Acute Lymphoblastic Leukaemia Week

Vincristine-induced apoptosis in vivo in peripheral blood mononuclear cells of children with acute lymphoblastic leukaemia (ALL)

British Journal of Haematology ◽

10.1046/j.1365-2141.2000.02420.x ◽

2000 ◽

Vol 111 (3) ◽

pp. 875-878 ◽

Cited By ~ 2

Author(s):

E. Groninger ◽

S. S. N. de Graaf ◽

G. J. Meeuwsen-de Boer ◽

W. J. Sluiter ◽

S. Poppema

Keyword(s):

Acute Lymphoblastic Leukaemia ◽

Peripheral Blood Mononuclear Cells ◽

Lymphoblastic Leukaemia ◽

Peripheral Blood ◽

Mononuclear Cells ◽

Peripheral Blood Mononuclear ◽

Blood Mononuclear Cells ◽

Induced Apoptosis

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Day Care in Infancy and Risk of Childhood Acute Lymphoblastic Leukaemia: Findings From UK Case-Control Study

Yearbook of Pediatrics ◽

10.1016/s0084-3954(08)70241-0 ◽

2007 ◽

Vol 2007 ◽

pp. 437-439

Author(s):

J.A. Stockman

Keyword(s):

Acute Lymphoblastic Leukaemia ◽

Lymphoblastic Leukaemia ◽

Day Care ◽

Case Control Study ◽

Case Control ◽

Childhood Acute Lymphoblastic Leukaemia ◽

Control Study

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Screening for the P2RY8-CRLF2 fusion gene in childhood acute lymphoblastic leukaemia

Klinische Pädiatrie ◽

10.1055/s-0031-1277073 ◽

2011 ◽

Vol 223 (03) ◽

Author(s):

M Morak ◽

R Joas ◽

S Fischer ◽

A Attarbaschi ◽

G Mann ◽

...

Keyword(s):

Acute Lymphoblastic Leukaemia ◽

Lymphoblastic Leukaemia ◽

Fusion Gene ◽

Childhood Acute Lymphoblastic Leukaemia

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Cerebral sinovenous thrombosis during asparaginase treatment

Hämostaseologie ◽

10.1055/s-0037-1619586 ◽

2003 ◽

Vol 23 (03) ◽

pp. 109-112

Author(s):

A. Hirt ◽

C. Zwicky ◽

W.A. Wuillemin ◽

K. Leibundgut

Keyword(s):

Acute Lymphoblastic Leukaemia ◽

Lymphoblastic Leukaemia ◽

Induction Treatment ◽

Pathogenic Mechanisms ◽

Cerebral Sinovenous Thrombosis ◽

Haemorrhagic Infarction ◽

Sinovenous Thrombosis

SummaryA boy (age: 71/12 years) with acute lymphoblastic leukaemia developed thrombosis of the sinus sagitalis superior with secondary haemorrhagic infarction while on induction treatment with vincristine, prednisone, and asparaginase. Based on this report, the potential pathogenic mechanisms are discussed with respect to congenital prothrombotic defects as well as to the role of antileukaemic treatment. Current hypotheses on mechanisms for thromboembolism in children and proposed prophylactic strategies are briefly summarized.

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