scholarly journals Methods for personal cooling in hot environment used in clothing and wearables

2021 ◽  
Vol 327 ◽  
pp. 03003
Author(s):  
Atanas Vasilev ◽  
Radostina A. Angelova ◽  
Rositsa Velichkova

The use of an efficient personal cooling system in hot environments is becoming increasingly popular, as the increased air temperature provokes thermophysiological discomfort, heat stress, reduced productivity and could lead to several health issues. Different methods and devices for personal and local cooling have been developed over the years. The paper summarises the cooling methods applied in clothing and wearable items: phase-change materials, Peltier elements, evaporative cooling, water cooling and hybrid cooling. The local vs total (of the whole body) cooling is examined. The passive and active colling are analysed in terms of advantages, disadvantages and application.

2020 ◽  
Vol 129 (4) ◽  
pp. 947-956
Author(s):  
Josh T. Arnold ◽  
Alex B. Lloyd ◽  
Stephen J. Bailey ◽  
Tomomi Fujimoto ◽  
Ryoko Matsutake ◽  
...  

When separated from local cooling, whole body cooling elicited cutaneous reflex vasoconstriction via mechanisms independent of nitric oxide removal. Hypoxia elicited cutaneous vasodilatation via mechanisms mediated primarily by nitric oxide synthase, rather than xanthine oxidase-mediated nitrite reduction. Cold-induced vasoconstriction was blunted by the opposing effect of hypoxic vasodilatation, whereas the underpinning mechanisms did not interrelate in the absence of local cooling. Full vasoconstriction was restored with nitric oxide synthase inhibition.


2007 ◽  
Vol 293 (5) ◽  
pp. H3187-H3192 ◽  
Author(s):  
Gary J. Hodges ◽  
Wojciech A. Kosiba ◽  
Kun Zhao ◽  
Guy E. Alvarez ◽  
John M. Johnson

Previous work showed that local cooling (LC) attenuates the vasoconstrictor response to whole body cooling (WBC). We tested the extent to which this attenuation was due to the decreased baseline skin blood flow following LC. In eight subjects, skin blood flow was assessed using laser-Doppler flowmetry (LDF). Cutaneous vascular conductance (CVC) was expressed as LDF divided by blood pressure. Subjects were dressed in water-perfused suits to control WBC. Four forearm sites were prepared with microdialysis fibers, local heating/cooling probe holders, and laser-Doppler probes. Three sites were locally cooled from 34 to 28°C, reducing CVC to 45.9 ± 3.9, 42 ± 3.9, and 44.5 ± 4.8% of baseline ( P < 0.05 vs. baseline; P > 0.05 among sites). At two sites, CVC was restored to precooling baseline levels with sodium nitroprusside (SNP) or isoproterenol (Iso), increasing CVC to 106.4 ± 12.4 and 98.9 ± 10.1% of baseline, respectively ( P > 0.05 vs. precooling). Whole body skin temperature, apart from the area of blood flow measurement, was reduced from 34 to 31°C. Relative to the original baseline, CVC decreased ( P < 0.05) by 44.9 ± 2.8 (control), 11.3 ± 2.4 (LC only), 29 ± 3.7 (SNP), and 45.8 ± 8.7% (Iso). The reductions at LC only and SNP sites were less than at control or Iso sites ( P < 0.05); the responses at those latter sites were not different ( P > 0.05), suggesting that the baseline change in CVC with LC is important in the attenuation of reflex vasoconstrictor responses to WBC.


2010 ◽  
Vol 108 (2) ◽  
pp. 328-333 ◽  
Author(s):  
Fumio Yamazaki

Local cooling (LC) of nonglabrous skin causes vasoconstriction via the adrenergic and removal of nitric oxide (NO) systems. Since cooling increases reactive oxygen species in smooth muscle cells and induces increased sensitivity of α-adrenergic receptors, antioxidant supplementation may attenuate the vasoconstrictor response to skin LC via adrenergic and/or NO systems. To test this hypothesis, we examined the effects of acute l-ascorbate (Asc, 10 mM) supplementation in human skin on the vasoconstrictor responses to LC in skin with and without NO synthase (NOS) inhibition or adrenergic receptor blockade. In a three-part study, forearm sites were instrumented with microdialysis fibers, local coolers, and laser-Doppler flow (LDF) probes in healthy volunteers. Sites were cooled from 34 to 24°C at −1°C/min and maintained at 24°C for 20 min ( parts 1 and 2) or 30 min ( part 3). During the last 10 min of LC in parts 1 and 2, whole body cooling was performed to increase sympathetic vasoconstrictor activity. Cutaneous vascular conductance (CVC) was calculated as the ratio of LDF to blood pressure and expressed relative to the baseline value before cooling. Treatments in each part were as follows: part 1) untreated, Asc; part 2) NG-nitro-l-arginine methyl ester (l-NAME) to inhibit NOS, combined l-NAME + Asc; part 3) yohimbine (YOH) + propranolol (PRO) to antagonize α- and β-adrenergic receptors and combined YOH + PRO + Asc. CVC reduction during LC was smaller ( P < 0.001) at Asc sites (−31 ± 4%) than at untreated sites (−56 ± 5%). LC-induced reduction in CVC was smaller ( P < 0.05) at l-NAME + Asc sites (−23 ± 8%) than at l-NAME sites (−43 ± 7%). LC-induced reduction in CVC did not differ between at PRO + YOH sites (−56 ± 3%) and at PRO + YOH + Asc sites (−50 ± 3%). These findings suggest that antioxidant supplementation inhibits the vasoconstrictor response to direct cooling through an adrenoceptor-dependent mechanism in human skin.


2006 ◽  
Vol 100 (6) ◽  
pp. 2083-2088 ◽  
Author(s):  
Guy E. Alvarez ◽  
Kun Zhao ◽  
Wojciech A. Kosiba ◽  
John M. Johnson

The reduction in skin blood flow (SkBF) with cold exposure is partly due to the reflex vasoconstrictor response from whole body cooling (WBC) and partly to the direct effects of local cooling (LC). Although these have been examined independently, little is known regarding their roles when acting together, as occurs in environmental cooling. We tested the hypothesis that the vasoconstrictor response to combined LC and WBC would be additive, i.e., would equal the sum of their independent effects. We further hypothesized that LC would attenuate the reflex vasoconstrictor response to WBC. We studied 16 (7 women, 9 men) young (30.5 ± 2 yr) healthy volunteers. LC and WBC were accomplished with metal Peltier cooler-heater probe holders and water-perfused suits, respectively. Forearm SkBF was monitored by laser-Doppler flowmetry (LDF). Cutaneous vascular conductance (CVC) was calculated as LDF/blood pressure. Subjects underwent 15 min of LC alone or 15 min of WBC with and without simultaneous LC, either at equal levels (34–31°C) or as equipotent stimuli (34–28°C LC; 34–31°C WBC). The fall in CVC with combined WBC and LC was greater ( P < 0.05) than for either alone (57.0 ± 5% combined vs. 39.2 ± 6% WBC; 34.4 ± 4% LC) with equipotent cooling, but it was only significantly greater than for LC alone with equal levels of cooling (51.3 ± 8% combined vs. 29.5 ± 4% LC). The sum of the independent effects of WBC and LC was greater than their combined effects (74.9 ± 4 vs. 51.3 ± 8% equal and 73.6 ± 7 vs. 57.0 ± 5% equipotent; P < 0.05). The fall in CVC with WBC at LC sites was reduced compared with control sites (17.6 ± 2 vs. 42.4 ± 8%; P < 0.05). Hence, LC contributes importantly to the reduction in SkBF with body cooling, but also suppresses the reflex response, resulting in a nonadditive effect of these two components.


2011 ◽  
Vol 70 ◽  
pp. 722-722
Author(s):  
S Sarkar ◽  
J R Bapuraj ◽  
S M Donn ◽  
I Bhagat ◽  
J D Barks

1991 ◽  
Vol 155 (1) ◽  
pp. 193-202 ◽  
Author(s):  
ALBERT CRAIG ◽  
JACQUES LAROCHELLE

The rate of heat loss through the stretched wings (Hwings) was studied in resting pigeons preheated to a body temperature (43.7°C) within the range of those recorded during flight. The experimental system was designed to allow the calculation of Hwings from the increase in whole-body cooling rates resulting from exposure of the wings to various wind speeds (0–50 km h−1) at 23°C. The maximum value of HWings was 3.8 W, less than twice the heat production of a resting pigeon. This indicates that the contribution of the wings to heat dissipation during flight may not be nearly as important as has been supposed. At low windspeeds (0–12.5 km h−1), HWings corresponded to about 40% of the resting rate of heat production, and this value is discussed in connection with the various wing postures observed in hyperthermic birds.


2018 ◽  
Vol 7 (1) ◽  
pp. 7
Author(s):  
Rahul Sinha ◽  
K Venkatnarayan ◽  
Vandana Negi ◽  
Kirandeep Sodhi ◽  
BM John

2020 ◽  
Vol 220 ◽  
pp. 73-79.e3
Author(s):  
Tai-Wei Wu ◽  
Jessica L. Wisnowski ◽  
Robert F. Geisler ◽  
Aaron Reitman ◽  
Eugenia Ho ◽  
...  

2019 ◽  
Vol 47 (9) ◽  
pp. 986-990 ◽  
Author(s):  
Mahdi Alsaleem ◽  
Lina Saadeh ◽  
Valerie Elberson ◽  
Vasantha H.S. Kumar

Abstract Objective To describe the clinical characteristics and risk factors in infants with subcutaneous fat necrosis (SFN) following therapeutic hypothermia for hypoxic-ischemic encephalopathy (HIE). Methods A case-control study was performed by a retrospective chart review of infants with moderate or severe HIE admitted to a level IV regional perinatal center and who underwent whole-body cooling. Results A total of 14 (8.1%) of 171 infants with moderate or severe HIE who underwent whole-body cooling developed SFN during hospitalization. There were more females [71% (10/14)] and large-for-gestational age (LGA) infants [28% (4/14)] in the SFN group vs. 36% females (57/157) and 8% LGA infants (13/157) in the group without SFN (P-values of 0.009 and 0.015, respectively). The mean lowest platelet count was lower 108 ± 55 109/L vs. 146 ± 62 109/L and the mean highest calcium level was higher 11.3 ± 2.5 vs. 10.6 ± 0.8 mg/dL in infants with SFN vs. infants without SFN, respectively (P-values of 0.0078 and 0.006, respectively). Distribution of skin lesions followed distinctive patterns representing the areas with direct contact with the cooling blanket. One infant developed severe, life-threatening hypercalcemia that required aggressive management, including diuretics, corticosteroids and bisphosphonates. Conclusion Although SFN is a rare complication of therapeutic hypothermia, it can be a life-threatening condition if complicated by severe hypercalcemia. Infants who undergo therapeutic hypothermia for HIE need regular skin examinations to evaluate for SFN. If SFN is identified, monitoring of serum calcium levels to prevent life-threatening hypercalcemia is recommended.


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