Aortic stenosis with marked right ventricular hypertrophy

1966 ◽  
Vol 17 (2) ◽  
pp. 273-277 ◽  
Author(s):  
George S. Husson ◽  
Marie S. Blackman
Keyword(s):  
Aortic Stenosis ◽  
Right Ventricular ◽  
Ventricular Hypertrophy ◽  
Right Ventricular Hypertrophy

Lactate dehydrogenase changes following several cardiac hypertrophic stresses

1976 ◽  
Vol 40 (6) ◽  
pp. 923-926 ◽  
Author(s):  
J. W. York ◽  
D. G. Penney ◽  
T. A. Weeks ◽  
P. A. Stagno
Keyword(s):  
Lactate Dehydrogenase ◽  
Left Ventricle ◽  
Left Ventricular Hypertrophy ◽  
Aortic Stenosis ◽  
Right Ventricle ◽  
Right Ventricular ◽  
Ventricular Hypertrophy ◽  
Left Ventricular ◽  
Right Ventricular Hypertrophy ◽  
Ldh Activity

The effects of four different cardiac hypertrophic stresses on cardiac lactate dehydrogenase (LDH) isozyme composition and activity were examined. Altitude-induced right ventricular hypertrophy was accompanied by an increase of 10% in the M subunit of LDH in right ventricle, left ventricle, and atria. Left ventricular hypertrophy induced by aortic stenosis also produced isozyme changes in the ventricles, but of only half the magnitude. Biventricular hypertrophy, induced by running or swimming, was accompanied by 4–5% increases in M LDH in the ventricles only. We conclude that changes in LDH activity are directly related to changes in the M subunit in all three portions of the heart. No changes in H subunit were noted under any of the stresses. It appears that the magnitude of changes in cardiac LDH isozyme composition are only marginally related to extent of hypertrophy.

Left and right ventricular hypertrophy at autopsy of hypertensive individuals

2012 ◽  
Vol 58 (1) ◽  
pp. 41-47 ◽  
Author(s):  
Mirella Pessoa Sant’Anna ◽  
Roberto José Vieira de Mello ◽  
Luciano Tavares Montenegro ◽  
Mônica Modesto Araújo
Keyword(s):  
Right Ventricular ◽  
Ventricular Hypertrophy ◽  
Right Ventricular Hypertrophy ◽  
Left And Right

Role of microRNA-21 in right ventricular hypertrophy and fibrosis

Pneumologie ◽  
2012 ◽  
Vol 66 (06) ◽  
Author(s):  
J Neumann ◽  
W Janssen ◽  
B Kojonazarov ◽  
C Döbele ◽  
HA Ghofrani ◽  
...  
Keyword(s):  
Right Ventricular ◽  
Ventricular Hypertrophy ◽  
Right Ventricular Hypertrophy

scholarly journals RNA Expression Profiles and Regulatory Networks in Human Right Ventricular Hypertrophy due to High Pressure Load

iScience ◽  
2021 ◽  
pp. 102232
Author(s):  
Philippe Chouvarine ◽  
Joachim Photiadis ◽  
Robert Cesnjevar ◽  
Jens Scheewe ◽  
Ulrike MM. Bauer ◽  
...  
Keyword(s):  
High Pressure ◽  
Right Ventricular ◽  
Regulatory Networks ◽  
Ventricular Hypertrophy ◽  
Expression Profiles ◽  
Right Ventricular Hypertrophy ◽  
Rna Expression ◽  
Human Right ◽  
Pressure Load

Transient severe isolated right ventricular hypertrophy in neonates

2003 ◽  
Vol 13 (4) ◽  
pp. 384-386 ◽  
Author(s):  
Munesh Tomar ◽  
Sitaraman Radhakrishnan ◽  
Savitri Shrivastava
Keyword(s):  
Pulmonary Hypertension ◽  
Right Ventricular ◽  
Ventricular Function ◽  
Ventricular Hypertrophy ◽  
Pressure Overload ◽  
Posterior Wall ◽  
Right Ventricular Hypertrophy ◽  
Pulmonary Vasculature ◽  
Normal Thickness ◽  
The Right

We report two instances of transient isolated right-sided myocardial hypertrophy in patients with an intact ventricular septum, normal thickness of the posterior wall of the left ventricle, and normal ventricular function, diagnosed by echocardiography on the third day of life. The two neonates, born at 36 and 38 weeks gestation respectively, had perinatal distress. Both were diagnosed as having isolated right ventricular hypertrophy with mild pulmonary hypertension, which disappeared in both cases within 8 weeks without any specific therapy. Though the cause of the ventricular hypertrophy remains unclear, we believe that it is the consequence of remodeling of pulmonary vasculature secondary to acute perinatal distress, resulting in persistent pulmonary hypertension and producing pressure overload on the right ventricle, and hence right ventricular hypertrophy. The finding of early and transient right ventricular hypertrophy, with normal left-sided structures and normal ventricular function, has thus far failed to gain attention in the paediatric cardiologic literature.

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