Diagnostic tests for Trichinella spiralis infection in pigs. A comparative study of ELISA for specific antibody and histamine release from blood cells in experimental infections

1991 ◽  
Vol 39 (3-4) ◽  
pp. 241-252 ◽  
Author(s):  
P. Lind ◽  
L. Eriksen ◽  
Sv.Aa. Henriksen ◽  
W.L. Homan ◽  
F. van Knapen ◽  
...  
Keyword(s):  
Comparative Study ◽  
Histamine Release ◽  
Specific Antibody ◽  
Blood Cells ◽  
Diagnostic Tests ◽  
Trichinella Spiralis ◽  
Experimental Infections

THE LIFE CYCLE OF TRICHINELLA SPIRALIS: I. THE INTESTINAL PHASE OF DEVELOPMENT

1967 ◽  
Vol 45 (6) ◽  
pp. 1255-1260 ◽  
Author(s):  
C. S. Shanta ◽  
E. Meerovitch
Keyword(s):  
Life Cycle ◽  
Post Infection ◽  
Sexual Maturity ◽  
Trichinella Spiralis ◽  
Seminal Vesicles ◽  
Intestinal Phase ◽  
Experimental Infections

In experimental infections in mice, Trichinella spiralis larvae in the intestines molted twice before reaching sexual maturity. In both sexes, the first molt occurred between 12 and 16 hours post infection; in males, the second molt occurred between 24 and 32 hours, and in females, between 22 and 30 hours. The females were inseminated after the 36th hour post infection, but some males had spermatozoa in the seminal vesicles before the completion of the second molt. Structures, believed to be amphids, were observed after 2 hours post infection; they increased in size up to the 6th hour, after which they regressed and finally disappeared. The function of these amphids is believed to be related to osmoregulation.

Histamine release induced by opioid analgesics: A comparative study using porcine mast cells

1991 ◽  
Vol 33 (1-2) ◽  
pp. 20-22 ◽  
Author(s):  
M. Ennis ◽  
C. Schneider ◽  
E. Nehring ◽  
W. Lorenz
Keyword(s):  
Mast Cells ◽  
Comparative Study ◽  
Histamine Release ◽  
Opioid Analgesics

scholarly journals IMMUNOLOGICAL UNRESPONSIVENESS TO SENSITIZATION WITH SIMPLE CHEMICAL COMPOUNDS

1963 ◽  
Vol 118 (6) ◽  
pp. 1021-1035 ◽  
Author(s):  
Jack R. Battisto ◽  
Merrill W. Chase
Keyword(s):  
Specific Antibody ◽  
Blood Cells ◽  
Guinea Pigs ◽  
White Blood Cells ◽  
Chemical Compounds ◽  
Lymphoid Cells ◽  
Delayed Type Hypersensitivity ◽  
Simple Chemical ◽  
Picryl Chloride

Guinea pigs fed picryl chloride to induce specific immunologic unresponsiveness cleared small amounts of venously infused antipicryl antibody at a rate equal to that of normal guinea pigs. Catabolism of passively administered picryl-specific antibody did not alter the unresponsive state of picryl chloride-fed guinea pigs or the responsive state of normal guinea pigs. Lymphoid cells of picryl chloride immunized guinea pigs produced equal amounts of picryl-specific antibody in picryl chloride-fed and normal animals. Allergen-fed guinea pigs remained unresponsive to attempted sensitization with the allergen in excess of 10 months after the final feeding, though some became feebly sensitive between 9 and 11 months. Second attempts to make unresponsive animals hypersensitive were unsuccessful. White blood cells of guinea pigs unresponsive to picryl chloride were unable to transfer delayed-type hypersensitivity for picryl chloride to normal recipients yet readily transferred tuberculin hypersensitivity.

Hereditary Spherocytosis in the Neonatal Period: A Case Report

2017 ◽  
Vol 36 (5) ◽  
pp. 280-288 ◽  
Author(s):  
Ashley Will ◽  
Cheryl A. Henderson ◽  
Amy J. Jnah ◽  
Desi Newberry
Keyword(s):  
Blood Cells ◽  
Diagnostic Tests ◽  
Deoxyribonucleic Acid ◽  
Hereditary Spherocytosis ◽  
Neonatal Period ◽  
Complete Blood Count ◽  
Hemolytic Disease ◽  
The Third ◽  
Erythrocyte Membrane Proteins ◽  
Severe Hyperbilirubinemia

AbstractHereditary spherocytosis (HS) is the third most common yet most frequently underrecognized, congenitally acquired hemolytic disease of the neonate. Hereditary spherocytosis is caused by a defect of one or more erythrocyte membrane proteins, which leads to an increased rate of destruction of circulating red blood cells. The HS spectrum of symptoms is varied from asymptomatic to intrauterine hydrops. Diagnostic tests range from a complete blood count (CBC) analysis to deoxyribonucleic acid (DNA) sequencing. Management in the neonatal period focuses primarily on associated comorbidities, including the prevention of severe hyperbilirubinemia and anemia. Life span implications of HS include hemolysis, jaundice, anemia, splenomegaly, and periodic gallstones. Early identification and diagnosis of HS is essential to ensure proper monitoring and medical management throughout infancy, childhood, and adulthood.

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