scholarly journals Evidence for magnetic clusters in Ni1-xVxclose to the quantum critical concentration

2015 ◽  
Vol 592 ◽  
pp. 012089 ◽  
Author(s):  
R Wang ◽  
S Ubaid-Kassis ◽  
A Schroeder ◽  
P J Baker ◽  
F L Pratt ◽  
...  
Keyword(s):  
Critical Concentration ◽  
Magnetic Clusters ◽  
Quantum Critical

Fermi liquid instabilities and superconductivity near quantum critical points in f-electron materials

2003 ◽  
Vol 802 ◽  
Author(s):  
M. B. Maple ◽  
N. A. Frederick ◽  
J. R. Jeffries ◽  
P.-C. Ho ◽  
V. S. Zapf
Keyword(s):  
Heavy Fermion ◽  
Critical Concentration ◽  
Quantum Critical Point ◽  
Electric Quadrupole ◽  
Residual Resistivity ◽  
Quadrupole Moments ◽  
Quantum Critical Points ◽  
Break Time ◽  
Quantum Critical ◽  
Skutterudite Compound

ABSTRACTRecent experiments on single crystals of the compounds CeRh1−xCoxIn5 and PrOs4Sb12 are briefly reviewed. The temperature-composition (T-x) phase diagram of the heavy fermion pseudoternary system CeRh1−xCoxIn5, delineating the regions in which superconductivity, antiferromagnetism, and the coexistence of these two phenomena occur, has been established. Entropy vs x isotherms and residual resistivity vs x plots exhibit peaks near the critical concentration xcr ≈ 0.8 at which the Néel temperature appears to vanish (quantum critical point). The filled skutterudite compound PrOs4Sb12 exhibits unconventional superconductivity below Tc = 1.85 K that involves heavy fermion quasiparticles with an effective mass m* ≈ 50 me, where me is the mass of the free electron. The unconventional superconducting state appears to consist of several distinct superconducting phases and to break time reversal symmetry. A high field ordered phase occurs below ∼ 1 K and between ∼ 4.5 T and ∼ 15 T that appears to be associated with quadrupolar order. The heavy fermion state and superconductivity in PrOs4Sb12 may originate from the interaction between Pr3+ electric quadrupole moments and the charges of the conduction electrons. When Ru is substituted for Os in PrOs4Sb12, a minimum in Tc occurs at Pr(Os0.4Ru0.6)4Sb12, suggesting a competition between two types of superconductivity.

MAGNETIC CLUSTERS IN Y-Ni AMORPHOUS ALLOYS NEAR THE CRITICAL CONCENTRATION

1980 ◽  
Vol 41 (C8) ◽  
pp. C8-658-C8-661 ◽  
Author(s):  
A. Liénard ◽  
J. P. Rebouillat ◽  
P. Garoche ◽  
J. J. Veyssié
Keyword(s):  
Amorphous Alloys ◽  
Critical Concentration ◽  
Magnetic Clusters

scholarly journals Evidence for magnetic clusters in stoichiometric quantum critical CeRu2Si2

2021 ◽  
Vol 104 (8) ◽  
Author(s):  
Alex Bretaña ◽  
Sean Fayfar ◽  
Wouter Montfrooij
Keyword(s):  
Magnetic Clusters ◽  
Quantum Critical

EXPERIMENTAL EVIDENCES OF MAGNETIC CLUSTERS IN AN AMORPHOUS Fe0.34Sn0.66 ALLOYS

1980 ◽  
Vol 41 (C8) ◽  
pp. C8-666-C8-669
Author(s):  
S. Nikolov ◽  
M. Piecuch ◽  
G. Marchal ◽  
Chr. Janot
Keyword(s):  
Magnetic Clusters

An equation for the empirical design of anoxic zones used to eliminate rising sludges at nitrifying activated sludge plants

1996 ◽  
Vol 33 (3) ◽  
pp. 185-194 ◽  
Author(s):  
M. Sarioglu ◽  
N. Horan
Keyword(s):  
Activated Sludge ◽  
Empirical Equation ◽  
Nitrate Nitrogen ◽  
Critical Concentration ◽  
Settling Tank ◽  
Limited Information ◽  
Batch Experiments ◽  
Anoxic Zone ◽  
Secondary Settling ◽  
Anoxic Zones

Anoxic zones are designed for the removal of nitrogen in nitrifying activated sludge plants. This can be carried out either to achieve a nitrogen discharge consent or to eliminate the problem of rising sludges. The rising sludge problem is mostly encountered in medium and small size plants in warm conditions and there is limited information as to the appropriate design of anoxic zones to protect against rising sludges in the secondary sedimentation tanks. Therefore a series of batch experiments were undertaken in order to establish the critical concentration of nitrate-nitrogen which causes rising sludge in the secondary settling tank and the effect of environmental factors such as temperature (15°C to 30°C) and residual carbon source (100 to 600 mg/1 COD) were examined. Based on the results of these experiments an empirical equation was presented which can be used to size an anoxic zone to eliminate rising sludges. The application of this equation at full-scale plants is discussed.

Serum Carboxypeptidase N1 Serves as a Potential Biomarker Complementing CA15-3 for Breast Cancer

2020 ◽  
Vol 20 (17) ◽  
pp. 2053-2065
Author(s):  
Ranliang Cui ◽  
Chaomin Wang ◽  
Qi Zhao ◽  
Yichao Wang ◽  
Yueguo Li
Keyword(s):  
Breast Cancer ◽  
Metastatic Breast Cancer ◽  
Critical Concentration ◽  
Tumour Size ◽  
Clinical Stage ◽  
Metastatic Breast ◽  
Breast Cancer Patients ◽  
Potential Biomarker ◽  
Incidence And Mortality ◽  
Combined Detection

Background: The incidence and mortality of breast cancer are increasing annually. Breast cancer seriously threatens women's health and quality of life. We aimed to measure the clinical value of CPN1, a new serum marker of breast cancer and to evaluate the efficacy of CPN1 in combination with CA15-3. Methods: Seventy samples of breast cancer with lymph node metastasis, seventy-three samples of nonmetastatic breast cancer and twenty-five samples of healthy human serum were collected. Serum CA15-3 concentration was determined by Roche Elecsys, and serum CPN1 concentration was determined by ELISA. Results: In breast cancer patients, serum CPN1 concentration was positively correlated with tumour size, clinical stage and CA15-3 concentration (r = 0.376, P<0.0001). ROC curve analysis showed that the optimal critical concentration of CPN1 for breast cancer diagnosis was 32.8pg/ml. The optimal critical concentration of CPN1 in the diagnosis of metastatic breast cancer was 66.121pg/ml. CPN1 has a greater diagnostic ability for breast cancer (AUCCA15-3=0.702 vs. AUCCPN1=0.886, P<0.0001) and metastatic breast cancer (AUCCA15-3=0.629 vs. AUCCPN1=0.887, P<0.0001) than CA15-3, and the combined detection of CA15-3 and CPN1 can improve the diagnostic efficiency for breast cancer (AUCCA15-3+CPN1=0.916) and for distinguishing between metastatic and non-metastatic breast cancer (AUCCA15-3+CPN1=0.895). Conclusion: CPN1 can be used as a new tumour marker to diagnose and evaluate the invasion and metastasis of breast cancer. The combined detection of CPN1 and CA15-3 is more accurate and has a certain value in clinical application.

scholarly journals Importance of beta-lactamase inhibitor pharmacokinetics in the pharmacodynamics of inhibitor-drug combinations: studies with piperacillin-tazobactam and piperacillin-sulbactam.

1997 ◽  
Vol 41 (4) ◽  
pp. 721-727 ◽  
Author(s):  
P D Lister ◽  
A M Prevan ◽  
C C Sanders
Keyword(s):  
Antibacterial Activity ◽  
Critical Concentration ◽  
Specific Inhibitor ◽  
Logarithmic Phase ◽  
Critical Ratio ◽  
Beta Lactamase ◽  
Beta Lactam ◽  
Dosing Regimens ◽  
Lactamase Inhibitor

An in vitro pharmacokinetic model was used to study the pharmacodynamics of piperacillin-tazobactam and piperacillin-sulbactam against gram-negative bacilli producing plasmid-encoded beta-lactamases. Logarithmic-phase cultures were exposed to peak antibiotic concentrations observed in human serum after the administration of intravenous doses of 3 g of piperacillin and 0.375 g of tazobactam or 0.5 g of sulbactam. Piperacillin and inhibitor were either dosed simultaneously or piperacillin was dosed sequentially 0.5 h after dosing with the inhibitor. In studies with all four test strains, the pharmacodynamics observed after simultaneous dosing were similar to those observed with the sequential regimen. Since the ratio between piperacillin and tazobactam was in constant fluctuation after sequential dosing, these data suggest that the pharmacodynamics of the piperacillin-inhibitor combinations were not dependent upon maintenance of a critical ratio between the components. Furthermore, when regrowth was observed, the time at which bacterial counts began to increase was similar between the simultaneous and sequential dosing regimens. Since the pharmacokinetics of the inhibitors were the same for all regimens, these data suggest that the length of time that the antibacterial activity was maintained over the dosing interval with these combinations was dictated by the pharmacokinetics of the beta-lactamase inhibitor in the combination. The antibacterial activity of the combination appeared to be lost when the amount of inhibitor available fell below some critical concentration. This critical concentration varied depending upon the type and amount of enzyme produced, as well as the specific inhibitor used. These results indicate that the antibacterial activity of drug-inhibitor combinations, when dosed at their currently recommended ratios, is more dependent on the pharmacokinetics of the inhibitor than on those of the beta-lactam drug.

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