Oscillatory Blood Flow in a Deformable Human Aortic Arch

2011 ◽  
Author(s):  
Jing Wang ◽  
Suzie Brown ◽  
Stephen W. Tullis
Keyword(s):  
Blood Flow ◽  
Cardiovascular Diseases ◽  
Aortic Arch ◽  
Vessel Wall ◽  
Cardiac Cycle ◽  
The Body ◽  
Mechanical Interaction ◽  
Pulsatile Blood Flow ◽  
Medical Device Design ◽  
Large Vessels

The aorta is the largest artery in humans, stemming from the left ventricle of the heart and stretching down to the abdomen. It is responsible for distributing oxygenated blood to the rest of the body during each cardiac cycle. The pulsatile blood flow is complex in nature and has been previously modeled computationally in an effort to understand its effect on cardiovascular diseases and medical device design interaction [4,8–9]. However, the majority of these models either treat the vessel wall as rigid or have significantly simplified geometries, which from a physiological perspective are not true of large vessels such as the aorta. Here, the complex mechanical interaction between pulsatile blood flow and wall dynamics in the aortic arch is investigated using geometry adopted directly from CT images.

scholarly journals An Ultrasound Simulation Model for the Pulsatile Blood Flow Modulated by the Motion of Stenosed Vessel Wall

2016 ◽  
Vol 2016 ◽  
pp. 1-16
Author(s):  
Qinghui Zhang ◽  
Yufeng Zhang ◽  
Yi Zhou ◽  
Kun Zhang ◽  
Kexin Zhang ◽  
...  
Keyword(s):  
Blood Flow ◽  
Flow Field ◽  
Simulation Model ◽  
Vessel Wall ◽  
Wall Motion ◽  
Simulation Software ◽  
Pulsatile Blood Flow ◽  
Ultrasound Simulation ◽  
Wall Movement ◽  
Stenosed Vessel

This paper presents an ultrasound simulation model for pulsatile blood flow, modulated by the motion of a stenosed vessel wall. It aims at generating more realistic ultrasonic signals to provide an environment for evaluating ultrasound signal processing and imaging and a framework for investigating the behaviors of blood flow field modulated by wall motion. This model takes into account fluid-structure interaction, blood pulsatility, stenosis of the vessel, and arterial wall movement caused by surrounding tissue’s motion. The axial and radial velocity distributions of blood and the displacement of vessel wall are calculated by solving coupled Navier-Stokes and wall equations. With these obtained values, we made several different phantoms by treating blood and the vessel wall as a group of point scatterers. Then, ultrasound echoed signals from oscillating wall and blood in the axisymmetric stenotic-carotid arteries were computed by ultrasound simulation software, Field II. The results show better consistency with corresponding theoretical values and clinical data and reflect the influence of wall movement on the flow field. It can serve as an effective tool not only for investigating the behavior of blood flow field modulated by wall motion but also for quantitative or qualitative evaluation of new ultrasound imaging technology and estimation method of blood velocity.

Aortic Arch Repair (Circulatory Arrest)

2019 ◽  
pp. 263-267
Author(s):  
Shyamal Asher
Keyword(s):  
Blood Flow ◽  
Aortic Arch ◽  
Circulatory Arrest ◽  
Anesthetic Management ◽  
Deep Hypothermia ◽  
The Body ◽  
Adequate Knowledge ◽  
Aortic Arch Repair ◽  
Aortic Dissections ◽  
The Brain

Aortic arch repair is a technically challenging surgery that requires collaboration between the anesthesiology, cardiac surgery, and perfusion teams. To accomplish a total aortic arch repair, blood flow to the brain and the rest of the body has to be interrupted. The most common aortic arch pathologies encountered for surgery are aortic arch aneurysms followed by aortic dissections. The need for hypothermia and circulatory arrest during aortic arch surgeries leads to unique implications for anesthetic management. Therefore, adequate knowledge of the planned surgery and specific surgical and nonsurgical cerebral protection techniques are necessary. Furthermore, an understanding of intraoperative neurophysiologic and temperature monitoring at deep hypothermia as well as postbypass coagulopathy management are needed in these challenging cases.

Effects of pulsatile blood flow in large vessels on thermal dose distribution during thermal therapy

2007 ◽  
Vol 34 (4) ◽  
pp. 1312-1320 ◽  
Author(s):  
Tzyy-Leng Horng ◽  
Win-Li Lin ◽  
Chihng-Tsung Liauh ◽  
Tzu-Ching Shih
Keyword(s):  
Blood Flow ◽  
Dose Distribution ◽  
Thermal Therapy ◽  
Thermal Dose ◽  
Pulsatile Blood Flow ◽  
Large Vessels

scholarly journals Could the heat sink effect of blood flow inside large vessels protect the vessel wall from thermal damage during RF-assisted surgical resection?

2014 ◽  
Vol 41 (8Part1) ◽  
pp. 083301 ◽  
Author(s):  
Ana González-Suárez ◽  
Macarena Trujillo ◽  
Fernando Burdío ◽  
Anna Andaluz ◽  
Enrique Berjano
Keyword(s):  
Blood Flow ◽  
Surgical Resection ◽  
Heat Sink ◽  
Vessel Wall ◽  
Thermal Damage ◽  
Sink Effect ◽  
Large Vessels

scholarly journals Finite Element Modelling of Pulsatile Blood Flow in Idealized Model of Human Aortic Arch: Study of Hypotension and Hypertension

2012 ◽  
Vol 2012 ◽  
pp. 1-14 ◽  
Author(s):  
Paritosh Vasava ◽  
Payman Jalali ◽  
Mahsa Dabagh ◽  
Pertti J. Kolari
Keyword(s):  
Finite Element ◽  
Blood Flow ◽  
Shear Stress ◽  
Aortic Arch ◽  
Human Aorta ◽  
Shear Stresses ◽  
Pulsatile Blood Flow ◽  
Increased Risk ◽  
Pulsatile Pressure ◽  
Wall Shear

A three-dimensional computer model of human aortic arch with three branches is reproduced to study the pulsatile blood flow with Finite Element Method. In specific, the focus is on variation of wall shear stress, which plays an important role in the localization and development of atherosclerotic plaques. Pulsatile pressure pulse is used as boundary condition to avoid flow entry development, and the aorta walls are considered rigid. The aorta model along with boundary conditions is altered to study the effect of hypotension and hypertension. The results illustrated low and fluctuating shear stress at outer and inner wall of aortic arch, proximal wall of branches, and entry region. Despite the simplification of aorta model, rigid walls and other assumptions results displayed that hypertension causes lowered local wall shear stresses. It is the sign of an increased risk of atherosclerosis. The assessment of hemodynamics shows that under the flow regimes of hypotension and hypertension, the risk of atherosclerosis localization in human aorta may increase.

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