scholarly journals Role of the Inositol 1,4,5-Trisphosphate Receptor in Ca2+Feedback Inhibition of Calcium Release-activated Calcium Current (Icrac)

1999 ◽  
Vol 274 (46) ◽  
pp. 32881-32888 ◽  
Author(s):  
Lisa M. Broad ◽  
David L. Armstrong ◽  
James W. Putney
Keyword(s):  
Calcium Current ◽  
Calcium Release ◽  
Feedback Inhibition ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor

scholarly journals Role of the Phospholipase C-Inositol 1,4,5-Trisphosphate Pathway in Calcium Release-activated Calcium Current and Capacitative Calcium Entry

2001 ◽  
Vol 276 (19) ◽  
pp. 15945-15952 ◽  
Author(s):  
Lisa M. Broad ◽  
Franz-Josef Braun ◽  
Jean-Philippe Lievremont ◽  
Gary St. J. Bird ◽  
Tomohiro Kurosaki ◽  
...  
Keyword(s):  
Phospholipase C ◽  
Calcium Current ◽  
Calcium Release ◽  
Calcium Entry ◽  
Capacitative Calcium Entry ◽  
Inositol 1,4,5 Trisphosphate

scholarly journals Unraveling the role of polycystin-2/inositol 1,4,5-trisphosphate receptor interaction in Ca2+signaling

2010 ◽  
Vol 3 (6) ◽  
pp. 530-532 ◽  
Author(s):  
Eva Sammels ◽  
Benoit Devogelaere ◽  
Djalila Mekahli ◽  
Geert Bultyncki ◽  
Ludwig Missiaen ◽  
...  
Keyword(s):  
Receptor Interaction ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor

scholarly journals Inositol 1,4,5-Trisphosphate Receptors in Human Disease: A Comprehensive Update

2020 ◽  
Vol 9 (4) ◽  
pp. 1096
Author(s):  
Jessica Gambardella ◽  
Angela Lombardi ◽  
Marco Bruno Morelli ◽  
John Ferrara ◽  
Gaetano Santulli
Keyword(s):  
Human Disease ◽  
Calcium Release ◽  
Current Knowledge ◽  
Association Studies ◽  
Genome Wide Association Studies ◽  
Loss Of Function ◽  
Genome Wide ◽  
Inositol 1,4,5 Trisphosphate ◽  
Human Disorders

Inositol 1,4,5-trisphosphate receptors (ITPRs) are intracellular calcium release channels located on the endoplasmic reticulum of virtually every cell. Herein, we are reporting an updated systematic summary of the current knowledge on the functional role of ITPRs in human disorders. Specifically, we are describing the involvement of its loss-of-function and gain-of-function mutations in the pathogenesis of neurological, immunological, cardiovascular, and neoplastic human disease. Recent results from genome-wide association studies are also discussed.

Partial calcium release in response to submaximal inositol 1,4,5-trisphosphate receptor activation

1994 ◽  
Vol 98 (2) ◽  
pp. 147-156 ◽  
Author(s):  
Ludwig Missiaen ◽  
Jan B. Parys ◽  
Humbert De Smedt ◽  
Masahiro Oike ◽  
Rik Casteels
Keyword(s):  
Calcium Release ◽  
Receptor Activation ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor

122 Role of calmodulin on the calcium-dependent regulation of the inositol 1,4,5-trisphosphate receptor

1996 ◽  
Vol 25 ◽  
pp. S24
Author(s):  
Takayuki Michikawa ◽  
Junji Hirota ◽  
Seiko Kawano ◽  
Masayasu Hiraoka ◽  
Teiichi Furuichi ◽  
...  
Keyword(s):  
Calcium Dependent ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor
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