scholarly journals Evaluation of some plant extracts in management of dry bubble (Verticillium fungicola) disease of white button mushroom [Agaricus bisporus (Lange) Imbach]

2016 ◽  
Vol 8 (3) ◽  
pp. 1205-1209
Author(s):  
Shivam Singh ◽  
Abhilasha A. Lal ◽  
Anurag Singh ◽  
Rao Yaduman ◽  
Rakhi Murmu
Keyword(s):  
Plant Extracts ◽  
Agaricus Bisporus ◽  
Ocimum Sanctum ◽  
Button Mushroom ◽  
In Vivo Test ◽  
White Button Mushroom ◽  
Verticillium Fungicola ◽  
Dry Bubble

The study was undertaken to determine antifungal potentials of some plant extracts against dry bubble (Verticillium fungicola) disease of white button mushroom (Agaricus bisporus). Twelve botanicals namely, Allium cepa, A. sativum, Saraca asoca, Aloe vera, Azadirachta indica, Lantana camara, Ocimum sanctum, Solanum lycopersicum (Lycopersicon esculentum), Tagetes erecta, Psidium guajava, Catharanthus roseus and Aparagus racemosus were evaluated in-vitro and in-vivo for their efficacy against both A. bisporus and V. fungicola, causing dry bubble disease of mushroom. The efficacy of botanicals was examined by poison food technique in in-vitro. The percent inhibition produced by botanicals against V. Fungicola recorded in-vitro was; A. cepa (25.87%), A. sativum (24.70%), S. asoca (12.35%), A. vera (22.35%), A. indica (35.11%), L. camara (28.48%), O. sanctum (20.59%), S. lycopersicum (20.34%), T. erecta (14.11%), P. guajava (15.11%), C. roseus (18.11%) and A. racemosus (13.52%). Among these plant extracts, A. indica was found best treatment followed by L. Camara and A. Cepa. Plant extracts showing maximum efficacy against V. fungicola and minimum inhibition against mushroom were further evaluated against V. fungicola infection in mushroom crop room (in-vivo test). In in-vivo test, the polybags which receive A. indica show maximum mean increase in yield (43.46%) over control and exhibited minimum mean disease incidence (27.7%).

scholarly journals Susceptibility of white button mushroom (agaricus bisporus lange) and its pathogens verticillium fungicola (preuss) hassebrauk and mycogone perniciosa (magnus) delacr. to fungicides

Pesticidi ◽  
2003 ◽  
Vol 18 (4) ◽  
pp. 237-244
Author(s):  
Brankica Tanovic ◽  
Maja Vracarevic ◽  
Ivana Potocnik
Keyword(s):  
Chemical Control ◽  
Agaricus Bisporus ◽  
Management Strategy ◽  
Resistance Management ◽  
Button Mushroom ◽  
White Button Mushroom ◽  
Verticillium Fungicola ◽  
Successful Control ◽  
Low Toxicity

The isolates of two mycopathogenic fungi Verticillium fungicola and Mycogone perniciosa, and a commercial isolate of white button mushroom Agaricus bisporus were tested for sensitivity to prochloraz, benomyl, and iprodione in vitro. The pathogens were isolated from the diseased mushrooms originating from mushroom farms in Vracevsnica (Gornji Milanovac) and Pozarevac in Serbia. Prochloraz and iprodione were highly toxic to both M. perniciosa and V. fungicola. The isolate of M. perniciosa was also very sensitive to benomyl, whereas the toxicity of benomyl to V.fungicola was extremely low. Among the fungicides investigated, iprodion was the most toxic and benomyl the least toxic to the isolate of white button mushroom. Chemical control of both dry and wet bubble is possible by prochloraz and iprodione. Moreover, successful control of wet bubble causal agent can be obtained by benomyl as well, due to high toxicity of this fungicide to the pathogen and low toxicity to white button mushroom. In addition, use of benomyl alternating prochloraz provides resistance management strategy providing that a given farm is free of V.fungicola population.

In vitro cultures and fruiting bodies of culinary-medicinal Agaricus bisporus (white button mushroom) as a source of selected biologically-active elements

2015 ◽  
Vol 52 (11) ◽  
pp. 7337-7344 ◽  
Author(s):  
Bożena Muszyńska ◽  
Agata Krakowska ◽  
Katarzyna Sułkowska-Ziaja ◽  
Włodzimierz Opoka ◽  
Witold Reczyński ◽  
...  
Keyword(s):  
Agaricus Bisporus ◽  
In Vitro Cultures ◽  
Biologically Active ◽  
Fruiting Bodies ◽  
Button Mushroom ◽  
Active Elements ◽  
White Button Mushroom

Genetic and physiological variation in isolates of Verticillium fungicola causing dry bubble disease of the cultivated button mushroom, Agaricus bisporus

2002 ◽  
Vol 106 (10) ◽  
pp. 1163-1170 ◽  
Author(s):  
Sergio Juarez Del Carmen ◽  
Michele L. Largeteau-Mamoun ◽  
Thierry Rousseau ◽  
Catherine Regnault-Roger ◽  
Jean-Michel Savoie
Keyword(s):  
Agaricus Bisporus ◽  
Button Mushroom ◽  
Physiological Variation ◽  
Verticillium Fungicola ◽  
Dry Bubble ◽  
Dry Bubble Disease

scholarly journals Genome Sequence of Lecanicillium fungicola 150-1, the Causal Agent of Dry Bubble Disease

2019 ◽  
Vol 8 (19) ◽  
Author(s):  
Alice M. Banks ◽  
Farhana Aminuddin ◽  
Katherine Williams ◽  
Thomas Batstone ◽  
Gary L. A. Barker ◽  
...  
Keyword(s):  
Genome Sequence ◽  
Agaricus Bisporus ◽  
Control Strategies ◽  
Causal Agent ◽  
Button Mushroom ◽  
Content Type ◽  
White Button Mushroom ◽  
Dry Bubble ◽  
Dry Bubble Disease

The fungus Lecanicillium fungicola causes dry bubble disease in the white button mushroom Agaricus bisporus. Control strategies are limited, as both the host and pathogen are fungi, and there is limited understanding of the interactions in this pathosystem.

Defibrotide Inhibits Platelet Activation by Cathepsin G Released From Stimulated Polymorphonuclear Leukocytes

1992 ◽  
Vol 67 (06) ◽  
pp. 660-664 ◽  
Author(s):  
Virgilio Evangelista ◽  
Paola Piccardoni ◽  
Giovanni de Gaetano ◽  
Chiara Cerletti
Keyword(s):  
Platelet Activation ◽  
Polymorphonuclear Leukocytes ◽  
Direct Interaction ◽  
Cathepsin G ◽  
In Vivo Test ◽  
Cell Functions ◽  
Test Models ◽  
Antithrombotic Effects

SummaryDefibrotide is a polydeoxyribonucleotide with antithrombotic effects in experimental animal models. Most of the actions of this drug have been observed in in vivo test models but no effects have been reported in in vitro systems. In this paper we demonstrate that defibrotide interferes with polymorphonuclear leukocyte-induced human platelet activation in vitro. This effect was not related to any direct interaction with polymorphonuclear leukocytes or platelets, but was due to the inhibition of cathepsin G, the main biochemical mediator of this cell-cell cooperation. Since cathepsin G not only induces platelet activation but also affects some endothelial cell functions, the anticathepsin G activity of defibrotide could help to explain the antithrombotic effect of this drug.

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