Exercise-induced ventricular tachycardia

1982 ◽  
Vol 2 (1) ◽  
pp. 51-55 ◽  
Author(s):  
Bruce S. Alpert ◽  
John Boineau ◽  
William B. Strong
1992 ◽  
Vol 70 (9) ◽  
pp. 875-878 ◽  
Author(s):  
Gilles E. O'Hara ◽  
Pedro Brugada ◽  
Luz-Maria Rodriguez ◽  
Mitermayer Brito ◽  
Luis Mont ◽  
...  

2018 ◽  
pp. 199-228
Author(s):  
Gregory S. Thomas ◽  
Maryam Balouch

The chapter Rhythm and Conduction Disturbances in Stress Testing reviews the frequency and significance of arrhythmias and conduction abnormalities precipitated by exercise. Case examples are provided. PVCs occurring prior to, during exercise, or during recovery all modestly increase the risk of all-cause mortality in patients with and without known coronary artery disease (CAD). Ventricular tachycardia and premature ventricular complexes are often not reproducible on a subsequent exercise test. Exercise induced left bundle branch block (LBBB) predicts increased risk of the presence of CAD, all cause mortality, and often permanent LBBB. Differentiating wide complex tachycardia during exercise testing between supraventricular tachycardia and ventricular tachycardia can be challenging. The Wellens, Brugada, and Vereckei algorithms to distinguish between these arrhythmias are detailed and compared.


2020 ◽  
Vol 30 (7) ◽  
pp. 1039-1042
Author(s):  
Utkarsh Kohli ◽  
Lisa Kuntz ◽  
Hemal M. Nayak

AbstractCatecholaminergic polymorphic ventricular tachycardia is a rare (prevalence: 1/10,000) channelopathy characterised by exercise-induced or emotion-triggered ventricular arrhythmias. There is an overall paucity of genotype-phenotype correlation studies in patients with catecholaminergic polymorphic ventricular tachycardia, and in vitro and in vivo effects of individual mutations have not been well characterised. We report an 8-year-old child who carried a mutation in the coding exon 8 of RYR2 (p.R169L) and presented with emotion-triggered sudden cardiac death. He was also found to have left ventricular hypertrophy, a combination which has not been reported before. We discuss the association between genetic variation in RYR2, particularly mutations causing replacement of arginine at position 169 of RYR2 and structural cardiac abnormalities.


CHEST Journal ◽  
1991 ◽  
Vol 99 (5) ◽  
pp. 1295-1296 ◽  
Author(s):  
Harry Feld ◽  
Victor Guadanino ◽  
Gerald Hollander ◽  
Alvin Greengart ◽  
Edgar Lichstein ◽  
...  

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