Damage resistance and damage tolerance of dispersed CFRP laminates: Effect of the mismatch angle between plies

2013 ◽  
Vol 101 ◽  
pp. 255-264 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
P. Maimí ◽  
...  

2013 ◽  
Vol 106 ◽  
pp. 96-103 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
J. Costa


2013 ◽  
Vol 106 ◽  
pp. 30-32 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
J. Costa


2013 ◽  
Vol 95 ◽  
pp. 569-576 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
J. Costa


Author(s):  
Andrew T. Rhead ◽  
Shi Hua ◽  
Richard Butler


2006 ◽  
Vol 326-328 ◽  
pp. 1833-1836 ◽  
Author(s):  
Seung Min Jang ◽  
Tadaharu Adachi ◽  
Akihiko Yamaji

The development characteristics of impact-induced damage in carbon-fiber-reinforcedplastics (CFRP) laminates were experimentally studied using a drop-weight impact tester. Five types of CFRP laminates were used to investigate the effect of stacking sequences and thicknesses. The efficiency of absorbed energy to impact energy was different for CFRP laminates with different stacking sequences or thicknesses. The DA/AE ratio of delamination area (DA) to absorbed energy (AE) was almost the same for CFRP laminates with the same stacking sequence regardless of the thickness. We found that the DA/AE ratio could be used as a parameter to characterize the impact damage resistance in CFRP laminates with different stacking sequences.



Author(s):  
R Prabhakaran ◽  
M Saha ◽  
M Douglas ◽  
AT Nettles






Author(s):  
Masafumi Hayashi ◽  
Kenji Keyamura ◽  
Asami Yoshida ◽  
Mariko Ariyoshi ◽  
Genki Akanuma ◽  
...  

In eukaryotes, genomic DNA is packaged into nucleosomes, which are the basal components coordinating both the structures and functions of chromatin. Here we screened a collection of mutation for histone H3/H4 mutants in Saccharomyces cerevisiae that affect the DNA damage sensitivity of DNA damage tolerance (DDT)-deficient cells. We identified a class of histone H3/H4 mutations that suppress MMS sensitivity of DDT-deficient cells (hereafter we refer to as the histone SDD mutations), which likely cluster on a specific H3-H4 interface of the nucleosomes. The histone SDD mutations did not suppress the MMS sensitivity of DDT-deficient cells in the absence of Rad51, indicating that homologous recombination (HR) is responsible for DNA damage resistance. Furthermore, the histone SDD mutants showed reduced levels of PCNA ubiquitination after exposure to MMS or UV irradiation, consistent with a decreased MMS-induced mutagenesis relative to wild-type cells. We also found that histone SDD mutants lacking the INO80 chromatin remodeler impair HR-dependent recovery from MMS-induced replication arrest, resulting in defective S-phase progression and increased Rad52 foci. Taken together, our data provide novel insights into nucleosome functions, which link INO80-dependent chromatin remodeling to the regulation of DDT and HR during the recovery from replication blockage.



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