Ramsay Hunt's Syndrome in Pediatric Age

2021 ◽  
Author(s):  
Marta Ribeiro Silva ◽  
Lídia Leite ◽  
Filipa Peixoto Sousa ◽  
Ricardo Maré ◽  
Teresa Pontes
Keyword(s):  
Facial Palsy ◽  
Antiviral Treatment ◽  
Gadolinium Contrast ◽  
Varicella Zoster ◽  
Peripheral Facial Palsy ◽  
Csf Analysis ◽  
Seventh Cranial Nerve ◽  
Blurry Vision ◽  
The Right ◽  
The Brain

AbstractThe Ramsay Hunt's syndrome (RHS), caused by the reactivation of the varicella-zoster virus (VZV), is an infrequent cause of peripheral facial palsy in the pediatric patients. A 16-year-old adolescent boy presented with headache, right earache, and signs of lower motor neuron type facial palsy. He was medicated with deflazacort. Four days later, he developed blurry vision in the right eye, gait imbalance, and vesicular lesions in the right ear with decreased ipsilateral hearing acuity. A diagnosis of RHS was considered. Cerebrospinal fluid (CSF) analysis showed pleocytosis, as well as VZV DNA. Magnetic resonance imaging of the brain identified abnormal uptake of gadolinium contrast in the right seventh cranial nerve, reinforcing the diagnosis. The patient was treated with acyclovir (14 days) and prednisone, with progressive clinical improvement. We emphasize the importance of recognizing this syndrome in patients with facial palsy, since the early antiviral treatment significantly improves the prognosis.

scholarly journals Neurological complications after H1N1 influenza vaccination: magnetic resonance imaging findings

2014 ◽  
Vol 72 (7) ◽  
pp. 496-499 ◽  
Author(s):  
Ronaldo Lessa ◽  
Maurício Castillo ◽  
Renata Azevedo ◽  
Fernanda Azevedo ◽  
Hildo Azevedo
Keyword(s):  
Facial Palsy ◽  
H1n1 Virus ◽  
Altered Mental Status ◽  
H1n1 Influenza ◽  
Neurological Complications ◽  
Abrupt Onset ◽  
Resonance Imaging ◽  
Post Vaccination ◽  
Peripheral Facial Palsy ◽  
The Right

Objective: To report 4 different neurological complications of H1N1 virus vaccination. Method: Four patients (9, 16, 37 and 69 years of age) had neurological symptoms (intracranial hypertension, ataxia, left peripheral facial palsy of abrupt onset, altered mental status, myelitis) starting 4-15 days after H1N1 vaccination. MRI was obtained during the acute period. Results: One patient with high T2 signal in the cerebellum interpreted as acute cerebellitis; another, with left facial palsy, showed contrast enhancement within both internal auditory canals was present, however it was more important in the right side; one patient showed gyriform hyperintensities on FLAIR with sulcal effacement in the right fronto-parietal region; and the last one showed findings compatible with thoracic myelitis. Conclusion: H1N1 vaccination can result in important neurological complications probably secondary to post-vaccination inflammation. MRI detected abnormalities in all patients.

Rapidly progressive vision loss due to fulminant idiopathic intracranial hypertension: a diagnostic and management dilemma

2020 ◽  
Vol 13 (11) ◽  
pp. e236188
Author(s):  
Jagadeesh Sutraye ◽  
Mohan Kannam ◽  
Rajat Kapoor ◽  
Virender Sachdeva
Keyword(s):  
Intracranial Hypertension ◽  
Idiopathic Intracranial Hypertension ◽  
Vision Loss ◽  
Obese Woman ◽  
Visual Fields ◽  
Csf Analysis ◽  
Contrast Enhanced ◽  
Grade Ii ◽  
The Right ◽  
The Brain

A 44-year-old obese woman presented with decrease in vision in the right eye (RE) for 3 days. She reported a simultaneous onset of holocranial headache that worsened on bending forward. She denied eye pain, pain on eye movements, and other ocular or neurological complaints. On examination, her distance best-corrected visual acuity was counting fingers at 1 m in the RE and 20/20 in the left eye (LE). Colour vision was subnormal in both eyes (BE). There was grade II relative afferent pupillary defect in the RE. Fundus examination showed disc oedema in BE . Visual fields in the LE showed central scotoma extending nasally. A provisional diagnosis of papillitis was considered. However, contrast-enhanced MRI of the brain and orbits showed evidence of elevated intracranial pressure. Cerebrospinal fluid (CSF) opening pressure was 42 cm H2O while rest of the CSF analysis was normal. Diagnosis was revised to fulminant idiopathic intracranial hypertension. Management with medical therapy and urgent thecoperiteoneal shunt improved visual function in BE.

Two cases of peripheral facial palsy with negative CSF analysis despite an infectious etiology

2019 ◽  
pp. 10.1212/CPJ.0000000000000791
Author(s):  
Johann Lambeck ◽  
Maren Hieber ◽  
Wolf-Dirk Niesen ◽  
Jürgen Bardutzky
Keyword(s):  
Facial Palsy ◽  
Peripheral Facial Palsy ◽  
Csf Analysis ◽  
Infectious Etiology

scholarly journals Presentation of Bilateral Peripheral Seventh Cranial Nerve Palsy in an HIV Patient

2012 ◽  
Vol 2012 ◽  
pp. 1-3 ◽  
Author(s):  
Lisa M. Ruiz ◽  
Batool Kirmani
Keyword(s):  
Human Immunodeficiency Virus ◽  
Facial Palsy ◽  
Nerve Palsy ◽  
Neurological Manifestations ◽  
Newly Diagnosed ◽  
Specific Location ◽  
Peripheral Facial Palsy ◽  
Seventh Cranial Nerve ◽  
Time Period ◽  
Immunodeficiency Virus

Neurological manifestations in patients infected with human immunodeficiency virus can significantly increase overall morbidity and mortality. These complications are neither limited to a specific location in the nervous system nor a focal time period in the disease’s progression. A literature review yielded several cases of peripheral facial palsy associated with HIV seropositivity, but few cases have been reported where the patient had bilateral peripheral facial palsy. In this paper, we present a patient with bilateral peripheral facial palsy and aseptic meningitis in the context of newly diagnosed HIV.

Serial MRI and CSF Analysis in a Dog Treated with Intrathecal Amphotericin B for Protothecosis

2012 ◽  
Vol 48 (2) ◽  
pp. 125-131 ◽  
Author(s):  
Martin Young ◽  
William Bush ◽  
Melissa Sanchez ◽  
Pat Gavin ◽  
Mathew Williams
Keyword(s):  
Cerebrospinal Fluid ◽  
Amphotericin B ◽  
Head Tilt ◽  
Mri Findings ◽  
Lipid Complex ◽  
Fluid Analysis ◽  
Csf Analysis ◽  
Transient Improvement ◽  
The Right ◽  
The Brain

A 3 yr old female spayed English setter mixed-breed dog presented with diarrhea, weight loss, side stepping to the right, and a right head tilt. Rectal and cerebrospinal fluid cytology and culture confirmed a diagnosis of Prototheca zopfii. MRI of the brain showed inflammation of the brain and meninges, ventriculomegaly, and syringomyelia. Treatment with prednisone, itraconazole, and amphotericin B lipid complex administered intrathecally yielded transient improvement. Progressive brainstem signs were noted, and a repeat MRI and cerebrospinal fluid analysis documented persistent disease. This is the first description of the MRI findings and treatment with intrathecal amphoteracin B lipid complex for protothecosis of the central nervous system. Protothecosis should be considered in dogs with chronic diarrhea and compatible MRI findings.

An ELISA Study on Varicella-Zoster Virus Infection in Acute Peripheral Facial Palsy

1987 ◽  
Vol 104 (sup446) ◽  
pp. 10-16
Author(s):  
Hiroshi Tomita ◽  
Masami Tanaka ◽  
Nobuo Kukimoto ◽  
Minoru Ikeda
Keyword(s):  
Virus Infection ◽  
Varicella Zoster Virus ◽  
Facial Palsy ◽  
Varicella Zoster Virus Infection ◽  
Varicella Zoster ◽  
Peripheral Facial Palsy

scholarly journals Protracted COVID-19 during Treatment of Facial Palsy

2021 ◽  
Vol 2021 ◽  
pp. 1-3
Author(s):  
Mehrdad Hasibi ◽  
Maral Seyed Ahadi ◽  
Hamed Abdollahi ◽  
Mehrdad Jafari
Keyword(s):  
Facial Palsy ◽  
Antiviral Treatment ◽  
Complete Recovery ◽  
Bell’S Palsy ◽  
Concomitant Treatment ◽  
Neurologic Manifestations ◽  
Bell's Palsy ◽  
Peripheral Facial Palsy ◽  
Case Summary

Neurologic manifestations are increasingly reported as the coronavirus disease 2019 (COVID-19) pandemic continues. This is a report of a COVID-19 patient with Bell's palsy. Case Summary. A 52-year-old man with fever and malaise was tested positive for COVID-19. After a week, he developed right-sided peripheral facial palsy and was treated with corticosteroids in conjunction with antiviral treatment which resulted in complete recovery. Discussion. Concomitant treatment of corticosteroids and antiviral treatment can decrease morbidity in patients with COVID-19-related Bell's palsy.

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