Stimulation of oxidative metabolism by thyroid hormones in propranolol/alloxan-treated bony fish,Anabas testudineus (Bloch)

1993 ◽  
Vol 266 (2) ◽  
pp. 85-91 ◽  
Author(s):  
M. C. Subash Peter ◽  
Oommen V. Oommen
Keyword(s):  
Thyroid Hormones ◽  
Oxidative Metabolism ◽  
Bony Fish ◽  
Anabas Testudineus ◽  
Stimulation Of

Adrenal hormones and oxidative metabolism of the garden lizard (Calotes versicolor)

1983 ◽  
Vol 99 (2) ◽  
pp. 211-216 ◽  
Author(s):  
B. B. Pd. Gupta ◽  
J. P. Thapliyal
Keyword(s):  
Oxygen Consumption ◽  
Thyroid Hormones ◽  
Oxidative Metabolism ◽  
Whole Body ◽  
Endocrine Glands ◽  
Adrenal Hormones ◽  
Calotes Versicolor ◽  
Specific Tissue ◽  
Almost All ◽  
Stimulation Of

Daily administration of adrenaline over a 10-day period invariably induced significant increases in the metabolic rate of the whole body and of specific tissue (liver, muscle, kidney and brain) of both intact and thyroidectomized lizards except during June (breeding season) when the presence of thyroid hormones was a prerequisite for the stimulation of oxygen consumption by the whole body, muscle, kidney and brain but not by the liver. Corticosterone had no effect on whole body oxygen consumption but stimulated, inhibited or was without influence on the oxygen consumption of individual tissues, depending on the season and the presence or absence of thyroid hormones. It is suggested that adrenaline, due to its temperature-independent calorigenic effect, acts as the emergency hormone for energy release and helps the animal to survive during hibernation (winter months) when almost all the endocrine glands are inactive.

Mechanism of increase of myocardial oxygen uptake produced by catecholamines1

1965 ◽  
Vol 209 (5) ◽  
pp. 913-918 ◽  
Author(s):  
Francis J. Klocke ◽  
Gerard A. Kaiser ◽  
John Ross ◽  
Eugene Braunwald
Keyword(s):  
Cardiac Arrest ◽  
Oxygen Uptake ◽  
Oxidative Metabolism ◽  
Beating Heart ◽  
Canine Heart ◽  
Direct Stimulation ◽  
Before And After ◽  
Heart Preparation ◽  
Hemodynamic Performance ◽  
Stimulation Of

The relative roles of augmented hemodynamic performance and direct stimulation of oxidative metabolism in mediating the increase of myocardial oxygen uptake (MVo2) produced by catecholamines have been examined in an isolated canine heart preparation. The responses of MVo2 to graded doses of isoproterenol, norepinephrine, or epinephrine were determined before and after the induction of cardiac arrest with potassium. Although increases of MVo2 occurred in the arrested state with the larger doses of the amines, they constituted only a small fraction, generally between 5 and 20%, of the increases produced by the same doses of amines when the hearts were beating. It is concluded that while large doses of catecholamines can increase oxidative metabolism of the nonbeating heart by a small amount, the increases of MVo2 produced by catecholamines in the beating heart are due in large part to the hemodynamic alterations which the amines induce.

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