Identical twins with malrotation and type IV jejunal atresia

1987 ◽  
Vol 22 (11) ◽  
pp. 1015-1016 ◽  
Author(s):  
Laurel Matthews Olson ◽  
L. Suzanne Flom ◽  
Catherine M.P. Kierney ◽  
Dennis W. Shermeta
Keyword(s):  
Identical Twins ◽  
Jejunal Atresia ◽  
Type Iv

scholarly journals Type IV jejunal atresia in a newborn: a rare birth defect presenting with bilious vomiting

2018 ◽  
Vol 6 (1) ◽  
pp. 224
Author(s):  
Pushwinder Kaur
Keyword(s):  
Birth Defect ◽  
Primary Anastomosis ◽  
Abdominal Distension ◽  
Intestinal Lumen ◽  
Abdominal Radiograph ◽  
Proximal Jejunum ◽  
Jejunal Atresia ◽  
Feeding Difficulties ◽  
Type Iv ◽  
Bilious Vomiting

Background: Type IV jejunal atresia is a rare birth defect, which occurs due to late intrauterine vascular accidents that results in complete obstruction of intestinal lumen. There is no genetic predilection. It occurs most commonly in proximal jejunum. Clinical presentation includes bilious vomiting, abdominal distension, feeding difficulties, failure to pass stools and/or absence of bowel movements after birth. Here author report a case of premature newborn, who was admitted in NICU at birth in view of prematurity. As the baby had bilious vomiting and bilious NG aspirate on day 3 of life, possibility of intestinal obstruction was kept. It was diagnosed as a case of jejunal atresia by abdominal radiograph displaying dilated stomach, duodenum and proximal jejunum with gasless abdomen. Further jejunal atresia confirmed by the upper GI study exhibiting the level of obstruction. Surgical resection of the atretic segment and primary anastomosis was done. This case emphasizes the need of rapid identification of jejunal atresia and its adequate management to prevent morbidity and mortality associated with fore shortened intestine.

scholarly journals Type I Jejunal Atresia in Identical Twins: A Rare Occurrence

2017 ◽  
Vol 6 (3) ◽  
pp. 68
Author(s):  
Amrollah Salimi ◽  
Shervin Rashidinia ◽  
Seyed Shahin Eftekhari ◽  
Mahsa Besharati ◽  
Sara Shahmoradi
Keyword(s):  
Intestinal Obstruction ◽  
Identical Twins ◽  
Type I ◽  
Rare Occurrence ◽  
Jejunal Atresia ◽  
Jejunoileal Atresia ◽  
Neonatal Intestinal Obstruction ◽  
Different Types ◽  
Common Etiology

Jejunoileal atresia is of familial and non-familial in origins and classified into four different types. We herein report a rare occurrence of type I jejunal atresia in identical twins who were presented with neonatal intestinal obstruction. This report points towards common etiology of atresia in our cases and factors more than vascular accident appear to be involved.

scholarly journals Jejunal atresia presenting as meconium cyst with polysplenia in a newborn: an extremely rare association

2019 ◽  
Vol 6 (8) ◽  
pp. 3023
Author(s):  
Rashi . ◽  
Kalyani Saha Basu ◽  
Samiran Biswas ◽  
Shibsankar Barman
Keyword(s):  
Rare Condition ◽  
Abdominal Distension ◽  
Proximal Segment ◽  
Large Cyst ◽  
Jejunal Atresia ◽  
Rare Association ◽  
Type Iv

Jejunal atresia (JA) presenting as meconium cyst with polysplenia is an extremely rare condition. We reported a case presenting with abdominal distension and feculent vomiting. Exploration revealed a type IV JA with dilated proximal segment presented as a large cyst filled with air and meconium. Polysplenia diagnosed incidentally.

scholarly journals Colonic and type IV jejunal atresias with apple-peel bowel segment as the content of right inguinal hernia: A case report

2021 ◽  
Vol 10 ◽  
pp. 35
Author(s):  
Deepa P Makhija ◽  
Abhijit Kumar ◽  
Rahul K Gupta ◽  
Beejal V Sanghvi ◽  
Kedar P Mudkhedkar ◽  
...  
Keyword(s):  
Case Report ◽  
Inguinal Hernia ◽  
Case Presentation ◽  
Jejunal Atresia ◽  
Colonic Atresia ◽  
Type Iv ◽  
Bowel Segment ◽  
Apple Peel

Background: Colonic atresia is an uncommon entity. It may be associated with multiple anomalies. Case Presentation: We present a case, which was initially suspected to be jejunal atresia, but was found to have colonic atresia as well intra-operatively. The associated jejunal atresia with apple-peel configuration was present as the content of inguinal hernia. Conclusion: This case report highlights the rarity of the condition and the postoperative issues in the management of such patients.

Type IV jejunal atresia with an unusual variation of enteric duplication: Report of a case

Surgery Today ◽  
2010 ◽  
Vol 41 (1) ◽  
pp. 130-132 ◽  
Author(s):  
Vikal Chandra Shakya ◽  
Chandra Shekhar Agrawal ◽  
Sudeep Khaniya ◽  
Rabin Koirala ◽  
Sagar Raj Pandey ◽  
...  
Keyword(s):  
Jejunal Atresia ◽  
Type Iv ◽  
Enteric Duplication

The LDE-Type Flare Occurrence (1969-1993)

1994 ◽  
Vol 144 ◽  
pp. 279-282
Author(s):  
A. Antalová
Keyword(s):  
Time Series ◽  
Fourier Analysis ◽  
Sunspot Cycle ◽  
List Type ◽  
Type Number ◽  
Solar Cycles ◽  
Type Iv ◽  
Long Duration ◽  
Cycle Maximum ◽  
Flare Index

AbstractThe occurrence of LDE-type flares in the last three cycles has been investigated. The Fourier analysis spectrum was calculated for the time series of the LDE-type flare occurrence during the 20-th, the 21-st and the rising part of the 22-nd cycle. LDE-type flares (Long Duration Events in SXR) are associated with the interplanetary protons (SEP and STIP as well), energized coronal archs and radio type IV emission. Generally, in all the cycles considered, LDE-type flares mainly originated during a 6-year interval of the respective cycle (2 years before and 4 years after the sunspot cycle maximum). The following significant periodicities were found:• in the 20-th cycle: 1.4, 2.1, 2.9, 4.0, 10.7 and 54.2 of month,• in the 21-st cycle: 1.2, 1.6, 2.8, 4.9, 7.8 and 44.5 of month,• in the 22-nd cycle, till March 1992: 1.4, 1.8, 2.4, 7.2, 8.7, 11.8 and 29.1 of month,• in all interval (1969-1992):a)the longer periodicities: 232.1, 121.1 (the dominant at 10.1 of year), 80.7, 61.9 and 25.6 of month,b)the shorter periodicities: 4.7, 5.0, 6.8, 7.9, 9.1, 15.8 and 20.4 of month.Fourier analysis of the LDE-type flare index (FI) yields significant peaks at 2.3 - 2.9 months and 4.2 - 4.9 months. These short periodicities correspond remarkably in the all three last solar cycles. The larger periodicities are different in respective cycles.

Labelling of non-A, non-B hepatitis infected chimpanzee hepatocytes with nuclease-gold conjugates

1984 ◽  
Vol 42 ◽  
pp. 250-251
Author(s):  
G. D. Gagne ◽  
M. F. Miller ◽  
D. A. Peterson
Keyword(s):  
Endoplasmic Reticulum ◽  
Experimental Infection ◽  
Uranyl Acetate ◽  
Type I ◽  
Cellular Injury ◽  
Type Ii ◽  
Tubular Structures ◽  
Type Iii ◽  
Type Iv ◽  
Infected Chimpanzee

Experimental infection of chimpanzees with non-A, non-B hepatitis (NANB) or with delta agent hepatitis results in the appearance of characteristic cytoplasmic alterations in the hepatocytes. These alterations include spongelike inclusions (Type I), attached convoluted membranes (Type II), tubular structures (Type III), and microtubular aggregates (Type IV) (Fig. 1). Type I, II and III structures are, by association, believed to be derived from endoplasmic reticulum and may be morphogenetically related. Type IV structures are generally observed free in the cytoplasm but sometimes in the vicinity of type III structures. It is not known whether these structures are somehow involved in the replication and/or assembly of the putative NANB virus or whether they are simply nonspecific responses to cellular injury. When treated with uranyl acetate, type I, II and III structures stain intensely as if they might contain nucleic acids. If these structures do correspond to intermediates in the replication of a virus, one might expect them to contain DNA or RNA and the present study was undertaken to explore this possibility.

Effects of EDTA, Hyaluronidase and Collagenase on Epiphyseal Cartilage Matrix

1968 ◽  
Vol 26 ◽  
pp. 56-57
Author(s):  
H. Clarke Anderson ◽  
Priscilla R. Coulter
Keyword(s):  
Light And Electron Microscopy ◽  
Matrix Vesicles ◽  
Cartilage Matrix ◽  
Collagen Fibrils ◽  
Epiphyseal Cartilage ◽  
Dense Matrix ◽  
Type Iv ◽  
Bovine Testicular Hyaluronidase ◽  
The Matrix ◽  
Testicular Hyaluronidase

Epiphyseal cartilage matrix contains fibrils and particles of at least 5 different types: 1. Banded collagen fibrils, present throughout the matrix, but not seen in the lacunae. 2. Non-periodic fine fibrils <100Å in diameter (Fig. 1), which are most notable in the lacunae, and may represent immature collagen. 3. Electron dense matrix granules (Fig. 1) which are often attached to fine fibrils and collagen fibrils, and probably contain protein-polysaccharide although the possibility of a mineral content has not been excluded. 4. Matrix vesicles (Fig. 2) which show a selective distribution throughout the epiphysis, and may play a role in calcification. 5. Needle-like apatite crystals (Fig. 2).Blocks of formalin-fixed epiphysis from weanling mice were digested with the following agents in 0.1M phosphate buffer: a) 5% ethylenediaminetetraacetate (EDTA) at pH 8.3, b) 0.015% bovine testicular hyaluronidase (Sigma, type IV, 750 units/mg) at pH 5.5, and c) 0.1% collagenase (Worthington, chromatograhically pure, 200 units/mg) at pH 7.4. All digestions were carried out at 37°C overnight. Following digestion tissues were examined by light and electron microscopy to determine changes in the various fibrils and particles of the matrix.

Fine Structure of Type IV Glycogenosis

1981 ◽  
Vol 39 ◽  
pp. 464-465
Author(s):  
P.K. Simons
Keyword(s):  
Needle Biopsy ◽  
Tissue Concentration ◽  
Inborn Errors ◽  
Rare Type ◽  
Type Iv ◽  
Iron Copper ◽  
Proper Diagnosis ◽  
Type Iv Glycogenosis ◽  
Loss Of Appetite ◽  
Enzymatic Defect

Glycogenosis is defined as any condition in which the tissue concentration of glycogen is increased. There are currently ten recognized variants of glycogenosis that are heritable inborn errors of metabolism. The specific enzymatic defect in each of the variants is known or at least suspected. In all cases, the enzymatic defect prevents the proper metabolism or formation of the glycogen molecule. The clinical and histologic differences between the types of glycogenosis is important to a proper diagnosis after the presence of such a condition is realized. This study was initiated to examine the ultrastructure of the rare Type IV Glycogenosis (Amylopectinosis) of which there is very little morphologic characterization in the literature.Liver tissue was obtained by needle biopsy from a 12-month-old Oriental female who was originally admitted to the hospital after observation of poor development, loss of appetite, and hepatomegaly. The majority of the tissue was fixed for light microscopy in neutral buffered formalin and processed using routine and special staining procedures (reticulin, trichrome, iron, copper, PAS, PAS-diastase and PAS-pectinase.

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