A SEX PHEROMONE IN THE INSECT RHODNIUS PROLIXUS (HEMIPTERA: REDUVIIDAE)

1971 ◽  
Vol 103 (1) ◽  
pp. 18-22 ◽  
Author(s):  
W. F. Baldwin ◽  
A. G. Knight ◽  
K. R. Lynn

AbstractIn Rhodnius, a volatile substance produced by mating pairs is attractive to male insects. The pheromone acts also as a sexual stimulant; males respond in the absence of females by attempting copulation with other males. Air swept over mating pairs and conducted to a cage at a considerable distance proved to be highly attractive to males. Proximity of the sexes, without opportunity for actual contact, does not stimulate production of the pheromone. Since males and females will copulate only after a blood meal, feeding is a prerequisite to pheromone production. Also, unfed males will not respond to the attractant. Males of this nocturnal species were stimulated only in complete darkness.

2007 ◽  
Vol 139 (5) ◽  
pp. 713-721 ◽  
Author(s):  
Nabil Nemer ◽  
Nasri S. Kawar ◽  
Linda Kfoury ◽  
Brigitte Frerot

AbstractThe cedar web-spinning sawfly, Cephalcia tannourinensis Chevin (Hymenoptera: Pamphiliidae), is a pest that has been causing serious damage to cedar (Cedrus libani) forests in Lebanon since 1990. The existence of a sex pheromone was shown in field experiments in a cedar forest in Lebanon and in laboratory tests in olfactometers with and without airflow. More males were caught in traps baited either with virgin females or with a hexane extract of the whole female body than in traps baited either with males alone or with mixed males and females. Male and female C. tannourinensis were active during the day. Mating and pheromone production were observed to occur during midday hours (1000–1400) in the field and under laboratory conditions. Olfactometer tests with extracts prepared from different body parts of the female indicated that the pheromone is produced in the abdominal region, and tests with different dilutions of female extract showed that the male response is dose-dependent.


1939 ◽  
Vol 30 (3) ◽  
pp. 295-301 ◽  
Author(s):  
H. S. Leeson

From January to July 1938, experiments with Anopheles maculipennis race atroparvus, Van Thiel, were undertaken to discover whether humidity and the age at which the females fed influenced their longevity. It was found that they lived longer at higher than at lower humidities; that most of the females which fed did so in the first three days; that those which fed on the second day after emergence lived longer than those which fed at other ages; and that the feeding period was slightly extended in the later experiments, though only a small proportion lived long enough to take their first blood meals on the fourth and fifth days.Rather more than 50 per cent. of each batch of newly emerged adults were females.A large proportion of the deaths of unfed males and females occurred during the first three days, most of them on the second day; this mortality decreased in successive experiments.Culex fatigans, Wied., behaved similarly.


2008 ◽  
pp. 3139-3142
Author(s):  
Norman C. Leppla ◽  
Bastiaan M. Drees ◽  
Allan T. Showler ◽  
John L. Capinera ◽  
Jorge E. Peña ◽  
...  

2018 ◽  
Vol 101 (2) ◽  
pp. 189-194 ◽  
Author(s):  
Tao Ma ◽  
Yuanyuan Zhang ◽  
Shengkun Wang ◽  
Laijiao Lan ◽  
Na Lin ◽  
...  

2020 ◽  
Vol 65 ◽  
pp. 259-267 ◽  
Author(s):  
Karolis Petkevicius ◽  
Christer Löfstedt ◽  
Irina Borodina

1984 ◽  
Vol 39 (11-12) ◽  
pp. 1155-1158 ◽  
Author(s):  
Eloi S. Garcia ◽  
Patricia de Azambuja ◽  
Hans Forster ◽  
Heinz Rembold

Abstract The antifeedant and antimolting effects of azadirachtins A and B, and of a synthetic derivative, 7-acetyl-azadirachtin A were examined. Given through a blood meal, the effective dose (ED50) for the antifeedant effect ranged from 25 to 30 μg/ml of blood for the three compounds. ATP, a phagostimulant, also given orally, reversed the antifeedant action of azadirachtin A. The ED50 values for molt inhibition were 0.04, 0.015, and 0.45 (μg/ml of blood for azadirachtins A, B, and 7-acetyl-azadirachtin A, respectively. The mechanisms of feeding and molt inhibitions by azadirachtin A B, and its 7-acetyl-azadirachtin A derivative are discussed.


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