scholarly journals Opposite regulation of epithelial-to-mesenchymal transition and cell invasiveness by periostin between prostate and bladder cancer cells

2011 ◽  
Author(s):  
Hirokazu Inoue
Keyword(s):  
Bladder Cancer ◽  
Cancer Cells ◽  
Epithelial To Mesenchymal Transition ◽  
Mesenchymal Transition ◽  
Bladder Cancer Cells ◽  
Cell Invasiveness

scholarly journals TP53INP2 Modulates Epithelial-to-Mesenchymal Transition via the GSK-3β/β-Catenin/Snail1 Pathway in Bladder Cancer Cells

2020 ◽  
Vol Volume 13 ◽  
pp. 9587-9597
Author(s):  
Zhengtao Zhou ◽  
Xiaoqiang Liu ◽  
Yulei Li ◽  
Junhua Li ◽  
Wen Deng ◽  
...  
Keyword(s):  
Bladder Cancer ◽  
Cancer Cells ◽  
Epithelial To Mesenchymal Transition ◽  
Mesenchymal Transition ◽  
Bladder Cancer Cells ◽  
Gsk 3Β

scholarly journals Epigenetic Mechanisms Influencing Epithelial to Mesenchymal Transition in Bladder Cancer

2019 ◽  
Vol 20 (2) ◽  
pp. 297 ◽  
Author(s):  
Sara Monteiro-Reis ◽  
João Lobo ◽  
Rui Henrique ◽  
Carmen Jerónimo
Keyword(s):  
Bladder Cancer ◽  
Cancer Cells ◽  
Cancer Biology ◽  
Epithelial To Mesenchymal Transition ◽  
Metastatic Potential ◽  
Underlying Disease ◽  
Epigenetic Mechanisms ◽  
Protein Coding ◽  
Mesenchymal Transition ◽  
Cell Invasiveness

Bladder cancer is one of the most incident neoplasms worldwide, and its treatment remains a significant challenge, since the mechanisms underlying disease progression are still poorly understood. The epithelial to mesenchymal transition (EMT) has been proven to play an important role in the tumorigenic process, particularly in cancer cell invasiveness and metastatic potential. Several studies have reported the importance of epigenetic mechanisms and enzymes, which orchestrate them in several features of cancer cells and, specifically, in EMT. In this paper, we discuss the epigenetic enzymes, protein-coding and non-coding genes, and mechanisms altered in the EMT process occurring in bladder cancer cells, as well as its implications, which allows for improved understanding of bladder cancer biology and for the development of novel targeted therapies.

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