Comparative study of myocardial high energy phosphate substrate content in slow and fast growing chicken and in chickens with heart failure and ascites

2007 ◽  
Vol 148 (1) ◽  
pp. 230-238 ◽  
Author(s):  
A.A. Olkowski ◽  
S. Nain ◽  
C. Wojnarowicz ◽  
B. Laarveld ◽  
J. Alcorn ◽  
...  
Keyword(s):  
Heart Failure ◽  
Comparative Study ◽  
High Energy ◽  
High Energy Phosphate ◽  
Fast Growing

scholarly journals Myocardial High Energy Phosphate Stores in Acutely Induced Hypoxic Heart Failure

1966 ◽  
Vol 19 (2) ◽  
pp. 221-229 ◽  
Author(s):  
PETER E. POOL ◽  
JAMES W. COVELL ◽  
CHARLES A. CHIDSEY ◽  
EUGENE BRAUNWALD
Keyword(s):  
Heart Failure ◽  
High Energy ◽  
High Energy Phosphate ◽  
Hypoxic Heart

scholarly journals Myocardial High Energy Phosphate Stores in Cardiac Hypertrophy and Heart Failure

1967 ◽  
Vol 21 (3) ◽  
pp. 365-374 ◽  
Author(s):  
PETER E. POOL ◽  
JAMES F. SPANN ◽  
ROBERT A. BUCCINO ◽  
EDMUND H. SONNENBLICK ◽  
EUGENE BRAUNWALD ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Hypertrophy ◽  
High Energy ◽  
High Energy Phosphate

HEART FAILURE IN CONGENITAL THYROTOXICOSIS

PEDIATRICS ◽  
1966 ◽  
Vol 37 (3) ◽  
pp. 460-466
Author(s):  
Cyrus Farrehi ◽  
Marvin Mitchell ◽  
David M. Fawcett
Keyword(s):  
Heart Failure ◽  
Thyroid Hormone ◽  
Neonatal Period ◽  
Developmental State ◽  
High Energy ◽  
Myocardial Contraction ◽  
Graves Disease ◽  
High Energy Phosphate ◽  
Myocardial Mitochondria ◽  
Cardiac Decompensation

Congenital thyrotoxicosis, or Graves' disease of the newborn, may be complicated by cardiac decompensation. Both the disease and its complication are self-limited, but they may become severe and endanger the life in the neonatal period. Failure of the heart may be related to interference with formation of high-energy phosphate bonds in the myocardial mitochondria by high levels of thyroid hormone. This phenomenon depletes the source of energy for myocardial contraction. The treatment should be primarily directed toward the hyperthyroidism. The enhanced rate of growth of the hyperthyroid fetus may lead to a developmental state where a small, apparently premature infant has well-formed and well-functioning organs, some aged far in advance of the relatively short intrauterine life. The term "accelerated maturity" is suggested to describe this state.

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