. This method assumes that the error in MODIS Rrs(547) is small, and MODIS Rrs(412) can be estimated from MODIS Rrs(547) using a linear relation between in situ Rrs(412) and Rrs(547). We also showed that the standard MODIS Chl-a algorithm, OC3M, underestimated Chl-a, which was mostly due to water column turbidity. A new empirical switching algorithm was generated based on the relationship between in situ Chl-a and the blue-to-green band ratio, max(Rrs(443), Rrs(448)/Rrs(547), which was the same as the OC3M algorithm. The criterion of Rrs(667) of 0.005 sr−1 was used to evaluate the extent of turbidity for the switching algorithm. The results showed that the switching algorithm performed better than OC3M, and the root mean square error (RMSE) of estimated Chl-a decreased from 0.414 to 0.326. The RMSE for MODIS Chl-a using the recalculated Rrs and the switching algorithm was 0.287, which was a significant improvement from the RMSE of 0.610, which was obtained using standard MODIS Chl-a. Finally, the accuracy of our method was tested with an independent dataset collected by the local Fisheries Research Institute, and the results revealed that the switching algorithm with the recalculated Rrs reduced the RMSE of MODIS Chl-a from 0.412 of the standard to 0.335.

scholarly journals Competition for Nutrient Uptake during Winter Season among Cultivated Nori Layer and Phytoplanktons Causing Red Tide in Ariake Bay

2005 ◽  
Vol 28 (5) ◽  
pp. 339-345 ◽  
Author(s):  
Masataka NAKASHIMA ◽  
Yoshihiro YOKOYAMA ◽  
Tadashi UCHIDA ◽  
Takuji NAKANO ◽  
Hiroshi NAKANISHI
Keyword(s):  
Nutrient Uptake ◽  
Red Tide ◽  
Winter Season ◽  
Ariake Bay

scholarly journals Study on the scarcity of red tide in the eutrophicated waters of Ariake Bay - V. Separation of dinoflagellate growth inhibitor from sea sediment.

1988 ◽  
Vol 54 (7) ◽  
pp. 1175-1181 ◽  
Author(s):  
Shinya Ishio ◽  
Takashi Nishimoto ◽  
Hisaki Nakagawa
Keyword(s):  
Red Tide ◽  
Growth Inhibitor ◽  
Ariake Bay

scholarly journals Study on the scarcity of red tide in the eutrophicated waters of Ariake Bay. II. H2S-extraction method for Fe-bound P in sea sediments.

1986 ◽  
Vol 52 (1) ◽  
pp. 103-108 ◽  
Author(s):  
Shinya Ishio ◽  
Masato Kuwahara ◽  
Hisaki Nakagawa
Keyword(s):  
Extraction Method ◽  
Red Tide ◽  
Ariake Bay

scholarly journals Study on the scarcity of red tide in the eutrophicated waters of Ariake Bay. III. Conversion of AlPO4-P to Fe-bound P in sea sediments.

1986 ◽  
Vol 52 (5) ◽  
pp. 901-911 ◽  
Author(s):  
Shinya Ishio ◽  
Masato Kuwahara ◽  
Hisaki Nakagawa
Keyword(s):  
Red Tide ◽  
Ariake Bay

Studies of the Cross Section of Water in the Innermost Areas of Ariake Bay with the Recent Occurrence of Hypoxic Water and Red Tide.

2003 ◽  
Vol 12 (3) ◽  
pp. 291-305 ◽  
Author(s):  
Hiroaki Tsutsumi ◽  
Emiko Okamura ◽  
Mitsuyo Ogawa ◽  
Tohru Takahashi ◽  
Hitomi Yamaguchi ◽  
...  
Keyword(s):  
Cross Section ◽  
Red Tide ◽  
Ariake Bay ◽  
Hypoxic Water ◽  
The Cross ◽  
Recent Occurrence

Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation

2019 ◽  
Vol 133 (20) ◽  
pp. 2045-2059 ◽  
Author(s):  
Da Zhang ◽  
Xiuli Wang ◽  
Siyao Chen ◽  
Selena Chen ◽  
Wen Yu ◽  
...  
Keyword(s):  
Hydrogen Sulfide ◽  
Endothelial Cell ◽  
Pulmonary Artery ◽  
Cell Model ◽  
Site Directed Mutagenesis ◽  
Critical Event ◽  
Hypertensive Rats ◽  
Pulmonary Artery Endothelial Cell

Abstract Background: Pulmonary artery endothelial cell (PAEC) inflammation is a critical event in the development of pulmonary arterial hypertension (PAH). However, the pathogenesis of PAEC inflammation remains unclear. Methods: Purified recombinant human inhibitor of κB kinase subunit β (IKKβ) protein, human PAECs and monocrotaline-induced pulmonary hypertensive rats were employed in the study. Site-directed mutagenesis, gene knockdown or overexpression were conducted to manipulate the expression or activity of a target protein. Results: We showed that hydrogen sulfide (H2S) inhibited IKKβ activation in the cell model of human PAEC inflammation induced by monocrotaline pyrrole-stimulation or knockdown of cystathionine γ-lyase (CSE), an H2S generating enzyme. Mechanistically, H2S was proved to inhibit IKKβ activity directly via sulfhydrating IKKβ at cysteinyl residue 179 (C179) in purified recombinant IKKβ protein in vitro, whereas thiol reductant dithiothreitol (DTT) reversed H2S-induced IKKβ inactivation. Furthermore, to demonstrate the significance of IKKβ sulfhydration by H2S in the development of PAEC inflammation, we mutated C179 to serine (C179S) in IKKβ. In purified IKKβ protein, C179S mutation of IKKβ abolished H2S-induced IKKβ sulfhydration and the subsequent IKKβ inactivation. In human PAECs, C179S mutation of IKKβ blocked H2S-inhibited IKKβ activation and PAEC inflammatory response. In pulmonary hypertensive rats, C179S mutation of IKKβ abolished the inhibitory effect of H2S on IKKβ activation and pulmonary vascular inflammation and remodeling. Conclusion: Collectively, our in vivo and in vitro findings demonstrated, for the first time, that endogenous H2S directly inactivated IKKβ via sulfhydrating IKKβ at Cys179 to inhibit nuclear factor-κB (NF-κB) pathway activation and thereby control PAEC inflammation in PAH.

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