scholarly journals Mechanisms by Which Chronic Ethanol Feeding Impairs the Migratory Capacity of Cutaneous Dendritic Cells

2013 ◽  
Vol 37 (12) ◽  
pp. 2098-2107 ◽  
Author(s):  
Corey P. Parlet ◽  
Annette J. Schlueter
Keyword(s):  
Dendritic Cells ◽  
Chronic Ethanol ◽  
Migratory Capacity ◽  
Ethanol Feeding

scholarly journals Mechanisms by Which Chronic Ethanol Feeding Limits the Ability of Dendritic Cells to Stimulate T-Cell Proliferation

2010 ◽  
Vol 35 (1) ◽  
pp. 47-59 ◽  
Author(s):  
Ji Fan ◽  
Michelle R. Edsen-Moore ◽  
Lucas E. Turner ◽  
Robert T. Cook ◽  
Kevin L. Legge ◽  
...  
Keyword(s):  
Cell Proliferation ◽  
Dendritic Cells ◽  
T Cell ◽  
Chronic Ethanol ◽  
T Cell Proliferation ◽  
Ethanol Feeding

Plasmin and regulators of plasmin activity control the migratory capacity and adhesion of human T cells and dendritic cells by regulating cleavage of the chemokine CCL21

2016 ◽  
Vol 94 (10) ◽  
pp. 955-963 ◽  
Author(s):  
Natalie Lorenz ◽  
Evert Jan Loef ◽  
Inken D Kelch ◽  
Daniel J Verdon ◽  
Moyra M Black ◽  
...  
Keyword(s):  
T Cells ◽  
Dendritic Cells ◽  
Plasmin Activity ◽  
Migratory Capacity ◽  
Human T Cells

Effect of Chronic Ethanol Feeding on Oxysterols in Rat Liver

2002 ◽  
Vol 36 (6) ◽  
pp. 661-666 ◽  
Author(s):  
Koichi Ariyoshi ◽  
Junko Adachi ◽  
Migiwa Asano ◽  
Yasuhiro Ueno ◽  
Rajkumar Rajendram ◽  
...  
Keyword(s):  
Rat Liver ◽  
Chronic Ethanol ◽  
Ethanol Feeding

scholarly journals Sarcoplasmic reticulum Ca2+ ATPase 2 (SERCA2) reduces the migratory capacity of CCL21-treated monocyte-derived dendritic cells

2016 ◽  
Vol 48 (8) ◽  
pp. e253-e253 ◽  
Author(s):  
Cheol Yi Hong ◽  
Hyun-Ju Lee ◽  
Nu-Ri Choi ◽  
Sung-Hoon Jung ◽  
Manh-Cuong Vo ◽  
...  
Keyword(s):  
Dendritic Cells ◽  
Sarcoplasmic Reticulum ◽  
Migratory Capacity

Rat mitochondrial ATP synthase ATP5G3: cloning and upregulation in pancreas after chronic ethanol feeding

2001 ◽  
Vol 6 (2) ◽  
pp. 91-98 ◽  
Author(s):  
HA-SHENG LI ◽  
JI-YING ZHANG ◽  
BRYAN S. THOMPSON ◽  
XIAO-YING DENG ◽  
MICHAEL E. FORD ◽  
...  
Keyword(s):  
Atp Synthase ◽  
Cellular Response ◽  
Molecular Mechanisms ◽  
Metabolic Stress ◽  
Nuclear Gene ◽  
Chronic Ethanol ◽  
Ethanol Ingestion ◽  
Pancreatic Acinar Cells ◽  
Mitochondrial Atp Synthase ◽  
Ethanol Feeding

Individuals with chronic excessive alcohol ingestion are put at the risk of acute and chronic pancreatitis. Underlying molecular mechanisms are unknown. Differential gene expression in the pancreas was profiled using mRNA differential display by comparison between control and ethanol-consuming rats. Male Wistar rats were fed with diets containing 6.7% (vol/vol) ethanol for 4 wk. A cDNA tag that was overexpressed in the pancreas of rats fed ethanol was isolated. A 723-bp cDNA was cloned from a rat pancreatic cDNA library, which encodes a novel rat mitochondrial ATP synthase subunit 9, isoform 3 (ATP5G3), which is homologous to a human ATP5G3 gene. Real-time PCR demonstrated that all three nuclear gene isoforms (ATP5G1, ATP5G2, and ATP5G3) were consistently upregulated in the pancreas of alcohol-consuming rats, parallel with mitochondrial injury. The cellular response to mitochondrial damage and metabolic stress may reflect an adaptive process for mitochondrial repair in pancreatic acinar cells during chronic ethanol ingestion.

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