Effects of Zibu Piyin Recipe on Endoplasmic Reticulum Stress in the Brain Tissue of Spleen-Yin Deficiency Alzheimer's Disease Rats

2011 ◽  
Vol 13 (6) ◽  
pp. 993-998
Author(s):  
Zhan Libin ◽  
Lin Haiyan ◽  
Gong Xiaoyang ◽  
Liu Li ◽  
Liang Lina
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Brain Tissue ◽  
Yin Deficiency ◽  
The Brain

scholarly journals Role of Endoplasmic Reticulum Stress in Learning and Memory Impairment and Alzheimer's Disease-Like Neuropathology in the PS19 and APPSwe Mouse Models of Tauopathy and Amyloidosis

eNeuro ◽  
2017 ◽  
Vol 4 (4) ◽  
pp. ENEURO.0025-17.2017 ◽  
Author(s):  
Denise Isabelle Briggs ◽  
Erwin Defensor ◽  
Pooneh Memar Ardestani ◽  
Bitna Yi ◽  
Michelle Halpain ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Learning And Memory ◽  
Mouse Models ◽  
Memory Impairment

scholarly journals Endoplasmic reticulum stress responses in mouse models of Alzheimer's disease: Overexpression paradigmversusknockin paradigm

2018 ◽  
Vol 293 (9) ◽  
pp. 3118-3125 ◽  
Author(s):  
Shoko Hashimoto ◽  
Ayano Ishii ◽  
Naoko Kamano ◽  
Naoto Watamura ◽  
Takashi Saito ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Mouse Models ◽  
Stress Responses

scholarly journals Endoplasmic Reticulum Stress Induces Tau Pathology and Forms a Vicious Cycle: Implication in Alzheimer's Disease Pathogenesis

2012 ◽  
Vol 28 (4) ◽  
pp. 839-854 ◽  
Author(s):  
Yuen-Shan Ho ◽  
Xifei Yang ◽  
Jeffery Chi-Fai Lau ◽  
Clara Hui-Ling Hung ◽  
Suthicha Wuwongse ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Vicious Cycle ◽  
Tau Pathology ◽  
Disease Pathogenesis

Exploring the brain tissue proteome of TgCRND8 Alzheimer's Disease model mice under B vitamin deficient diet induced hyperhomocysteinemia by LC-MS top-down platform

2019 ◽  
Vol 1124 ◽  
pp. 165-172
Author(s):  
Daniela Delfino ◽  
Diana Valeria Rossetti ◽  
Claudia Martelli ◽  
Ilaria Inserra ◽  
Federica Vincenzoni ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Brain Tissue ◽  
Disease Model ◽  
Top Down ◽  
Deficient Diet ◽  
B Vitamin ◽  
The Brain ◽  
Tissue Proteome

scholarly journals Ca2+-dependent endoplasmic reticulum stress correlates with astrogliosis in oligomeric amyloid β-treated astrocytes and in a model of Alzheimer's disease

Aging Cell ◽  
2013 ◽  
Vol 12 (2) ◽  
pp. 292-302 ◽  
Author(s):  
Elena Alberdi ◽  
Ane Wyssenbach ◽  
María Alberdi ◽  
Mª V. Sánchez-Gómez ◽  
Fabio Cavaliere ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Amyloid Β ◽  
Model Of Alzheimer’S Disease

scholarly journals Intracellular Accumulation of Toxic Turn Amyloid-β is Associated with Endoplasmic Reticulum Stress in Alzheimer's Disease

2013 ◽  
Vol 10 (1) ◽  
pp. 11-20 ◽  
Author(s):  
Naoko Soejima ◽  
Yasumasa Ohyagi ◽  
Norimichi Nakamura ◽  
Eri Himeno ◽  
Kyoko M. Iinuma ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Endoplasmic Reticulum ◽  
Endoplasmic Reticulum Stress ◽  
Amyloid Β ◽  
Intracellular Accumulation

Cerebrospinal Fluid pH and Monoamine and Glucolytic Metabolites in Alzheimer's Disease

1974 ◽  
Vol 124 (580) ◽  
pp. 280-287 ◽  
Author(s):  
C. G. Gottfries ◽  
Åke Kjällquist ◽  
Urban Pontén ◽  
B. E. Roos ◽  
G. Sundbärg
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Cerebrospinal Fluid ◽  
Brain Tissue ◽  
Homovanillic Acid ◽  
Monoamine Metabolites ◽  
Hydroxyindoleacetic Acid ◽  
The Brain ◽  
Valid Information

Determinations of acid monoamine metabolites, such as homovanillic acid (HVA) and 5-hydroxyindoleacetic acid (5-HIAA), in cerebrospinal fluid (CSF) give valid information on the metabolism of the corresponding amines in the brain tissue (Moir et al., 1970; Roos, 1970). The monoamine metabolites in the CSF are related to age. The concentrations of HVA and 5-HIAA increase with age (Gottfries et al., 1971). Probenecid blocks the elimination of HVA and 5-HIAA from brain tissue to blood (Neff et al., 1964, 1967; Werdinius, 1966) and from CSF to blood (Guldberg et al., 1966; Olsson and Roos, 1968). Probenecid thus normally induces an increase in the concentrations of the acid monoamine metabolites in the CSF, which is related to the turnover of monoamines in the brain tissue.

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