Horner’s syndrome in dogs and cats – a review

2019 ◽  
Vol XXIV (138) ◽  
pp. 34-40
Author(s):  
Laís Limeira Rodrigues ◽  
Leonardo Pereira Mesquita ◽  
Denis Alberto Zanatto ◽  
Paulo César Maiorka

In dogs and cats, Horner's syndrome is characterized by a group of clinical signs arising from an ipsilateral dysfunction of sympathetic innervation of the eye and its appendages. These signs include ptosis, miosis, anisocoria, prolapse of the third eyelid, and enophtalmos. Lesions that may affect directly or indirectly the sympathetic innervation of the eye, such as trauma, neoplasms, iatrogenic lesions, otitis, encephalitis, among others, can induce HS. The aim of the present study was to review the anatomical and physiological characteristics of sympathetic innervation for the ocular globe, with emphasis on the description of the peculiarities of HS, including its etiology, clinical signs, diagnostic tools, as well as its possible treatments and prognosis.

2009 ◽  
Vol 11 (8) ◽  
pp. 714-716 ◽  
Author(s):  
Luisa De Risio ◽  
James Fraser McConnell

This case report describes the clinical and magnetic resonance imaging (MRI) findings of a 3.5-year-old, male neutered, domestic shorthair cat with second order Horner's syndrome as the only clinical abnormality. The neuroanatomical pathway of the sympathetic innervation to the eye, differential diagnoses for Horner's syndrome in cats, and the interpretation of pharmacological testing are reviewed. The unusual MRI findings and the value of fat-suppressed MRI sequences are discussed.


2016 ◽  
Vol 2016 ◽  
pp. 1-3
Author(s):  
Nicholas S. Mastronikolis ◽  
Sofia P. Spiliopoulou ◽  
Vassiliki Zolota ◽  
Theodoros A. Papadas

Horner’s syndrome is characterized by a combination of ipsilateral miosis, blepharoptosis, enophthalmos, facial anhidrosis, and iris heterochromia in existence of congenital lesions. The syndrome results from a disruption of the ipsilateral sympathetic innervation of the eye and ocular adnexa at different levels. Though rare, thyroid and neck surgery could be considered as possible causes of this clinical entity. We present a case of Horner’s syndrome in a patient after total thyroidectomy and neck dissection for medullary thyroid cancer with neck nodal disease and attempt a brief review of the relevant literature.


2021 ◽  
Vol 14 (2) ◽  
pp. e234973
Author(s):  
Saadat Ali Saleemi ◽  
Ramesh Sahathevan

Horner’s syndrome results from interruption of the sympathetic innervation to the eye. This interruption may occur at three anatomical levels along the sympathetic trunk pathway. There are numerous causes of Horner’s syndrome, including injury to the carotid artery, of which arterial dissection is the commonest pathology. Occlusive carotid disease secondary to atherosclerosis is a relatively rare cause of Horner’s syndrome. We describe a patient with Horner’s syndrome due to complete occlusion of the ipsilateral internal carotid artery.


2013 ◽  
Vol 49 (3) ◽  
pp. 160-168 ◽  
Author(s):  
Rebecca E. Spivack ◽  
A. Derrell Elkins ◽  
George E. Moore ◽  
Gary C. Lantz

The medical records for 133 total ear canal ablations combined with lateral bulla osteotomies (TECA-LBOs) performed on 82 dogs (121 ears) and 11 cats (12 ears) between 2004 and 2010 were reviewed to determine if the duration of preoperative clinical signs was associated with the incidence of postoperative facial nerve injury and Horner's syndrome. Other perioperative complications, such as a head tilt, nystagmus, incisional drainage, draining tracts, hearing loss, as well as bacterial culture results, were noted. Postoperative facial nerve paresis occurred in 36 of 133 ears (27.1%), and paralysis occurred in 29 of 133 ears (21.8%), with no significant difference between species. Thus, postoperative facial nerve deficits occurred in 48.9% of ears. The median duration of clinically evident temporary facial nerve deficits was 2 wk for dogs and 4 wk for cats. Dogs had a significantly longer duration of preoperative clinical signs and were less likely than cats to have a mass in the ear canal. Dogs were less likely to have residual (> 1 yr) postoperative facial nerve deficits. The incidence of postoperative Horner's syndrome was significantly higher in cats than dogs. The duration of preoperative clinical signs of ear disease was not associated with postoperative facial nerve deficits.


2014 ◽  
Vol 3 (2) ◽  
pp. 150
Author(s):  
Alladi Mohan ◽  
J Harikrishna ◽  
BRaga Deepthi ◽  
VArun Raja ◽  
P Suneetha ◽  
...  

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