Metabolism of14C-abscisic acid during its IAA-promoted transport in etiolated segments of pea (Pisum sativum L.) epicotyls†

1984 ◽  
Vol 26 (2) ◽  
pp. 108-112 ◽  
Author(s):  
V. Borkovec ◽  
Hanno Lehmann ◽  
S. Prochazka
Author(s):  
Wifak A Al-Kaisy ◽  
Sahar F Mahadi

A field experiment was conducted at botanical garden of Department of Biology, College of Education for Pure Science (Ibn Al-Haitham), University of Baghdad, during the growth winter season of 2016-2017 to study the effect of different concentrations (0, 10, 20) mg.L-1 of abscisic acid and (0, 50, 100, 150) mg.L-1 of vitamin C and their interaction on some plant hormones of pea plant (Pisum sativum L.). The results showed that ABA 20 mg.L-1 decreased IAA about 27.44%, GA3 about 19.73% and Kinetin 15.37% while vitamin C with 150 mg.L-1 increased IAA 27.43%, GA3 45.31% and Kinetin 58.53%, but ABA increased about 23.01% for ABA and 34.93% for vitamin C compared with control plants. The interaction between them is significant for four plant hormones.


1987 ◽  
Vol 65 (5) ◽  
pp. 907-915 ◽  
Author(s):  
Arlette Nougarède ◽  
Pierre Rondet ◽  
Pierre Landré ◽  
Jacques Rembur

When applied to pea (Pisum sativum L. var. nain hâtif d'Annonay) cotyledonary bud at the time of decapitation, cis-abscisic acid (ABA) induced a decrease in the mitotic indices of the meristem and subapical regions of the bud, in comparison with decapitated but not ABA treated control plants. ABA treatment delayed the entry of the G0–1 nuclei of the inhibited bud into the S phase and then into mitosis. Whatever the ABA concentration (10−3 to 10−7 M), the overall growth of the bud and the elongation of the cortical cells in its subapical region were not affected during the first 48 h. ABA induced an inhibition of bud elongation, all the more important since it was applied later after decapitation. When applied on a 48-h released bud, ABA induced an inhibition of elongation of the bud, a decrease in its mitotic activity, as well as a concentration of the shoot apex nuclei in the G1 phase of the cell cycle. However, this exogenous ABA treatment could not entirely replace the main axis in maintaining the inhibited state of the bud.


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