STRENTHENING BY DIVIDING: TREATMENT OF GRADE III FURCATION INVOLVEMENT WITH PLATELET RICH FIBRIN

The modern dentistry has provided opportunity to the patient to increase the longevity of functional dentition. Therapeutic measures may vary in the complexity of the teeth. The treatment may involve combination of restorative dentistry, endodontics, and periodontics

Recombinant human PDGF-BB in combination with mineralized freeze-dried bone allograft in the treatment of Grade II furcation involvement: A case report

Recombinant human platelet-derived growth factor-BB (rhPDGF-BB) is commercially available biomaterial that can be used to regenerate the lost periodontal structure due to progression of periodontitis. The present case describes the surgical treatment of localized periodontitis with furcation Grade II involvement using platelet-derived growth factor BB (growth-factor enhanced matrix 21S) in combination with particulate allograft bone (mineralize freeze-dried bone allograft [FDBA]).This case report showed complete furcation closure after using rhPDGF-BB in combination with FDBA to treat localized periodontitis case with Grade II furcation involvement.

Bicuspidisation: Two is Better than None - A Case Report

A periodontally compromised tooth with severe furcation involvement may as well be retained of their roots rather than its removal in toto. Bicuspidisation (Premolarisation) is a surgical technique of sectioning mandibular molar roots with their respective crown portions followed by rehabilitation with fixed prosthesis in the individual segments. This not only eliminates furcation involvement but also facilitates effective oral hygiene maintenance. The present case report demonstrates the successful management of grade III furcation involved mandibular molar by bicuspidisation followed by subsequent prosthodontic rehabilitation. It gives a better alternative to salvage a periodontally compromised tooth rather than opting for extraction.

Effects of DSPP Gene Mutations on Periodontal Tissues

Dentin sialophosphoprotein (DSPP) gene mutations cause autosomal dominantly inherited diseases. DSPP gene mutations lead to abnormal expression of DSPP, resulting in a series of histological, morphological, and clinical abnormalities. A large number of previous studies demonstrated that DSPP is a dentinal-specific protein, and DSPP gene mutations lead to dentin dysplasia and dentinogenesis imperfecta. Recent studies have found that DSPP is also expressed in bone, periodontal tissues, and salivary glands. DSPP is involved in the formation of the periodontium as well as tooth structures. DSPP deficient mice present furcation involvement, cementum, and alveolar bone defect. We speculate that similar periodontal damage may occur in patients with DSPP mutations. This article reviews the effects of DSPP gene mutations on periodontal status. However, almost all of the research is about animal study, there is no evidence that DSPP mutations cause periodontium defects in patients yet. We need to conduct systematic clinical studies on DSPP mutation families in the future to elucidate the effect of DSPP gene on human periodontium.

RADIOGRAPHIC IMAGE OF PERIODONTAL DISEASE ON MALE DOMESTIC SHORT HAIR CAT FROM UNIVERSITAS PADJADJARAN JATINANGOR CAMPUS

Periodontal disease is the most common disease affecting pet animals including cats and dogs. This disease can cause pain and discomfort affecting the animal’s life quality. The purpose of this study is to evaluate periodontal disease from dental radiographs taken from male domestic short hair cats. Eight male domestic short hair cats from Universitas Padjadjaran Jatinangor campus were recruited for the study. This study observed several changes within the periodontium from dental radiography with simple quantification and analyzed using descriptive analysis. Dental radiographs were taken on the second right mandibula premolar (408). Several changes found in the dental radiography including widening of the periodontal ligament, bone loss and furcation involvement. This study shows varied changes in the periodontium due to the cat’s immune responses. The examined cats were experiencing widening of the periodontal ligament, bone loss and furcation involvement (37.5%, 25% and 87.5%) respectively. All cats examined were experiencing periodontal disease.