Three-Dimensional Analysis of the Movement of Lumbar Spinal Nerve Roots in Nonsimulated and Simulated Adhesive Conditions

Spine ◽  
2003 ◽  
Vol 28 (20) ◽  
pp. 2373-2380 ◽  
Author(s):  
Hiroshi Miyamoto ◽  
Genevieve A. Dumas ◽  
Urs P. Wyss ◽  
Leif Ryd
2014 ◽  
Vol 38 (3) ◽  
pp. 229-235 ◽  
Author(s):  
Erkut Turan ◽  
Cengiz Unsal ◽  
Mehmet Utkan Oren ◽  
Omer Gurkan Dilek ◽  
Ismail Gokce Yildirim ◽  
...  

1989 ◽  
Vol 11 (3) ◽  
pp. 221-225 ◽  
Author(s):  
F. de Peretti ◽  
J. P. Micalef ◽  
A. Bourgeon ◽  
C. Argenson ◽  
P. Rabischong

1994 ◽  
Vol 6 (2) ◽  
pp. 230-237
Author(s):  
Donal O'Toole ◽  
Gerald Wells ◽  
James Ingram ◽  
William Cooley ◽  
Stephan Hawkins

The ultrastructural features of a recently described inherited lower motor neuron disease were studied in 5 affected pigs. Clinical signs comprised progressive ataxia and paresis of variable severity. Affected pigs, 6, 7, 15, 15, and 19 weeks of age, and 2 unrelated healthy pigs, 9 and 15 weeks of age, were anesthetized and their tissues were fixed by whole body perfusion with mixed aldehydes. From 1 or more affected pigs, samples of cervical and lumbar spinal ventral horn, lateral and ventral spinal columns, dorsal and ventral lumbar spinal nerve roots, 2 peripheral nerves (Nn. phrenicus and fibularis communis), and 2 skeletal muscles (Mm. diaphragma and tibialis cranialis) were examined ultrastructurally. There was widespread degeneration of myelinated axons in peripheral nerves and in lateral and ventral columns of lumbar and cervical segments of spinal cord. Axonal degeneration was present in ventral spinal nerve roots and was absent in dorsal spinal nerve roots sampled at the same lumbar levels. Unmyelinated axons in peripheral nerves and spinal nerve roots were unaffected. In 4 of 5 affected pigs, there were atrophic alpha motor neurons in cervical spinal cord that contained dense, round osmiophilic perikaryal inclusions up to 4 μm in diameter and round swollen mitochondria. Axonal regeneration was present in N. phrenicus of the 19-week-old affected pig that had clinical signs of longest duration (10 weeks). There was no morphologic evidence of axonal degeneration or spinal neuronal atrophy in either control pig. The ultrastructural features of this motor neuron disease distinguish it from other reported progressive spinal neuropathies of pigs.


2013 ◽  
Vol 20 (11) ◽  
pp. 1546-1549 ◽  
Author(s):  
Christian Strong ◽  
Vijay Yanamadala ◽  
Arjun Khanna ◽  
Brian P. Walcott ◽  
Brian V. Nahed ◽  
...  

2001 ◽  
Vol 57 (5) ◽  
pp. 581-586
Author(s):  
HIDETADA YAMATO ◽  
TOSHIYUKI TAKAHASHI ◽  
TOMONARI FUNATA ◽  
MASARU NITTA ◽  
YASUO NAKAZAWA

2021 ◽  
Vol 64 (4) ◽  
pp. 104187
Author(s):  
Leoni Chiara ◽  
Tedesco Marta ◽  
Talloa Dario ◽  
Verdolotti Tommaso ◽  
Onesimo Roberta ◽  
...  

1987 ◽  
Vol 67 (2) ◽  
pp. 269-277 ◽  
Author(s):  
Wesley W. Parke ◽  
Ryo Watanabe

✓ An epispinal system of motor axons virtually covers the ventral and lateral funiculi of the human conus medullaris between the L-2 and S-2 levels. These nerve fibers apparently arise from motor cells of the ventral horn nuclei and join spinal nerve roots caudal to their level of origin. In all observed spinal cords, many of these axons converged at the cord surface and formed an irregular group of ectopic rootlets that could be visually traced to join conventional spinal nerve roots at one to several segments inferior to their original segmental level; occasional rootlets joined a dorsal nerve root. As almost all previous reports of nerve root interconnections involved only the dorsal roots and have been cited to explain a lack of an absolute segmental sensory nerve distribution, it is believed that these intersegmental motor fibers may similarly explain a more diffuse efferent distribution than has previously been suspected.


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