scholarly journals Page kidney: Rare cause of acute kidney injury after complicated renal artery angioplasty

Author(s):  
Adrianna Douvris ◽  
Januvi Jegatheswaran ◽  
Adnan Hadziomerovic ◽  
Marcel Ruzicka



Author(s):  
Ilias P. Doulamis ◽  
Alvise Guariento ◽  
Mossab Y. Saeed ◽  
Rio S. Nomoto ◽  
Thomas Duignan ◽  
...  


PLoS ONE ◽  
2013 ◽  
Vol 8 (5) ◽  
pp. e64104 ◽  
Author(s):  
Jingang Yang ◽  
Changlin Lu ◽  
Li Yan ◽  
Xinran Tang ◽  
Wei Li ◽  
...  


2017 ◽  
Vol 51 (5) ◽  
pp. 342-345 ◽  
Author(s):  
Jeko M. Madjarov ◽  
Michael G. Katz ◽  
Hector Crespo-Soto ◽  
Svetozar Madzharov ◽  
Timothy Roush ◽  
...  

Acute dissection of thoracic aorta carries a risk of renal ischemia followed by the development of a kidney failure. The optimal surgical and nonsurgical management of these patients, timing of intervention, and the factors predicting renal recovery are not well delineated and remain controversial. We present a case of acute type B thoracic aortic dissection with left kidney ischemia. Evaluation of renal function was performed by the means of internationally accepted Risk, Injury, Failure, Loss of kidney function, End stage kidney disease and Acute Kidney Injury Network classifications for acute kidney injury, renal duplex sonography, and intravascular ultrasound that demonstrated left renal artery dissection with a flap completely compressing the true lumen. The patient underwent thoracic endovascular aortic repair and left renal artery stent and recovered well. Six months later, at the follow-up visit, retrograde type A aortic dissection was found, which was successfully repaired. Reversal of renal ischemia after aortic dissection depends on the precise assessment of renal function and prompt intervention.



2017 ◽  
pp. bcr-2017-220438
Author(s):  
Subash Somalanka ◽  
Fiona E Harris ◽  
Eric Chemla ◽  
Rebecca Jo Suckling ◽  
Pauline A Swift


2014 ◽  
Vol 4 (1) ◽  
pp. 82-87 ◽  
Author(s):  
Niina Koivuviita ◽  
Risto Tertti ◽  
Maija Heiro ◽  
Ilkka Manner ◽  
Kaj Metsärinne




Author(s):  
Lesley K. Bowker ◽  
James D. Price ◽  
Ku Shah ◽  
Sarah C. Smith

This chapter provides information on the ageing kidney, acute kidney injury, management of acute kidney injury, chronic kidney disease, complications of chronic kidney disease, dialysis in renal replacement therapy, transplantation in renal replacement therapy, nephrotic syndrome, glomerulonephritis, and renal artery stenosis.



Author(s):  
Tim Raine ◽  
James Dawson ◽  
Stephan Sanders ◽  
Simon Eccles

Acute kidney injuryChronic renal failureHaematuriaProteinuriaGlomerular diseaseUrological disordersLow urine outputIV fluidsPotassium emergenciesElectrolyte imbalanceAcute rise from baseline of serum urea and creatinine ±oliguria ( Table 12.1);1there are three basic mechanisms:•Prerenal hypoperfusion of kidney due to eg ↓BP, hypovolaemia, renal artery occlusion (mass, emboli)...



2018 ◽  
Vol 7 (12) ◽  
pp. 470 ◽  
Author(s):  
Hae Kil ◽  
Ji Kim ◽  
Young Choi ◽  
Hye Lee ◽  
Tae Kim ◽  
...  

We evaluated postoperative renal function in patients with/without combined therapy of ketorolac and remote ischemic preconditioning during partial nephrectomy. Sixteen patients were randomly allocated to either the ketorolac combined with RIPC group (KI, n = 8) or control group (n = 8). The KI group received both remote ischemic preconditioning before surgery and intravenous ketorolac of 1 mg/kg before renal artery clamping. Renal parameters were measured before induction, after anesthesia induction, and 2, 12, 24, 48, and 72 h after renal artery declamping. Acute kidney injury was assessed by Acute Kidney Injury Network criteria. The estimated glomerular filtration rate decreased in both groups, but then increased significantly at 48 h and 72 h after declamping only in the KI group compared to 24 h (p = 0.001 and p = 0.016). Additionally, it was higher at 48 h and 72 h after declamping in the KI group compared to the control group (p = 0.025 and p = 0.044). The incidence of acute kidney injury was significantly reduced in the KI group (13%) compared to the control group (83%) (p = 0.026). FENa was markedly increased at 2 h after declamping, and recovered in both groups, but it was more significant at 12 h after declamping in the KI group (p = 0.022). Urinary N-acetyl-1-β-D-glucosoaminidase and serum neutrophil gelatinase-associated lipocalin were similar (p = 0.291 and p = 0.818). There is a possibility that combined therapy of ketorolac and remote ischemic preconditioning prior to ischemia may alleviate renal dysfunction and reduce the incidence of acute kidney injury in patients undergoing partial nephrectomy.



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