CEREBRAL PALSY: DIAGNOSIS AND PSYCHOLOGICAL CORRECTION OF MENTAL RETARDATION IN PRESCHOOL CHILDREN(Literature review)

The analysis of the studied scientific and medical literature has shown that there are no clear criteria for diagnosing mental retardation in children with cerebral palsy (CP). The Wexler test, which is used to determine the form of mental retardation, practically can not be used in the diagnosis of intellectual disabilities in cerebral palsy. All this dictates the need for additional research, development of criteria for diagnosing mental retardation in cerebral palsy

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Minimum alveolar concentration for halothane in children with cerebral palsy and severe mental retardation

Anaesthesia ◽

10.1111/j.1365-2044.1997.257-az0376.x ◽

1997 ◽

Vol 52 (11) ◽

pp. 1056-1060 ◽

Cited By ~ 47

Author(s):

F. J. Frei ◽

M. H. Haemmerle ◽

R. Brunner ◽

C. Kern

Keyword(s):

Cerebral Palsy ◽

Mental Retardation ◽

Minimum Alveolar Concentration ◽

Severe Mental Retardation ◽

Children With Cerebral Palsy

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Discontinuation of Antiepileptic Drug Treatment After Two Seizure-free Years in Children With Cerebral Palsy

PEDIATRICS ◽

10.1542/peds.97.2.192 ◽

1996 ◽

Vol 97 (2) ◽

pp. 192-197

Author(s):

Mauricio R. Delgado ◽

Anthony R. Riela ◽

Janith Mills ◽

Alan Pitt ◽

Richard Browne

Keyword(s):

Cerebral Palsy ◽

Mental Retardation ◽

Antiepileptic Drug ◽

Spastic Diplegia ◽

Multiple Factors ◽

Spastic Hemiparesis ◽

Children With Cerebral Palsy ◽

Epileptic Children ◽

Seizure Relapse ◽

Antiepileptic Drug Treatment

Objective. The risk of seizure relapse after antiepileptic drug (AED) discontinuation in children has been reported to vary between 6% and 40%. It has been suggested that neurologic deficit and mental retardation are poor prognostic factors for seizure relapse after AED discontinuation. Because epileptic children with cerebral palsy (CP) have neurologic deficits, and many have mental retardation, it is important to know their risk for seizure relapse. Methods. AED treatment was discontinued in 65 children with CP and histories of epilepsy after 2 seizure-free years. All of the patients were followed until they had seizure relapses or for at least 2 years without seizures after AEDs were stopped. Multiple factors were analyzed for possible association with seizure relapse. Results. Twenty-seven patients (41.5%) had seizure relapses. Patients with spastic hemiparesis had the highest relapse rate (61.5%), and those with spastic diplegia had the lowest rate (14.3%). No other factor correlated significantly with the risk of seizure relapse. Conclusions. Discontinuation of AEDs in children with CP can, and should, be practiced when possible after patients have been seizure-free for at least 2 years. AED discontinuation in patients with spastic hemiparesis is significantly more likely to lead to seizure relapse than in patients with other CP types, but no other factor is yet known to increase the chance of relapse.

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Does food and fluid texture consumption relate to dietary intake in preschool children with cerebral palsy?

Developmental Medicine & Child Neurology ◽

10.1111/dmcn.12775 ◽

2015 ◽

Vol 57 (11) ◽

pp. 989-990

Author(s):

Erika Gisel

Keyword(s):

Cerebral Palsy ◽

Preschool Children ◽

Dietary Intake ◽

Children With Cerebral Palsy

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Developmental Enamel Defects in Primary Teeth in Children with Cerebral Palsy, Mental Retardation, or Hearing Defects: A Review

Advances in Dental Research ◽

10.1177/08959374890030021101 ◽

1989 ◽

Vol 3 (2) ◽

pp. 132-142 ◽

Cited By ~ 31

Author(s):

M. Bhat ◽

K.B. Nelson

Keyword(s):

Cerebral Palsy ◽

Mental Retardation ◽

Primary Teeth ◽

Viral Infections ◽

Dental Enamel ◽

Tooth Germ ◽

Developmental Defects ◽

Enamel Defects ◽

Children With Cerebral Palsy ◽

Neurologic Development

Developmental enamel defects in primary teeth have been found at least twice as frequently in children with cerebral palsy or mental retardation as in control children, and frequently also in children with sensori-neural hearing deficits. The developing tooth germ is sensitive to a range of systemic disturbances, some of which may also affect neurologic development. Because the enamel cannot recover once it is damaged, it may provide a repository of information on the timing and nature of insults potentially affecting other ectodermally derived structures, including the brain. This paper reviews the literature on developmental defects of enamel in primary teeth, asking whether these might be useful as biological markers of the timing and in some cases the nature of insults. Among systemic factors related to development of enamel that might also have implications for neurologic development are certain genetic disorders including tuberous sclerosis, premature birth, neonatal nutritional disturbances (especially hypocalcemia), viral infections (such as rubella and cytomegalovirus during gestation), thyroid disorders, and maternal diabetes. It is concluded that further research is warranted concerning whether developmental defects of dental enamel can be useful markers for the timing of intra-uterine or perinatal events associated with certain neurologic and sensory disorders of children.

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