Formation of electrostatic structures by wakefield acceleration in ultrarelativistic plasma flows: Electron acceleration to cosmic ray energies

2006 ◽  
Vol 13 (6) ◽  
pp. 062905 ◽  
Author(s):  
M. E. Dieckmann ◽  
P. K. Shukla ◽  
B. Eliasson
2019 ◽  
Vol 34 (34) ◽  
pp. 1943018
Author(s):  
T. Ebisuzaki ◽  
T. Tajima

We consider that electromagnetic pulses produced in the jets of this innermost part of the accretion disk accelerate charged particles (protons, ions, electrons) to very high energies via wakefield acceleration, including energies above 10[Formula: see text] eV for the case of protons and nucleus and 10[Formula: see text] eV for electrons by electromagnetic wave-particle interaction. Thereby, the wakefield acceleration mechanism supplements the pervasive Fermi’s stochastic acceleration mechanism (and overcomes its difficulties in the highest energy cosmic ray generation). The episodic eruptive accretion in the disk by the magneto-rotational instability gives rise to the strong Alfvenic pulses, which acts as the driver of the collective accelerating pondermotive force. This pondermotive force drives the wakes. The accelerated hadrons (protons and nuclei) are released to the intergalactic space to be ultra-high energy cosmic rays. The high-energy electrons, on the other hand, emit photons to produce various non-thermal emissions (radio, IR, visible, UV, and gamma-rays) of active galactic nuclei in an episodic manner, giving observational telltale signatures.


2020 ◽  
Vol 499 (2) ◽  
pp. 2785-2802
Author(s):  
Georg Winner ◽  
Christoph Pfrommer ◽  
Philipp Girichidis ◽  
Maria Werhahn ◽  
Matteo Pais

ABSTRACT Supernova remnants (SNRs) are believed to be the source of Galactic cosmic rays (CRs). SNR shocks accelerate CR protons and electrons which reveal key insights into the non-thermal physics by means of their synchrotron and γ-ray emission. The remnant SN 1006 is an ideal particle acceleration laboratory because it is observed across all electromagnetic wavelengths from radio to γ-rays. We perform 3D magnetohydrodynamics (MHD) simulations where we include CR protons and follow the CR electron spectrum. By matching the observed morphology and non-thermal spectrum of SN 1006 in radio, X-rays, and γ-rays, we gain new insight into CR electron acceleration and magnetic field amplification. (1) We show that a mixed leptonic–hadronic model is responsible for the γ-ray radiation: while leptonic inverse-Compton emission and hadronic pion-decay emission contribute equally at GeV energies observed by Fermi, TeV energies observed by imaging air Cherenkov telescopes are hadronically dominated. (2) We show that quasi-parallel acceleration (i.e. when the shock propagates at a narrow angle to the upstream magnetic field) is preferred for CR electrons and that the electron acceleration efficiency of radio-emitting GeV electrons at quasi-perpendicular shocks is suppressed at least by a factor ten. This precludes extrapolation of current 1D plasma particle-in-cell simulations of shock acceleration to realistic SNR conditions. (3) To match the radial emission profiles and the γ-ray spectrum, we require a volume-filling, turbulently amplified magnetic field and that the Bell-amplified magnetic field is damped in the immediate post-shock region. Our work connects microscale plasma physics simulations to the scale of SNRs.


1982 ◽  
Vol 43 (C8) ◽  
pp. C8-69-C8-88 ◽  
Author(s):  
B. Rossi
Keyword(s):  

1997 ◽  
Vol 78 (03) ◽  
pp. 1150-1156 ◽  
Author(s):  
Christina Jern ◽  
Heléne Seeman-Lodding ◽  
Bjӧrn Biber ◽  
Ola Winsӧ ◽  
Sverker Jern

SummaryExperimental data indicate large between-organs variations in rates of synthesis of tissue-type plasminogen activator (t-PA), which may reflect important differences in the capacity for constitutive and stimulated t-PA release from the vascular endothelium. In this report we describe a new multiple-organ experimental in vivo model for simultaneous determinations of net release/uptake rates of t-PA across the coronary, splanchnic, pulmonary, and hepatic vascular beds. In eleven intact anesthetized pigs, blood samples were obtained simultaneously from the proximal aorta, coronary sinus, pulmonary artery, and portal and hepatic veins. Plasma flows were monitored separately for each vascular region. Total plasma t-PA was determined by ELISA with a porcine t-PA standard. Regional net release/uptake rates were defined as the product of arteriovenous concentration gradients and local plasma flows. The net release of t-PA across the splanchnic vascular bed was very high, with a mean output of 1,919 ng total t-PA X min-1 (corresponding to 90 ng per min and 100 g tissue). The net coronary t-PA release was 68 ng X min-1 (30 ng X min-1 X 100 g"1)- Pulmonary net fluxes of t-PA were variable without any significant net t-PA release. The net hepatic uptake rate was 4,855 ng X min-1 (436 ng X min-1 X 100 g-1). Net trans-organ changes of active t-PA mirrored those of total t-PA. The results demonstrate marked regional differences in net release rates of t-PA in vivo. The experimental model we present offers new possibilities for evaluation of regional secretion patterns in the intact animal.


2010 ◽  
Vol 180 (5) ◽  
pp. 519 ◽  
Author(s):  
L.I. Dorman
Keyword(s):  

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