Epstein-Barr Virus-Associated Gastric Lymphomas Are Distinct From Mucosa-Associated Lymphoid Tissue-Type Lymphomas: Genetic Abnormalities of p53 Gene

2001 ◽  
Vol 10 (3) ◽  
pp. 153-160 ◽  
Author(s):  
Wing-Yee Chan ◽  
Emily Ka-Lee Chan ◽  
John Hei-Sing Chow
1989 ◽  
Vol 91 (1) ◽  
pp. 1-5 ◽  
Author(s):  
Stephen P. Staal ◽  
Richard Ambinder ◽  
William E. Beschorner ◽  
Gary S. Hayward ◽  
Risa Mann

1995 ◽  
Vol 15 (2) ◽  
pp. 283-290 ◽  
Author(s):  
Gareth P. Jevon ◽  
Leela J. Elavathil ◽  
Delworth G. Harnish ◽  
Derek J. deSa

2019 ◽  
Vol 29 (2) ◽  
pp. 195-200
Author(s):  
Qing’e Jin ◽  
Jianrong Su ◽  
Donghui Yan ◽  
Shanna Wu

Objective: An association of Epstein-Barr virus (EBV) infection with breast carcinoma (BC) risk has so far been disputed in the literature. Therefore, we performed a meta-analysis to clarify this relationship. Materials and Methods: An electronic database search for eligible case-control studies was performed using PubMed, Embase, Web of Science, the Cochrane Library, CNKI, and Wanfang Data until May 17, 2018. The pooled OR and 95% CI were used to estimate the relationship between EBV infection and BC risk using a fixed or random-effects model depending on heterogeneity. Subgroup analysis and meta-regression were used to explore the heterogeneity. Publication bias was assessed using Egger’s and Harbord’s tests. Results: A total of 16 studies with 1,279 patients and 814 controls were reviewed based on our inclusion and exclusion criteria. Compared with the control group, EBV infection had a significant association with BC risk (OR 4.75, 95% CI 2.53–8.92, p < 0.01) with significant heterogeneity observed (I2 = 65.3%). The subgroup analysis revealed that region and tissue type might explain potential sources of heterogeneity. The sensitivity analyses yielded stable results. No significant publication bias was observed. Conclusion: The current results suggest that EBV infection is significantly associated with increased risk of BC.


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