scholarly journals Chronic Fluorosis: A Disease of Concern

Author(s):  
Mendez DC ◽  
Shashidhar KN
Keyword(s):  
1934 ◽  
Vol 105 (2) ◽  
pp. 405-410
Author(s):  
Paul H. Phillips ◽  
Chang Y. Chang
Keyword(s):  

2004 ◽  
Vol 18 (2) ◽  
pp. 141-147 ◽  
Author(s):  
M. Oncu ◽  
K. Gülle ◽  
E. Karaoz ◽  
F. Gultekin ◽  
S. Karaoz ◽  
...  

2004 ◽  
Vol 102 (1-3) ◽  
pp. 199-208 ◽  
Author(s):  
Erdal Karaoz ◽  
Meral Oncu ◽  
Kanat Gulle ◽  
Mehmet Kanter ◽  
Fatih Gultekin ◽  
...  

2015 ◽  
Vol 39 (4) ◽  
pp. 354 ◽  
Author(s):  
B.J. Trangadia ◽  
P.L. Kaul ◽  
B.J. Patel ◽  
D.V. Joshi ◽  
Lalita Kaul
Keyword(s):  

2012 ◽  
Vol 2012 ◽  
pp. 1-7 ◽  
Author(s):  
Y. Q. He ◽  
Y. Pan ◽  
L. J. Ying ◽  
R. Zhao

The fluoride has volcanic activity and abundantly exists in environment combining with other elements as fluoride compounds. Recent researches indicated that the molecular mechanisms of intracellular fluoride toxicity were very complex. However, the molecular mechanisms underlying the effects on gene expression of chronic fluoride-induced damage is unknown, especially the detailed regulatory process of mitochondria. In the present study, we screened the differential expression ESTs associated with fluorosis by DDRT-PCR in rat liver. We gained 8 genes, 3 new ESTs, and 1 unknown function sequence and firstly demonstrated that microsomal glutathione S-transferase 1 (MGST1), ATP synthase H+transporting mitochondrial F0complex subunit C1, selenoprotein S, mitochondrial IF1 protein, and mitochondrial succinyl-CoA synthetase alpha subunit were participated in mitochondria metabolism, functional and structural damage process caused by chronic fluorosis. This information will be very helpful for understanding the molecular mechanisms of fluorosis.


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