Sedative-Hypnotics and Anxiolytics

2010 ◽  
pp. 511-523 ◽  
Author(s):  
Bachaar Arnaout ◽  
Ismene L. Petrakis
Keyword(s):  
Sedative Hypnotics

Sedative-hypnotics and human performance

2013 ◽  
Author(s):  
Laverne C. Johnson ◽  
Doris A. Chernik
Keyword(s):  
Human Performance ◽  
Sedative Hypnotics

Stimulant and depressant properties of sedative-hypnotics in mice selectively bred for differential sensitivity to ethanol

1983 ◽  
Vol 82 (1-2) ◽  
pp. 46-51 ◽  
Author(s):  
Bruce C. Dudek ◽  
Michael E. Abbott ◽  
Tamara J. Phillips
Keyword(s):  
Differential Sensitivity ◽  
Sedative Hypnotics

Sedative Hypnotics

2006 ◽  
Vol 4 (4) ◽  
pp. 472-479
Author(s):  
Milton Erman
Keyword(s):  
Sedative Hypnotics

834 Help Me Stop Eating Please…

SLEEP ◽  
2021 ◽  
Vol 44 (Supplement_2) ◽  
pp. A325-A325
Author(s):  
Ahmad Arslan ◽  
Mazen El Ali ◽  
Charles Atwood
Keyword(s):  
Selective Serotonin Reuptake Inhibitors ◽  
Food Allergies ◽  
Sleep Diary ◽  
Safety Measures ◽  
Obstructive Sleep ◽  
History Of ◽  
Sedative Hypnotics ◽  
Body Image Issues ◽  
Choice Of Therapy ◽  
Living Area

Abstract Introduction Sleep related eating disorder (SRED) is an uncommon NREM parasomnia, predominantly seen in females in their 20s and 30s and is commonly associated with sleep walking, daytime eating disorders (anorexia and bulimia) and use of certain sedative-hypnotics. Here, we describe a case of unique therapeutic dilemma in a middle-aged female, in whom SRED affected the use of continuous positive airway pressure device (CPAP) for her obstructive sleep apnea (OSA) which lead to worsening sleep quality and daytime functionality. Report of case(s) 42-year-old female with history of migraines and OSA, came to the clinic with 6-month history of SRED which started after the demise of her husband and was associated with significant weight gain and injuries. During typical episode, she would take CPAP mask off, walk down the stairs and eat in the kitchen area and then fall asleep in the living area. She denied predilection for particular food, food allergies, consumption of inedible materials or use of offending drugs. Upon further inquiry, she reported recent unsuccessful attempts at smoking cessation, frequent nighttime smoking, significant body image issues and discontinuation of topiramate use for her migraines recently. Telemedicine visit limited the physical examination but included increase in BMI from 32 to 35 kg/m2. Compliance report, sleep diary and surveillance videos were consistent with the history with compliance report showing 17% of nights with optimal usage and 59% of nights with any usage. Beside locking the food, safety measures and napping, we recommended her to use clonazepam 1.5 mg. Unfortunately, she continued to have episodes. She was then started on Topiramate 50 mg which was gradually increased to 100 mg with reduction in her episodes and increase in her compliance to 60%. Conclusion In sleep medicine, treatment of OSA usually precede the treatment of comorbid conditions. However, SRED with comorbid OSA, requires a reverse strategy where improvement in compliance and coexisting daytime symptoms requires the treatment of SRED first. Moreover, detailed history to investigate the potential precipitating factors, co-existing medical and sleep conditions can help with choice of therapy e.g. patients with depression and SRED can benefit from Selective Serotonin Reuptake Inhibitors rather than other choices. Support (if any):

scholarly journals The Chicken and/or the Egg?

2019 ◽  
pp. 555-570
Author(s):  
Ketan Deoras ◽  
Jonathan Oliver ◽  
Mita S. Deoras
Keyword(s):  
Cognitive Behavioral Therapy ◽  
Sleep Disturbance ◽  
Behavioral Therapy ◽  
Cognitive Behavioral ◽  
Chronic Insomnia ◽  
Comorbid Insomnia ◽  
Time Treatment ◽  
Bidirectional Relationship ◽  
Prevalence Of Depression ◽  
Sedative Hypnotics

This chapter covers the bidirectional relationship between depression and insomnia. Patients with insomnia are more likely to develop depression; the prevalence of depression in people with comorbid insomnia is almost 10 times greater than in those without insomnia. Conversely, depression itself has sleep disturbance as a symptom 80% of the time. Treatment of comorbid insomnia and depression should aim at treating both conditions. While cognitive-behavioral therapy for insomnia (CBT-i) should always be considered in the treatment of chronic insomnia, instances may arise when medications are required. Sedative–hypnotics derive from a wide variety of classes of medications and may need to be used in conjunction with antidepressants in the depressed insomniac.

scholarly journals Drug-Drug Interaction between Psychiatric Medications and Experimental Treatments for Coronavirus Disease-19: A Mini-Review

2020 ◽  
Vol 8 (T1) ◽  
pp. 216-228
Author(s):  
Hananeh Baradaran ◽  
Nazanin Gorgzadeh ◽  
Houman Seraj ◽  
Anahita Asadi ◽  
Danial Shamshirian ◽  
...  
Keyword(s):  
Mental Illness ◽  
Drug Interaction ◽  
Severe Acute Respiratory Syndrome ◽  
Clinical Care ◽  
Mood Stabilizers ◽  
Care Staff ◽  
Experimental Drugs ◽  
The World ◽  
Sedative Hypnotics ◽  
Experimental Treatments

The pandemic of coronavirus disease (COVID)-2019 has been affected many people all around the world. Patients with mental disorders are not as safe as others; also, they might be more vulnerable in such situations. These patients take various medications, which can lead to numerous drug-drug interactions with experimental drugs uses against COVID-19. According to the potential critical interactions, we reviewed the reputable databases to find the interactions between main categories of psychiatric medications (e.g., antidepressants, anti-psychotics, sedative/hypnotics, and mood stabilizers) when used in concomitant with COVID-19 experimental agents (e.g., hydroxychloroquine, lopinavir/ritonavir, atazanavir, and chloroquine). We hope the list provided in this review helps the clinical care staff in treating patients with mental illness infected with severe acute respiratory syndrome coronavirus 2 during the COVID-19 pandemic.

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