Why Do Students Become Cyberbullies? Elucidating the Contributions of Specific Developmental Risks to Cyberbullying

Cyberbullying is currently considered as a widespread problem among children and adolescents; in particular, the risks of cyberbullying have recently been examined. The empirical analyses of the present work are based on data from a German longitudinal study. The self-reports of adolescents from Dortmund and Nuremberg on both cyberbullying and individual and contextual characteristics were taken into account. The two-wave panel encompasses N = 871 adolescents (44.5% male); the average age was M = 15.1 years (SD = 0.83) at t1. Data on cyberbullying refer to sending insults or threats to others via the Internet, spreading rumours or talking badly about others via the Internet, and sending private e-mails, photos or similar from others in order to embarrass or ridicule the persons concerned. Other characteristics relate to single aspects of psychopathy (egocentric egotism, low self-control, empathy deficits), acceptance of violence, and delinquent peers. The path analytical findings illustrate the predictive relationships between both individual and contextual risks and cyberbullying in adolescence. The empirical results are discussed, among others, from the perspective of developmental and life-course criminology.

Cognitive and Affective Empathy in Huntington’s Disease

Background: Empathy is a multidimensional construct and a key component of social cognition. In Huntington’s disease (HD), little is known regarding the phenomenology and the neural correlates of cognitive and affective empathy, and regarding how empathic deficits interact with other behavioral and cognitive manifestations. Objective: To explore the cognitive and affective empathy disturbances and related behavioral and neural correlates in HD. Methods: Clinical and sociodemographic data were obtained from 36 healthy controls (HC) and 54 gene-mutation carriers (17 premanifest and 37 early-manifest HD). The Test of Cognitive and Affective Empathy (TECA) was used to characterize cognitive (CE) and affective empathy (AE), and to explore their associations with grey matter volume (GMV) and cortical thickness (Cth). Results: Compared to HC, premanifest participants performed significantly worse in perspective taking (CE) and empathic distress (AE). In symptomatic participants, scores were significantly lower in almost all the TECA subscales. Several empathy subscales were associated with the severity of apathy, irritability, and cognitive deficits. CE was associated with GMV in thalamic, temporal, and occipital regions, and with Cth in parietal and temporal areas. AE was associated with GMV in the basal ganglia, limbic, occipital, and medial orbitofrontal regions, and with Cth in parieto-occipital areas. Conclusion: Cognitive and affective empathy deficits are detectable early, are more severe in symptomatic participants, and involve the disruption of several fronto-temporal, parieto-occipital, basal ganglia, and limbic regions. These deficits are associated with disease severity and contribute to several behavioral symptoms, facilitating the presentation of maladaptive patterns of social interaction.

Investigating the Contribution of White Matter Hyperintensities and Cortical Thickness to Empathy in Neurodegenerative and Cerebrovascular Diseases

Introduction: Change in empathy is an increasingly recognised symptom of neurodegenerative diseases and contributes to caregiver burden and patient distress. Empathy impairment has been associated with brain atrophy but its relationship to white matter hyperintensities (WMH) is unknown. We aimed to investigate the relationships amongst WMH, brain atrophy, and empathy deficits in neurodegenerative and cerebrovascular diseases. Methods: 513 participants with Alzheimers Disease/Mild Cognitive Impairment, Amyotrophic Lateral Sclerosis, Frontotemporal Dementia (FTD), Parkinsons Disease, or Cerebrovascular Disease (CVD) were included. Empathy was assessed using the Interpersonal Reactivity Index. WMH were measured using a semi-automatic segmentation and FreeSurfer was used to measure cortical thickness. Results: A heterogeneous pattern of cortical thinning was found between groups, with FTD showing thinning in frontotemporal regions and CVD in left superior parietal, left insula, and left postcentral. Results from both univariate and multivariate analyses revealed that several variables were associated with empathy, particularly cortical thickness in the fronto-insulo-temporal and cingulate regions, sex(female), global cognition, and right parietal and occipital WMH. Conclusions: Our results suggest that cortical atrophy and WMH may be associated with empathy deficits in neurodegenerative and cerebrovascular diseases. Future work should consider investigating the longitudinal effects of WMH and atrophy on empathy deficits in neurodegenerative and cerebrovascular diseases.

Dissociating cognitive and affective empathy in unipolar depression

One aspect of higher order social cognition is empathy, a psychological construct comprising a cognitive (i.e., recognizing emotions) and an affective (i.e., responding to emotions) component. Empathy deficits have been linked to executive and emotion regulation dysfunction in depression that might underlie a patient’s interpersonal difficulties. However, most studies measure empathy through the Interpersonal Reactivity Index (IRI), which may not accurately capture affective empathy. Critically, prior studies have not examined a potential dissociation between cognitive and affective empathy in depressed individuals. In Study 1, a factor analysis determined the appropriateness of using the IRI to measure affective empathy. Results showed poor model fit with the affective empathy factor. In Study 2, we developed a behavioral measure of empathy, and examined differences in empathic processing as it relates to depression and ruminative thought. Results revealed differences in empathy between depressed and healthy controls, as well as differences by level of rumination. We discuss the implications of these findings for depression treatment.

Dissociating cognitive and affective empathy in unipolar depression

One aspect of higher order social cognition is empathy, a psychological construct comprising a cognitive (i.e., recognizing emotions) and an affective (i.e., responding to emotions) component. Empathy deficits have been linked to executive and emotion regulation dysfunction in depression that might underlie a patient’s interpersonal difficulties. However, most studies measure empathy through the Interpersonal Reactivity Index (IRI), which may not accurately capture affective empathy. Critically, prior studies have not examined a potential dissociation between cognitive and affective empathy in depressed individuals. In Study 1, a factor analysis determined the appropriateness of using the IRI to measure affective empathy. Results showed poor model fit with the affective empathy factor. In Study 2, we developed a behavioral measure of empathy, and examined differences in empathic processing as it relates to depression and ruminative thought. Results revealed differences in empathy between depressed and healthy controls, as well as differences by level of rumination. We discuss the implications of these findings for depression treatment.

The association between toddlerhood empathy deficits and antisocial personality disorder symptoms and psychopathy in adulthood

The present study examined empathy deficits in toddlerhood (age 14 to 36 months) as predictors of antisocial personality disorder (ASPD) symptoms and psychopathy measured by the Levenson Self-Report Psychopathy scale (Levenson, Kiehl, & Fitzpatrick, 1995) in adulthood (age 23 years) in 956 individuals from the Colorado Longitudinal Twin Study. Consistent with the hypothesis that antisocial behavior is associated with “active” rather than “passive” empathy deficits, early disregard for others, not lack of concern for others, predicted later ASPD symptoms. Early disregard for others was also significantly associated with factor 1 of the Levenson Self-Report Psychopathy Scale, which includes items assessing interpersonal and affective deficits, but not with factor 2, which includes items assessing impulsivity and poor behavioral control. The association between early disregard for others and psychopathy factor 2 was near zero after controlling for the shared variance between psychopathy factors 1 and 2. These results suggest that there is a propensity toward adulthood ASPD symptoms and psychopathy factor 1 that can be assessed early in development, which may help identify individuals most at risk for stable antisocial outcomes.